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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1902 1
1903 1
1964 1
1971 1
1975 2
1976 1
1977 2
1978 1
1981 2
1983 1
1984 2
1985 1
1986 1
1987 3
1988 5
1989 4
1990 3
1991 3
1992 5
1993 5
1994 4
1995 5
1996 3
1997 1
1998 10
1999 10
2000 9
2001 10
2002 12
2003 7
2004 12
2005 16
2006 22
2007 22
2008 26
2009 46
2010 55
2011 102
2012 140
2013 214
2014 277
2015 368
2016 352
2017 385
2018 498
2019 514
2020 551
2021 661
2022 570
2023 539
2024 488
2025 301

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5,585 results

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Page 1
CircGPR137B/miR-4739/FTO feedback loop suppresses tumorigenesis and metastasis of hepatocellular carcinoma.
Liu L, Gu M, Ma J, Wang Y, Li M, Wang H, Yin X, Li X. Liu L, et al. Mol Cancer. 2022 Jul 20;21(1):149. doi: 10.1186/s12943-022-01619-4. Mol Cancer. 2022. PMID: 35858900 Free PMC article.
The role of circGPR137B in HCC was estimated in vitro and in vivo. RT-qPCR, western blot, m(6)A dot blot, RIP, MeRIP and dual-luciferase reporter assays were used to validate the reciprocal regulation of the feedback loop among circGPR137B, miR-4739 and m(6)A …
The role of circGPR137B in HCC was estimated in vitro and in vivo. RT-qPCR, western blot, m(6)A dot blot, RIP, MeRIP and dual-lucifer …
m(6)A modification of circSPECC1 suppresses RPE oxidative damage and maintains retinal homeostasis.
Chen X, Wang Y, Wang JN, Cao QC, Sun RX, Zhu HJ, Zhang YR, Ji JD, Liu QH. Chen X, et al. Cell Rep. 2022 Nov 15;41(7):111671. doi: 10.1016/j.celrep.2022.111671. Cell Rep. 2022. PMID: 36384115 Free article.
Mechanically, nuclear export of circSPECC1 transcript depends on its N(6)-methyladenosine (m(6)A) level with YTHDC1 as the reader. CircSPECC1 directly sponges miR-145-5p to block its interaction with CDKN1A. ...Collectively, circSPECC1, mediated by m(6)A modi …
Mechanically, nuclear export of circSPECC1 transcript depends on its N(6)-methyladenosine (m(6)A) level with YTHDC1 as the reader. Ci …
M(7)G modification of FTH1 and pri-miR-26a regulates ferroptosis and chemotherapy resistance in osteosarcoma.
He M, Wang Y, Xie J, Pu J, Shen Z, Wang A, Li T, Wang T, Li G, Liu Y, Mei Z, Ren Z, Wang W, Liu X, Hong J, Liu Q, Lei H, He X, Du W, Yuan Y, Yang L. He M, et al. Oncogene. 2024 Jan;43(5):341-353. doi: 10.1038/s41388-023-02882-5. Epub 2023 Dec 1. Oncogene. 2024. PMID: 38040806
We then identified primary (pri)-miR-26a and pri-miR-98 in the Top 20 m(7)G-methylated pri-miRNAs with highly conserved species. Further results confirmed that METTL1 enhances cell ferroptosis by targeting FTH1 and primary (pri)-miR-26a, promoting thei …
We then identified primary (pri)-miR-26a and pri-miR-98 in the Top 20 m(7)G-methylated pri-miRNAs with highly conserved …
MiR-155 regulates m(6)A level and cell progression by targeting FTO in clear cell renal cell carcinoma.
Yang W, Xie L, Wang P, Zhuang C. Yang W, et al. Cell Signal. 2022 Mar;91:110217. doi: 10.1016/j.cellsig.2021.110217. Epub 2021 Dec 16. Cell Signal. 2022. PMID: 34921979
Here we show that miR-155 directly targets FTO to negatively regulate its expression and increased m(6)A level in ccRCC. ...Moreover, cell function assays, xenografts assays and m(6)A dot blot assays revealed that overexpression of miR-155 enhanced tum …
Here we show that miR-155 directly targets FTO to negatively regulate its expression and increased m(6)A level in ccRCC. ...Mo …
MiR-103-3p targets the m(6) A methyltransferase METTL14 to inhibit osteoblastic bone formation.
Sun Z, Wang H, Wang Y, Yuan G, Yu X, Jiang H, Wu Q, Yang B, Hu Z, Shi F, Cao X, Zhang S, Guo T, Zhao J. Sun Z, et al. Aging Cell. 2021 Feb;20(2):e13298. doi: 10.1111/acel.13298. Epub 2021 Jan 13. Aging Cell. 2021. PMID: 33440070 Free PMC article.
