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1993 1
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Comparative study of cell cycle kinetics and induction of apoptosis or necrosis after exposure of human Mono Mac 6 cells to radiofrequency radiation.
Lantow M, Viergutz T, Weiss DG, Simkó M. Lantow M, et al. Radiat Res. 2006 Sep;166(3):539-43. doi: 10.1667/RR3601.1. Radiat Res. 2006. PMID: 16953672
The possible harmful effects of radiofrequency electromagnetic fields (RF EMFs) are controversial. We have used human Mono Mac 6 cells to investigate the influence of RF EMFs in vitro on cell cycle alterations and Brd …
The possible harmful effects of radiofrequency electromagnetic fields (RF EMFs) are controversial. We have used human
Stathmo-apoptosis: arresting apoptosis by fluorochrome-labeled inhibitor of caspases.
Smolewski P, Grabarek J, Phelps DJ, Darzynkiewicz Z. Smolewski P, et al. Int J Oncol. 2001 Oct;19(4):657-63. doi: 10.3892/ijo.19.4.657. Int J Oncol. 2001. PMID: 11562738
Apoptosis, like mitosis, is a kinetic event. The entire duration of apoptosis, from its onset to total disintegration of the cell, is often short and may be of variable duration. ...The present approach offers a unique capability to accurately estimate
Apoptosis, like mitosis, is a kinetic event. The entire duration of apoptosis, from its onset to total disintegration o
Onset of hepatocarcinogen-specific cell proliferation and cell cycle aberration during the early stage of repeated hepatocarcinogen administration in rats.
Kimura M, Abe H, Mizukami S, Tanaka T, Itahashi M, Onda N, Yoshida T, Shibutani M. Kimura M, et al. J Appl Toxicol. 2016 Feb;36(2):223-37. doi: 10.1002/jat.3163. Epub 2015 May 22. J Appl Toxicol. 2016. PMID: 26011634
We have previously reported that a 28-day treatment of carcinogens evoking target cell proliferation activates G1 /S checkpoint function and apoptosis, as well as induction of aberrant ubiquitin D (Ubd) expression, suggesting disruptive spindle checkpo …
We have previously reported that a 28-day treatment of carcinogens evoking target cell proliferation activates G1 /S checkpoin …
B16 and CLS91 mouse melanoma cells susceptibility to apoptosis after vincristin treatment in vitro.
Olszewska-Słonina D, Drewa T, Styczyński J, Olszewski K, Musiałkiewicz D. Olszewska-Słonina D, et al. Acta Pol Pharm. 2004 Mar-Apr;61(2):113-21. Acta Pol Pharm. 2004. PMID: 15493293 Free article.
Determination of vincristin's influence on cell proliferation kinetics, cell cycle progression based on DNA content and percentage of apoptosis and necrosis in B16 and ClS91 cells, was the object of the present study. …
Determination of vincristin's influence on cell proliferation kinetics, cell cycle progression based on D …
Mechanisms for 2-methoxyestradiol-induced apoptosis of prostate cancer cells.
Bu S, Blaukat A, Fu X, Heldin NE, Landström M. Bu S, et al. FEBS Lett. 2002 Nov 6;531(2):141-51. doi: 10.1016/s0014-5793(02)03478-6. FEBS Lett. 2002. PMID: 12417302 Free article.
Here, we report that 2-methoxyestradiol (2-ME), an endogenous metabolite of estrogen that does not bind to nuclear estrogen receptors, effectively induces apoptosis in Bcl-2-expressing human prostate and breast carcinoma cells in vitro and in a rat prostate t …
Here, we report that 2-methoxyestradiol (2-ME), an endogenous metabolite of estrogen that does not bind to nuclear estrogen receptors, effec …
The uptake kinetics and immunotoxic effects of microcystin-LR in human and chicken peripheral blood lymphocytes in vitro.
Lankoff A, Carmichael WW, Grasman KA, Yuan M. Lankoff A, et al. Toxicology. 2004 Nov 1;204(1):23-40. doi: 10.1016/j.tox.2004.05.016. Toxicology. 2004. PMID: 15369846
Peripheral blood lymphocytes were treated with microcystin-LR at environmentally relevant doses of 1, 10 and 25 microg/ml for 12, 24, 48, 72 h (for proliferation assay cells were treated for 72 h). T-cell and B-cell proliferation as well as apoptosis a …
Peripheral blood lymphocytes were treated with microcystin-LR at environmentally relevant doses of 1, 10 and 25 microg/ml for 12, 24, 48, 72 …
Exposure to continuous bromodeoxyuridine (BrdU) differentially affects cell cycle progression of human breast and bladder cancer cell lines.
Diermeier S, Schmidt-Bruecken E, Kubbies M, Kunz-Schughart LA, Brockhoff G. Diermeier S, et al. Cell Prolif. 2004 Apr;37(2):195-206. doi: 10.1111/j.1365-2184.2004.00296.x. Cell Prolif. 2004. PMID: 15030553 Free PMC article.
Both uni- and bi-parametric DNA measurements were performed to identify BrdU-induced alterations in the S-phase fraction and in cell cycle progression. An annexinV/propidium iodide (PI) assay was used to identify potential induction of apoptosis
Both uni- and bi-parametric DNA measurements were performed to identify BrdU-induced alterations in the S-phase fraction and in ce
Relationships between genomic, cell cycle, and mutagenic responses of TK6 cells exposed to DNA damaging chemicals.
Islaih M, Halstead BW, Kadura IA, Li B, Reid-Hubbard JL, Flick L, Altizer JL, Thom Deahl J, Monteith DK, Newton RK, Watson DE. Islaih M, et al. Mutat Res. 2005 Oct 15;578(1-2):100-16. doi: 10.1016/j.mrfmmm.2005.04.012. Mutat Res. 2005. PMID: 16109433
To better understand this interplay, we investigated cytotoxicty, mutagenesis, cell cycle profile, and global gene expression in the human TK6 lymphoblastoid cell line exposed to six genotoxicants. ...Cells exposed to all compounds experienced …
To better understand this interplay, we investigated cytotoxicty, mutagenesis, cell cycle profile, and global gene expression …
Comparison of burst of reactive oxygen species and activation of caspase-3 in apoptosis of K562 and HL-60 cells induced by docetaxel.
Cao DX, Qiao B, Ge ZQ, Yuan YJ. Cao DX, et al. Cancer Lett. 2004 Oct 8;214(1):103-13. doi: 10.1016/j.canlet.2004.03.047. Cancer Lett. 2004. PMID: 15331178
Apoptosis-resistant K562 cells and apoptosis-proficient HL-60 acute myelomonocytic leukemia cells were selected to study the cell-type-specific characteristics of docetaxel. ...After treatment with 10(-8)M docetaxel, DNA fragmentation and
Apoptosis-resistant K562 cells and apoptosis-proficient HL-60 acute myelomonocytic leukemia cells were selected
Expression of a non-functional p53 affects the sensitivity of cancer cells to gemcitabine.
Galmarini CM, Clarke ML, Falette N, Puisieux A, Mackey JR, Dumontet C. Galmarini CM, et al. Int J Cancer. 2002 Feb 1;97(4):439-45. doi: 10.1002/ijc.1628. Int J Cancer. 2002. PMID: 11802204 Free article.
Gemcitabine induced an S-phase arrest in both cell lines. A more pronounced block in G1 phase, however, was observed in MN1 cells. Exposure to gemcitabine induced a higher degree of apoptosis in MN-1 than in MDD2 cells. ...
Gemcitabine induced an S-phase arrest in both cell lines. A more pronounced block in G1 phase, however, was observed in MN1 …
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