Induction by antitumoral drugs of proteins that functionally complement CFTR: a novel therapy for cystic fibrosis?

Lancet. 1997 Sep 6;350(9079):711-2. doi: 10.1016/s0140-6736(05)63510-6.
No abstract available

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • ATP-Binding Cassette Transporters / drug effects*
  • ATP-Binding Cassette Transporters / metabolism
  • ATP-Binding Cassette, Sub-Family C Proteins
  • Antineoplastic Agents / pharmacology*
  • Antineoplastic Agents / therapeutic use
  • Antiporters / drug effects
  • Antiporters / metabolism
  • Cystic Fibrosis / drug therapy*
  • Cystic Fibrosis / genetics
  • Cystic Fibrosis / metabolism
  • Cystic Fibrosis Transmembrane Conductance Regulator / drug effects*
  • Cystic Fibrosis Transmembrane Conductance Regulator / genetics
  • Cystic Fibrosis Transmembrane Conductance Regulator / metabolism
  • Humans
  • Male
  • Mutation
  • Neoplasm Proteins / drug effects
  • Neoplasm Proteins / metabolism
  • RNA, Messenger / genetics

Substances

  • ATP-Binding Cassette Transporters
  • Antineoplastic Agents
  • Antiporters
  • Cystic Fibrosis Transmembrane Conductance Regulator
  • ATP-Binding Cassette, Sub-Family C Proteins
  • Neoplasm Proteins
  • RNA, Messenger
  • CFTR protein, human