Additionally, miR-103-3p directly targeted Mettl14 to inhibit osteoblast activity, and METTL14-dependent N(6) -methyladenosine (m(6) A) methylation inhibited miR-103-3p processing by the microprocessor protein DGCR8 and promoted osteoblast activity. ...Collec …
Additionally, miR-103-3p directly targeted Mettl14 to inhibit osteoblast activity, and METTL14-dependent N(6) -methyladenosine (m …
WTAP-mediated m(6)A modification modulates bone marrow mesenchymal stem cells differentiation potential and osteoporosis.
You Y, Liu J, Zhang L, Li X, Sun Z, Dai Z, Ma J, Jiao G, Chen Y. You Y, et al. Cell Death Dis. 2023 Jan 17;14(1):33. doi: 10.1038/s41419-023-05565-x. Cell Death Dis. 2023. PMID: 36650131 Free PMC article.
Mechanistically, the expression of WTAP increased during osteogenic differentiation and significantly promoted pri-miR-181a and pri-miR-181c methylation, which was recognized by YTHDC1, and increased the maturation to miR-181a and miR-181c. MiR- …
Mechanistically, the expression of WTAP increased during osteogenic differentiation and significantly promoted pri-miR-181a and pri- …
m(6)A-modified miR-143-3p inhibits epithelial mesenchymal transition in bronchial epithelial cells and extracellular matrix production in lung fibroblasts by targeting Smad3.
Wang J, Jian Q, Yan K, Yang J, Yan L, Cheng W. Wang J, et al. Pulm Pharmacol Ther. 2023 Dec;83:102251. doi: 10.1016/j.pupt.2023.102251. Epub 2023 Sep 2. Pulm Pharmacol Ther. 2023. PMID: 37666296
EMT and ECM proteins were detected by western blot, RT-qPCR, and ELISA. To determine the level of miR-143-3p m(6)A methylation, we employed the meRIP-qPCR assay. Additionally, the binding of miR-143-3p with Smad3 were projected by bioinformatics and validated …
EMT and ECM proteins were detected by western blot, RT-qPCR, and ELISA. To determine the level of miR-143-3p m(6)A methylation …
As3MT via consuming SAM is involved in arsenic-induced nonalcoholic fatty liver disease by blocking m(6)A-mediated miR-142-5p maturation.
Li H, Wu L, Ye F, Wang D, Wang L, Li W, Xu Y, Li Z, Zhang J, Wang S, Zhang A, Liu Q. Li H, et al. Sci Total Environ. 2023 Sep 20;892:164746. doi: 10.1016/j.scitotenv.2023.164746. Epub 2023 Jun 8. Sci Total Environ. 2023. PMID: 37301390
Here, we found that chronic exposure to environmental-related doses of arsenic disturbed fatty acid and methionine metabolism in mice, caused liver steatosis, increased arsenic (3) methyltransferase (As3MT), sterol regulatory element binding protein 1 (SREBP1) and lipogenic gene …
Here, we found that chronic exposure to environmental-related doses of arsenic disturbed fatty acid and methionine metabolism in mice, cause …
Suxiao Jiuxin Pill alleviates myocardial ischemia/reperfusion-induced autophagy via miR-193a-3p/ALKBH5 pathway.
Wang D, Wang D, Jin Q, Wang X. Wang D, et al. Phytomedicine. 2024 Mar;125:155359. doi: 10.1016/j.phymed.2024.155359. Epub 2024 Jan 24. Phytomedicine. 2024. PMID: 38301300 Free article.
ALKBH5, a target gene of miR-193, is negatively regulated by miR-193a-3p. Upon overexpression of miR-193a-3p or silencing of ALKBH5, m(6)A methylation decreased, and the autophagy-attenuating effects of SJP and its components, senkyunolide A and l-born …
ALKBH5, a target gene of miR-193, is negatively regulated by miR-193a-3p. Upon overexpression of miR-193a-3p or silenci …
RNA m(6)A reader YTHDF2 facilitates precursor miR-126 maturation to promote acute myeloid leukemia progression.
Zhang Z, Zhou K, Han L, Small A, Xue J, Huang H, Weng H, Su R, Tan B, Shen C, Li W, Zhao Z, Qing Y, Qin X, Wang K, Leung K, Boldin M, Chen CW, Ann D, Qian Z, Deng X, Chen J, Chen Z. Zhang Z, et al. Genes Dis. 2023 Mar 28;11(1):382-396. doi: 10.1016/j.gendis.2023.01.016. eCollection 2024 Jan. Genes Dis. 2023. PMID: 37588203 Free PMC article.
Here, we found that YTHDF2 is aberrantly overexpressed in acute myeloid leukemia (AML) patients, especially in relapsed patients, and plays an oncogenic role in AML. Moreover, YTHDF2 promotes expression of miR-126-3p (also known as miR-126, as it is the main product …
Here, we found that YTHDF2 is aberrantly overexpressed in acute myeloid leukemia (AML) patients, especially in relapsed patients, and plays …
5,585 results