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964 results

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Page 1
Showing results for h willi
Search for H Nilli instead (2 results)
Clinical Indications for Growth Hormone Therapy.
Danowitz M, Grimberg A. Danowitz M, et al. Adv Pediatr. 2022 Aug;69(1):203-217. doi: 10.1016/j.yapd.2022.03.005. Epub 2022 Jun 17. Adv Pediatr. 2022. PMID: 35985710 Free PMC article. Review.
In the United States, there are 8 Food and Drug Administration (FDA)-approved indications for pediatric GH therapy: GH deficiency, Prader-Willi Syndrome, small for gestational age (SGA) without catch-up growth, idiopathic short stature, Turner syndrome, SHOX gene haploinsu …
In the United States, there are 8 Food and Drug Administration (FDA)-approved indications for pediatric GH therapy: GH deficiency, Prader- …
Liraglutide for Weight Management in Children and Adolescents With Prader-Willi Syndrome and Obesity.
Diene G, Angulo M, Hale PM, Jepsen CH, Hofman PL, Hokken-Koelega A, Ramesh C, Turan S, Tauber M. Diene G, et al. J Clin Endocrinol Metab. 2022 Dec 17;108(1):4-12. doi: 10.1210/clinem/dgac549. J Clin Endocrinol Metab. 2022. PMID: 36181471 Free PMC article. Clinical Trial.
CONTEXT: Prader-Willi syndrome (PWS) is characterized by lack of appetite control and hyperphagia, leading to obesity. ...
CONTEXT: Prader-Willi syndrome (PWS) is characterized by lack of appetite control and hyperphagia, leading to obesity. ...
[Prader-Willi (H.H.H.O.) syndrome].
Barta L, Sziklay L. Barta L, et al. Orv Hetil. 1970 Jul 19;111(29):1710-1. Orv Hetil. 1970. PMID: 5495296 Hungarian. No abstract available.
Prader-Willi syndrome.
Cassidy SB, Driscoll DJ. Cassidy SB, et al. Eur J Hum Genet. 2009 Jan;17(1):3-13. doi: 10.1038/ejhg.2008.165. Epub 2008 Sep 10. Eur J Hum Genet. 2009. PMID: 18781185 Free PMC article.
Prader-Willi syndrome (PWS) is a highly variable genetic disorder affecting multiple body systems whose most consistent major manifestations include hypotonia with poor suck and poor weight gain in infancy; mild mental retardation, hypogonadism, growth hormone insufficienc …
Prader-Willi syndrome (PWS) is a highly variable genetic disorder affecting multiple body systems whose most consistent major manifes …
Prader-Willi syndrome.
Martin A, State M, Koenig K, Schultz R, Dykens EM, Cassidy SB, Leckman JF. Martin A, et al. Am J Psychiatry. 1998 Sep;155(9):1265-73. doi: 10.1176/ajp.155.9.1265. Am J Psychiatry. 1998. PMID: 9734553 No abstract available.
Oxytocin in neurodevelopmental disorders: Autism spectrum disorder and Prader-Willi syndrome.
Josselsohn A, Zhao Y, Espinoza D, Hollander E. Josselsohn A, et al. Pharmacol Ther. 2024 Dec;264:108734. doi: 10.1016/j.pharmthera.2024.108734. Epub 2024 Oct 23. Pharmacol Ther. 2024. PMID: 39455012 Free PMC article. Review.
This manuscript reviews recent work on oxytocin and its use in neurodevelopmental disorders including spectrum disorder (ASD) and Prader-Willi syndrome (PWS). Oxytocin is involved in social recognition, bonding, maternal behaviors, anxiety, food motivation, and hyperphagia …
This manuscript reviews recent work on oxytocin and its use in neurodevelopmental disorders including spectrum disorder (ASD) and Prader- …
Epigenetic therapy of Prader-Willi syndrome.
Kim Y, Wang SE, Jiang YH. Kim Y, et al. Transl Res. 2019 Jun;208:105-118. doi: 10.1016/j.trsl.2019.02.012. Epub 2019 Mar 5. Transl Res. 2019. PMID: 30904443 Free PMC article. Review.
Prader-Willi syndrome (PWS) is a complex and multisystem neurobehavioral disorder. The molecular mechanism of PWS is deficiency of paternally expressed gene gene or genes from the chromosome 15q11-q13. ...
Prader-Willi syndrome (PWS) is a complex and multisystem neurobehavioral disorder. The molecular mechanism of PWS is deficiency of pa …
Rescue of imprinted genes by epigenome editing in human cellular models of Prader-Willi syndrome.
Nemoto A, Imaizumi K, Miya F, Hiroi Y, Yamada M, Ideno H, Saitoh S, Kosaki K, Okuno H, Okano H. Nemoto A, et al. Nat Commun. 2025 Oct 28;16(1):9442. doi: 10.1038/s41467-025-64932-8. Nat Commun. 2025. PMID: 41152294 Free PMC article.
Prader-Willi syndrome (PWS) is a genomic imprinting disorder caused by the loss of function of the paternal chromosome 15q11-13, resulting in a spectrum of symptoms associated with hypothalamic dysfunction. ...
Prader-Willi syndrome (PWS) is a genomic imprinting disorder caused by the loss of function of the paternal chromosome 15q11-13, resu …
Epigenetics meets GPCR: inhibition of histone H3 methyltransferase (G9a) and histamine H(3) receptor for Prader-Willi Syndrome.
Reiner D, Seifert L, Deck C, Schüle R, Jung M, Stark H. Reiner D, et al. Sci Rep. 2020 Aug 11;10(1):13558. doi: 10.1038/s41598-020-70523-y. Sci Rep. 2020. PMID: 32782417 Free PMC article.
Inhibition of histone H3 lysine N-methyltransferase, especially G9a, has been recently shown to restore candidate genes from silenced parental chromosomes in the imprinting disorder Prader-Willi syndrome (PWS). In addition to this epigenetic approach, pitolisant as G-prote …
Inhibition of histone H3 lysine N-methyltransferase, especially G9a, has been recently shown to restore candidate genes from silenced parent …
964 results