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2016 1
2017 2
2018 1
2021 0
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Page 1
Persistent KSHV Infection Increases EBV-Associated Tumor Formation In Vivo via Enhanced EBV Lytic Gene Expression.
McHugh D, Caduff N, Barros MHM, Rämer PC, Raykova A, Murer A, Landtwing V, Quast I, Styles CT, Spohn M, Fowotade A, Delecluse HJ, Papoudou-Bai A, Lee YM, Kim JM, Middeldorp J, Schulz TF, Cesarman E, Zbinden A, Capaul R, White RE, Allday MJ, Niedobitek G, Blackbourn DJ, Grundhoff A, Münz C. McHugh D, et al. Among authors: styles ct. Cell Host Microbe. 2017 Jul 12;22(1):61-73.e7. doi: 10.1016/j.chom.2017.06.009. Cell Host Microbe. 2017. PMID: 28704654 Free article.
The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency.
Styles CT, Paschos K, White RE, Farrell PJ. Styles CT, et al. Pathogens. 2018 Mar 17;7(1):31. doi: 10.3390/pathogens7010031. Pathogens. 2018. PMID: 29562595 Free PMC article. Review.
EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.
Styles CT, Bazot Q, Parker GA, White RE, Paschos K, Allday MJ. Styles CT, et al. PLoS Biol. 2017 Aug 3;15(8):e2001992. doi: 10.1371/journal.pbio.2001992. eCollection 2017 Aug. PLoS Biol. 2017. PMID: 28771465 Free PMC article.
Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells.
Kalchschmidt JS, Bashford-Rogers R, Paschos K, Gillman AC, Styles CT, Kellam P, Allday MJ. Kalchschmidt JS, et al. Among authors: styles ct. J Exp Med. 2016 May 30;213(6):921-8. doi: 10.1084/jem.20160120. Epub 2016 May 23. J Exp Med. 2016. PMID: 27217538 Free PMC article.