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2008 1
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2019 1
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The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency.
Styles CT, Paschos K, White RE, Farrell PJ. Styles CT, et al. Among authors: paschos k. Pathogens. 2018 Mar 17;7(1):31. doi: 10.3390/pathogens7010031. Pathogens. 2018. PMID: 29562595 Free PMC article. Review.
Requirement for PRC1 subunit BMI1 in host gene activation by Epstein-Barr virus protein EBNA3C.
Paschos K, Bazot Q, Lees J, Farrell PJ, Allday MJ. Paschos K, et al. Nucleic Acids Res. 2019 Apr 8;47(6):2807-2821. doi: 10.1093/nar/gky1323. Nucleic Acids Res. 2019. PMID: 30649516 Free PMC article.
Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.
Bazot Q, Paschos K, Allday MJ. Bazot Q, et al. Among authors: paschos k. J Virol. 2018 Mar 14;92(7):e01918-17. doi: 10.1128/JVI.01918-17. Print 2018 Apr 1. J Virol. 2018. PMID: 29367247 Free PMC article.
EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.
Styles CT, Bazot Q, Parker GA, White RE, Paschos K, Allday MJ. Styles CT, et al. Among authors: paschos k. PLoS Biol. 2017 Aug 3;15(8):e2001992. doi: 10.1371/journal.pbio.2001992. eCollection 2017 Aug. PLoS Biol. 2017. PMID: 28771465 Free PMC article.
Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression.
Paschos K, Bazot Q, Ho G, Parker GA, Lees J, Barton G, Allday MJ. Paschos K, et al. Nucleic Acids Res. 2017 Mar 17;45(5):2368-2383. doi: 10.1093/nar/gkw1167. Nucleic Acids Res. 2017. PMID: 27903901 Free PMC article.
Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells.
Kalchschmidt JS, Bashford-Rogers R, Paschos K, Gillman AC, Styles CT, Kellam P, Allday MJ. Kalchschmidt JS, et al. Among authors: paschos k. J Exp Med. 2016 May 30;213(6):921-8. doi: 10.1084/jem.20160120. Epub 2016 May 23. J Exp Med. 2016. PMID: 27217538 Free PMC article.
BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV.
Paschos K, Parker GA, Watanatanasup E, White RE, Allday MJ. Paschos K, et al. Nucleic Acids Res. 2012 Aug;40(15):7233-46. doi: 10.1093/nar/gks391. Epub 2012 May 14. Nucleic Acids Res. 2012. PMID: 22584624 Free PMC article.
EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells.
Kalchschmidt JS, Gillman AC, Paschos K, Bazot Q, Kempkes B, Allday MJ. Kalchschmidt JS, et al. Among authors: paschos k. PLoS Pathog. 2016 Jan 11;12(1):e1005383. doi: 10.1371/journal.ppat.1005383. eCollection 2016 Jan. PLoS Pathog. 2016. PMID: 26751214 Free PMC article.
Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.
Bazot Q, Paschos K, Skalska L, Kalchschmidt JS, Parker GA, Allday MJ. Bazot Q, et al. Among authors: paschos k. PLoS Pathog. 2015 Jul 8;11(7):e1005031. doi: 10.1371/journal.ppat.1005031. eCollection 2015 Jul. PLoS Pathog. 2015. PMID: 26153983 Free PMC article.
Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.
Skalska L, White RE, Parker GA, Turro E, Sinclair AJ, Paschos K, Allday MJ. Skalska L, et al. Among authors: paschos k. PLoS Pathog. 2013 Feb;9(2):e1003187. doi: 10.1371/journal.ppat.1003187. Epub 2013 Feb 21. PLoS Pathog. 2013. PMID: 23436997 Free PMC article.
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