Skip to main page content
Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

My NCBI Filters
Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1986 2
1988 1
1989 2
1990 1
1991 5
1992 3
1993 5
1994 5
1995 5
1996 3
1997 3
1998 5
1999 6
2000 6
2001 1
2002 3
2003 2
2004 5
2005 3
2006 5
2007 2
2008 2
2009 7
2010 5
2011 6
2012 3
2013 5
2014 4
2015 2
2016 2
2018 1
2019 2
2020 3
2021 1
2022 2
Text availability
Article attribute
Article type
Publication date

Search Results

109 results
Results by year

Citations

1 article found by citation matching

Search results

Filters applied: . Clear all
Page 1
Activation of cultured vascular endothelial cells by antiphospholipid antibodies.
Simantov R, LaSala JM, Lo SK, Gharavi AE, Sammaritano LR, Salmon JE, Silverstein RL. Simantov R, et al. J Clin Invest. 1995 Nov;96(5):2211-9. doi: 10.1172/JCI118276. J Clin Invest. 1995. PMID: 7593607 Free PMC article.
We have demonstrated the activation of cultured human umbilical vein endothelial cells (HUVEC) by IgG from patients with anticardiolipin antibodies (aCL). ...These data support the hypothesis that aPL activate vascular endotheli
We have demonstrated the activation of cultured human umbilical vein endothelial cells (HUVEC) by IgG from patie …
Endothelial cell activation by antiphospholipid antibodies is modulated by Kruppel-like transcription factors.
Allen KL, Hamik A, Jain MK, McCrae KR. Allen KL, et al. Blood. 2011 Jun 9;117(23):6383-91. doi: 10.1182/blood-2010-10-313072. Epub 2011 Apr 11. Blood. 2011. PMID: 21482710 Free PMC article.
The majority of APLAs are directed against phospholipid-binding proteins, particularly beta2-glycoprotein I (beta2GPI). Anti-beta2GPI antibodies activate endothelial cells in a beta2GPI-dependent manner through a pathway that involves NF-kappaB. Kruppe …
The majority of APLAs are directed against phospholipid-binding proteins, particularly beta2-glycoprotein I (beta2GPI). Anti-beta2GPI ant
Antiphospholipid antibodies prolong the activation of endothelial cells induced by necrotic trophoblastic debris: implications for the pathogenesis of preeclampsia.
Chen Q, Guo F, Hensby-Bennett S, Stone P, Chamley L. Chen Q, et al. Placenta. 2012 Oct;33(10):810-5. doi: 10.1016/j.placenta.2012.07.019. Epub 2012 Aug 16. Placenta. 2012. PMID: 22902008
Antiphospholipid antibodies alone did not active untreated endothelial cells, but did prolong the activation of endothelial cells which had been activated by pre-treatment with necrotic trophoblastic debris. When expo
Antiphospholipid antibodies alone did not active untreated endothelial cells, but did prolong the acti
Development of a thrombin generation test in cultured endothelial cells: Evaluation of the prothrombotic effects of antiphospholipid antibodies.
Billoir P, Miranda S, Damian L, Richard V, Benhamou Y, Le Cam Duchez V. Billoir P, et al. Thromb Res. 2018 Sep;169:87-92. doi: 10.1016/j.thromres.2018.07.021. Epub 2018 Jul 17. Thromb Res. 2018. PMID: 30031291
INTRODUCTION: Circulating antiphospholipid antibodies (APL) induce vascular injury and endothelial dysfunction, which are associated with thrombotic events and/or fetal loss. ...Endothelial cells were incubated with the purified anti-beta …
INTRODUCTION: Circulating antiphospholipid antibodies (APL) induce vascular injury and endothelial dysfunction, …
Characterization of Antiphospholipid Syndrome Atherothrombotic Risk by Unsupervised Integrated Transcriptomic Analyses.
Pérez-Sánchez L, Patiño-Trives AM, Aguirre-Zamorano MÁ, Luque-Tévar M, Ábalos-Aguilera MC, Arias-de la Rosa I, Seguí P, Velasco-Gimena F, Barbarroja N, Escudero-Contreras A, Collantes-Estévez E, Pérez-Sánchez C, López-Pedrera C. Pérez-Sánchez L, et al. Arterioscler Thromb Vasc Biol. 2021 Feb;41(2):865-877. doi: 10.1161/ATVBAHA.120.315346. Epub 2020 Dec 24. Arterioscler Thromb Vasc Biol. 2021. PMID: 33356391 Free article.
