Incomplete lacunar infarction (Type Ib lacunes)

Acta Neuropathol. 1998 Aug;96(2):163-71. doi: 10.1007/s004010050877.


The aetiopathogenesis of small, deep (lacunar) infarcts remains controversial. The view that they are caused by occlusive intrinsic small vessel disease is widely held, but is based on only a small number of detailed pathology studies. We describe and illustrate a variant of small, microvessel-associated basal ganglia lesion, the histopathological features of which are distinct from those of classical Types I, II and III lacunes. Their appearances suggest a state of incomplete infarction. The pathogenetic significance of such lesions is discussed, in particular the role of mechanisms causing temporary or only moderately severe ischaemia.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Basal Ganglia / diagnostic imaging
  • Basal Ganglia / pathology*
  • Blood Vessels / pathology
  • Cerebral Infarction / diagnostic imaging
  • Cerebral Infarction / pathology*
  • Cerebrovascular Circulation
  • Female
  • Humans
  • Intracranial Embolism and Thrombosis / pathology
  • Male
  • Middle Aged
  • Radiography