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Showing results for K. zamzami n
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Plasma membrane potential in thymocyte apoptosis.
Dallaporta B, Marchetti P, de Pablo MA, Maisse C, Duc HT, Métivier D, Zamzami N, Geuskens M, Kroemer G. Dallaporta B, et al. Among authors: zamzami n. J Immunol. 1999 Jun 1;162(11):6534-42. J Immunol. 1999. PMID: 10352269
Unexpectedly, we found that blockade of plasma membrane K+ channels by tetrapentylammonium (TPA), which leads to a DeltaP sip collapse, can prevent the thymocyte apoptosis induced by exposure to the glucocorticoid receptor agonist dexamethasone, the topoisomerase inhibitor …
Unexpectedly, we found that blockade of plasma membrane K+ channels by tetrapentylammonium (TPA), which leads to a DeltaP sip collaps …
Potassium leakage during the apoptotic degradation phase.
Dallaporta B, Hirsch T, Susin SA, Zamzami N, Larochette N, Brenner C, Marzo I, Kroemer G. Dallaporta B, et al. Among authors: zamzami n. J Immunol. 1998 Jun 1;160(11):5605-15. J Immunol. 1998. PMID: 9605166
Irrespective of the death-inducing stimulus (glucocorticoids, topoisomerase inhibition, or Fas-crosslinking), a significant K+ outflow was observed during apoptosis, as determined on the single-cell level by means of the K+-sensitive fluorochrome, benzofuran isophta …
Irrespective of the death-inducing stimulus (glucocorticoids, topoisomerase inhibition, or Fas-crosslinking), a significant K+ outflo …