Abstract
Snakes and cone snails produce toxins which block muscular and/or neuronal nicotinic acetylcholine receptors (AChRs). This paper mostly focuses on the determinants by which a snake long chain curaremimetic toxin and the cone snail toxin ImI bind specifically to the alpha 7 neuronal receptor. In both cases, the site involves a small turn-like structure constrained by two half-cystines.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Amino Acid Sequence
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Animals
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Charybdotoxin / chemistry
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Charybdotoxin / pharmacology
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Conotoxins*
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Curare / analogs & derivatives
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Curare / chemistry
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Curare / pharmacology
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Erabutoxins / chemistry
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Erabutoxins / pharmacology
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Humans
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Kinetics
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Molecular Sequence Data
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Mollusk Venoms / chemical synthesis
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Mollusk Venoms / chemistry*
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Mollusk Venoms / pharmacology
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Mutagenesis, Site-Directed
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Neurons / physiology*
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Nicotinic Antagonists / chemical synthesis
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Nicotinic Antagonists / chemistry*
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Nicotinic Antagonists / pharmacology
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Oligopeptides / chemical synthesis
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Oligopeptides / chemistry*
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Oligopeptides / pharmacology
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Receptors, Nicotinic / chemistry*
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Receptors, Nicotinic / metabolism*
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Recombinant Fusion Proteins / chemistry
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Recombinant Fusion Proteins / metabolism
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Sequence Alignment
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Snails
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Snake Venoms / chemical synthesis
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Snake Venoms / chemistry*
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Snake Venoms / pharmacology
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alpha7 Nicotinic Acetylcholine Receptor
Substances
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Chrna7 protein, human
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Conotoxins
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Mollusk Venoms
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Nicotinic Antagonists
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Oligopeptides
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Receptors, Nicotinic
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Recombinant Fusion Proteins
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Snake Venoms
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alpha7 Nicotinic Acetylcholine Receptor
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Charybdotoxin
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alpha-conotoxin ImI
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Erabutoxins
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Curare