Transfection studies and luciferase assays established the relationship between specific miRNAs and their identified target genes and proteins, along with their involvement in the regulation of monocytes procoagulant activity and cell adhesion. Correlation analyses showed …
Transfection studies and luciferase assays established the relationship between specific miRNAs and their identified target genes and protei …
Toll-like receptor 2 mediates the activation of human monocytes and endothelial cells by antiphospholipid antibodies.
Satta N, Kruithof EK, Fickentscher C, Dunoyer-Geindre S, Boehlen F, Reber G, Burger D, de Moerloose P. Satta N, et al. Blood. 2011 May 19;117(20):5523-31. doi: 10.1182/blood-2010-11-316158. Epub 2011 Feb 17. Blood. 2011. PMID: 21330474 Free article.
The proposed pathogenic mechanisms for aPLA effects include the inflammatory activation of monocytes and endothelial cells. Toll-like receptors (TLRs) are candidate signaling intermediates. ...In HUVECs, inflammatory responses to these aPLAs were increased by …
The proposed pathogenic mechanisms for aPLA effects include the inflammatory activation of monocytes and endothelial cells
Antiphospholipid-mediated thrombosis: interplay between anticardiolipin antibodies and vascular cells.
López-Pedrera C, Buendía P, Barbarroja N, Siendones E, Velasco F, Cuadrado MJ. López-Pedrera C, et al. Clin Appl Thromb Hemost. 2006 Jan;12(1):41-5. doi: 10.1177/107602960601200107. Clin Appl Thromb Hemost. 2006. PMID: 16444433 Free article. Review.
Different theories have been proposed, including the effect of aPL on endothelial cells, monocytes, and platelets. aPL are able to recognize, injure, or activate cultured vascular endothelial cells. Cultured endothelial
Different theories have been proposed, including the effect of aPL on endothelial cells, monocytes, and platelets. aPL are abl …
A novel pathway for human endothelial cell activation by antiphospholipid/anti-β2 glycoprotein I antibodies.
Allen KL, Fonseca FV, Betapudi V, Willard B, Zhang J, McCrae KR. Allen KL, et al. Blood. 2012 Jan 19;119(3):884-93. doi: 10.1182/blood-2011-03-344671. Epub 2011 Nov 21. Blood. 2012. PMID: 22106343 Free PMC article.
Antiphospholipid Abs (APLAs) are associated with thrombosis and recurrent fetal loss. These Abs are primarily directed against phospholipid-binding proteins, particularly beta(2)GPI, and activate endothelial cells (ECs) in a beta(2)GPI-dependent manner
Antiphospholipid Abs (APLAs) are associated with thrombosis and recurrent fetal loss. These Abs are primarily directed against phosph
Endothelial microparticles in diseases.
Chironi GN, Boulanger CM, Simon A, Dignat-George F, Freyssinet JM, Tedgui A. Chironi GN, et al. Cell Tissue Res. 2009 Jan;335(1):143-51. doi: 10.1007/s00441-008-0710-9. Epub 2008 Nov 7. Cell Tissue Res. 2009. PMID: 18989704 Review.
Microparticles of various cellular origin (platelets, leukocytes, endothelial cells) are found in the plasma of healthy subjects, and their amount increases under pathological conditions. Endothelial microparticles (EMP) not only constitute an emerging marker …
Microparticles of various cellular origin (platelets, leukocytes, endothelial cells) are found in the plasma of healthy subjec …
The influence of anti-endothelial/antiphospholipid antibodies on fibrin formation and lysis on endothelial cells.
Patterson AM, Ford I, Graham A, Booth NA, Greaves M. Patterson AM, et al. Br J Haematol. 2006 May;133(3):323-30. doi: 10.1111/j.1365-2141.2006.05994.x. Br J Haematol. 2006. PMID: 16643435
The prothrombotic mechanisms associated with antiphospholipid antibodies remain incompletely defined. Antibody binding to endothelial cells in vitro is a feature of antiphospholipid antibody-positive sera. ...We confirmed antibody binding to …
The prothrombotic mechanisms associated with antiphospholipid antibodies remain incompletely defined. Antibody binding to e
109 results