[Development of prediction for NIDDM from a view point of genetics]

Nihon Rinsho. 1997 Nov;55 Suppl:362-8.
[Article in Japanese]
No abstract available

Publication types

  • Review

MeSH terms

  • Amyloid / genetics
  • DNA, Mitochondrial / genetics
  • DNA-Binding Proteins*
  • Diabetes Mellitus, Type 2 / genetics*
  • Glucokinase / genetics
  • Glycogen Synthase / genetics
  • Hepatocyte Nuclear Factor 1
  • Hepatocyte Nuclear Factor 1-alpha
  • Hepatocyte Nuclear Factor 1-beta
  • Humans
  • Insulin / genetics
  • Islet Amyloid Polypeptide
  • Mutation
  • Nuclear Proteins*
  • Receptor, Insulin / genetics
  • Transcription Factors / genetics

Substances

  • Amyloid
  • DNA, Mitochondrial
  • DNA-Binding Proteins
  • HNF1A protein, human
  • HNF1B protein, human
  • Hepatocyte Nuclear Factor 1-alpha
  • Insulin
  • Islet Amyloid Polypeptide
  • Nuclear Proteins
  • Transcription Factors
  • Hepatocyte Nuclear Factor 1
  • Hepatocyte Nuclear Factor 1-beta
  • Glycogen Synthase
  • Glucokinase
  • Receptor, Insulin