Di Vona C[AUTHOR] - Search Results

Year	Number of Results
2015	1
2018	2
2019	3
2020	1
2021	1
2022	2
2023	2
2024	1
2025	0

1

The dual-specificity kinase DYRK1A interacts with the Hepatitis B virus genome and regulates the production of viral RNA.

Pastor F, Charles E, Di Vona C, Chapelle M, Rivoire M, Passot G, Chabot B, de la Luna S, Lucifora J, Durantel D, Salvetti A. Pastor F, et al. Among authors: di vona c. PLoS One. 2024 Oct 15;19(10):e0311655. doi: 10.1371/journal.pone.0311655. eCollection 2024. PLoS One. 2024. PMID: 39405283 Free PMC article.

2

A novel CDC25A/DYRK2 regulatory switch modulates cell cycle and survival.

Lara-Chica M, Correa-Sáez A, Jiménez-Izquierdo R, Garrido-Rodríguez M, Ponce FJ, Moreno R, Morrison K, Di Vona C, Arató K, Jiménez-Jiménez C, Morrugares R, Schmitz ML, de la Luna S, de la Vega L, Calzado MA. Lara-Chica M, et al. Among authors: di vona c. Cell Death Differ. 2022 Jan;29(1):105-117. doi: 10.1038/s41418-021-00845-5. Epub 2021 Aug 6. Cell Death Differ. 2022. PMID: 34363019 Free PMC article.

3

DYRK1A Kinase Positively Regulates Angiogenic Responses in Endothelial Cells.

Rozen EJ, Roewenstrunk J, Barallobre MJ, Di Vona C, Jung C, Figueiredo AF, Luna J, Fillat C, Arbonés ML, Graupera M, Valverde MA, de la Luna S. Rozen EJ, et al. Among authors: di vona c. Cell Rep. 2018 May 8;23(6):1867-1878. doi: 10.1016/j.celrep.2018.04.008. Cell Rep. 2018. PMID: 29742440 Free article.

4

An RNA Polymerase III General Transcription Factor Engages in Cell Type-Specific Chromatin Looping.

de Llobet Cucalon L, Di Vona C, Morselli M, Vezzoli M, Montanini B, Teichmann M, de la Luna S, Ferrari R. de Llobet Cucalon L, et al. Among authors: di vona c. Int J Mol Sci. 2022 Feb 18;23(4):2260. doi: 10.3390/ijms23042260. Int J Mol Sci. 2022. PMID: 35216376 Free PMC article.

5

Arginine Citrullination at the C-Terminal Domain Controls RNA Polymerase II Transcription.

Sharma P, Lioutas A, Fernandez-Fuentes N, Quilez J, Carbonell-Caballero J, Wright RHG, Di Vona C, Le Dily F, Schüller R, Eick D, Oliva B, Beato M. Sharma P, et al. Among authors: di vona c. Mol Cell. 2019 Jan 3;73(1):84-96.e7. doi: 10.1016/j.molcel.2018.10.016. Epub 2018 Nov 21. Mol Cell. 2019. PMID: 30472187 Free article.

6

TFIIIC Binding to Alu Elements Controls Gene Expression via Chromatin Looping and Histone Acetylation.

Ferrari R, de Llobet Cucalon LI, Di Vona C, Le Dilly F, Vidal E, Lioutas A, Oliete JQ, Jochem L, Cutts E, Dieci G, Vannini A, Teichmann M, de la Luna S, Beato M. Ferrari R, et al. Among authors: di vona c. Mol Cell. 2020 Feb 6;77(3):475-487.e11. doi: 10.1016/j.molcel.2019.10.020. Epub 2019 Nov 20. Mol Cell. 2020. PMID: 31759822 Free PMC article.

7

TFIIIC as a Potential Epigenetic Modulator of Histone Acetylation in Human Stem Cells.

Vezzoli M, de Llobet Cucalon LI, Di Vona C, Morselli M, Montanini B, de la Luna S, Teichmann M, Dieci G, Ferrari R. Vezzoli M, et al. Among authors: di vona c. Int J Mol Sci. 2023 Feb 11;24(4):3624. doi: 10.3390/ijms24043624. Int J Mol Sci. 2023. PMID: 36835038 Free PMC article.

8

Loss of the DYRK1A Protein Kinase Results in the Reduction in Ribosomal Protein Gene Expression, Ribosome Mass and Reduced Translation.

Di Vona C, Barba L, Ferrari R, de la Luna S. Di Vona C, et al. Biomolecules. 2023 Dec 25;14(1):31. doi: 10.3390/biom14010031. Biomolecules. 2023. PMID: 38254631 Free PMC article.

9

Chromatin-wide profiling of DYRK1A reveals a role as a gene-specific RNA polymerase II CTD kinase.

Di Vona C, Bezdan D, Islam AB, Salichs E, López-Bigas N, Ossowski S, de la Luna S. Di Vona C, et al. Mol Cell. 2015 Feb 5;57(3):506-20. doi: 10.1016/j.molcel.2014.12.026. Epub 2015 Jan 22. Mol Cell. 2015. PMID: 25620562 Free article.

10

A comprehensive proteomics-based interaction screen that links DYRK1A to RNF169 and to the DNA damage response.

Roewenstrunk J, Di Vona C, Chen J, Borras E, Dong C, Arató K, Sabidó E, Huen MSY, de la Luna S. Roewenstrunk J, et al. Among authors: di vona c. Sci Rep. 2019 Apr 12;9(1):6014. doi: 10.1038/s41598-019-42445-x. Sci Rep. 2019. PMID: 30979931 Free PMC article.

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