Requirement for membrane lipid peroxidation in HIV-1 gp120-induced neuroblastoma cell death

Biochem Biophys Res Commun. 1998 May 29;246(3):686-9. doi: 10.1006/bbrc.1998.8687.

Abstract

The HIV-1 envelope protein gp120 engenders Ca(2+)-mediated, excitotoxic damage of rodent neuronal and human neuroblastoma cells in culture. Here we report that human CHP100 neuroblastoma cells undergo early peroxidation of membrane lipids following a brief exposure to gp120. This effect is prevented by preincubating cell cultures with the 21-aminosteroid U-74389G, an inhibitor of membrane lipid peroxidation, which also rescues neuroblastoma cultures from gp120-induced cell death; conversely, no protection from cell death is observed when the 21-aminosteroid is added to neuroblastoma cultures after the induction of membrane lipid peroxidation by gp120. These data indicate that membrane lipid peroxidation has a causative role in the expression of cell death produced by the viral protein.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antioxidants / pharmacology
  • Calcium / metabolism
  • Cell Death*
  • HIV Envelope Protein gp120 / toxicity*
  • HIV-1*
  • Humans
  • Lipid Peroxidation*
  • Membrane Lipids / metabolism*
  • Neuroblastoma
  • Neurons / drug effects*
  • Pregnatrienes / pharmacology
  • Receptors, N-Methyl-D-Aspartate / metabolism
  • Tumor Cells, Cultured

Substances

  • Antioxidants
  • HIV Envelope Protein gp120
  • Membrane Lipids
  • Pregnatrienes
  • Receptors, N-Methyl-D-Aspartate
  • U 74389F
  • Calcium