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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1999 1
2000 1
2001 4
2002 1
2004 3
2021 0
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Page 1
Evolution of expression of cardiac phenotypes over a 4-year period in the beta-myosin heavy chain-Q403 transgenic rabbit model of human hypertrophic cardiomyopathy.
Nagueh SF, Chen S, Patel R, Tsybouleva N, Lutucuta S, Kopelen HA, Zoghbi WA, Quiñones MA, Roberts R, Marian AJ. Nagueh SF, et al. Among authors: lutucuta s. J Mol Cell Cardiol. 2004 May;36(5):663-73. doi: 10.1016/j.yjmcc.2004.02.010. J Mol Cell Cardiol. 2004. PMID: 15135661 Free PMC article.
Aldosterone, through novel signaling proteins, is a fundamental molecular bridge between the genetic defect and the cardiac phenotype of hypertrophic cardiomyopathy.
Tsybouleva N, Zhang L, Chen S, Patel R, Lutucuta S, Nemoto S, DeFreitas G, Entman M, Carabello BA, Roberts R, Marian AJ. Tsybouleva N, et al. Among authors: lutucuta s. Circulation. 2004 Mar 16;109(10):1284-91. doi: 10.1161/01.CIR.0000121426.43044.2B. Epub 2004 Mar 1. Circulation. 2004. PMID: 14993121 Free PMC article.
[Primary hyperaldosteronism--12 clinical cases].
Telles EC, Machado FS, da Silva EP, Lutucuta S, Almeida G, Coucelo J, Vagueiro C. Telles EC, et al. Among authors: lutucuta s. Rev Port Cardiol. 1999 Sep;18(9):801-12. Rev Port Cardiol. 1999. PMID: 10536470 Portuguese.