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1989 1
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1995 4
1996 3
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1998 2
2000 3
2001 2
2002 5
2003 2
2004 3
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2006 1
2007 5
2008 4
2009 3
2010 3
2011 1
2012 2
2013 1
2014 5
2015 2
2017 1
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Novel repair mechanisms in a renal ischaemia/reperfusion model: Subsequent saxagliptin treatment modulates the pro-angiogenic GLP-1/cAMP/VEGF, ANP/eNOS/NO, SDF-1α/CXCR4, and Kim-1/STAT3/HIF-1α/VEGF/eNOS pathways.
Kamel NM, Abd El Fattah MA, El-Abhar HS, Abdallah DM. Kamel NM, et al. Eur J Pharmacol. 2019 Oct 15;861:172620. doi: 10.1016/j.ejphar.2019.172620. Epub 2019 Aug 19. Eur J Pharmacol. 2019. PMID: 31437429
By inhibiting DPP-4, saxagliptin maintained stromal cell-derived factor-1alpha expression and upregulated its chemokine receptor CXCR4 to trigger vasculogenesis through the enhanced migration of endothelial progenitor cells (EPCs). Additionally, this compound …
By inhibiting DPP-4, saxagliptin maintained stromal cell-derived factor-1alpha expression and upregulated its chemokine receptor CXCR …
Cell Type-Specific Roles of NF-κB Linking Inflammation and Thrombosis.
Mussbacher M, Salzmann M, Brostjan C, Hoesel B, Schoergenhofer C, Datler H, Hohensinner P, Basílio J, Petzelbauer P, Assinger A, Schmid JA. Mussbacher M, et al. Front Immunol. 2019 Feb 4;10:85. doi: 10.3389/fimmu.2019.00085. eCollection 2019. Front Immunol. 2019. PMID: 30778349 Free PMC article. Review.
The response of endothelial cells to inflammation and NF-kappaB activation is characterized by the induction of adhesion molecules promoting binding and transmigration of leukocytes, while simultaneously increasing their thrombogenic potential. ...M
The response of endothelial cells to inflammation and NF-kappaB activation is characterized by the induction of adhe
STING-IRF3 Triggers Endothelial Inflammation in Response to Free Fatty Acid-Induced Mitochondrial Damage in Diet-Induced Obesity.
Mao Y, Luo W, Zhang L, Wu W, Yuan L, Xu H, Song J, Fujiwara K, Abe JI, LeMaire SA, Wang XL, Shen YH. Mao Y, et al. Arterioscler Thromb Vasc Biol. 2017 May;37(5):920-929. doi: 10.1161/ATVBAHA.117.309017. Epub 2017 Mar 16. Arterioscler Thromb Vasc Biol. 2017. PMID: 28302626 Free PMC article.
In the present study, we sought to determine the role of STING in palmitic acid-induced endothelial activation/inflammation. APPROACH AND RESULTS: In cultured endothelial cells, palmitic acid treatment activated STING, as indicated by its perinuclear transloc …
In the present study, we sought to determine the role of STING in palmitic acid-induced endothelial activation/inflammation. APPROACH …
Induction of tissue factor on monocytes by adhesion to endothelial cells.
Lo SK, Cheung A, Zheng Q, Silverstein RL. Lo SK, et al. J Immunol. 1995 May 1;154(9):4768-77. J Immunol. 1995. PMID: 7536780
Activated monocytes express tissue factor (TF), a protein that is important in the pathogenesis of thrombotic disorders. We sought to characterize an adhesion-dependent pathway for monocyte activation. In this study, we showed that adhesion
Activated monocytes express tissue factor (TF), a protein that is important in the pathogenesis of thrombotic disorders …
Interaction of endothelial microparticles with monocytic cells in vitro induces tissue factor-dependent procoagulant activity.
Sabatier F, Roux V, Anfosso F, Camoin L, Sampol J, Dignat-George F. Sabatier F, et al. Blood. 2002 Jun 1;99(11):3962-70. doi: 10.1182/blood.v99.11.3962. Blood. 2002. PMID: 12010795
Using flow cytometry, we demonstrated that EMPs express adhesive receptors similar to those expressed by activated endothelial cells. ...Concomitantly, THP-1 exhibited increased levels of tissue factor (TF) antigen and TF mRNA compared to contro …
Using flow cytometry, we demonstrated that EMPs express adhesive receptors similar to those expressed by activated endothelial
Cholesterol-induced membrane microvesicles as novel carriers of damage-associated molecular patterns: mechanisms of formation, action, and detoxification.
Liu ML, Scalia R, Mehta JL, Williams KJ. Liu ML, et al. Arterioscler Thromb Vasc Biol. 2012 Sep;32(9):2113-21. doi: 10.1161/ATVBAHA.112.255471. Epub 2012 Jul 19. Arterioscler Thromb Vasc Biol. 2012. PMID: 22814745 Free PMC article.
OBJECTIVE: Cholesterol enrichment occurs in vivo when phagocytes ingest retained and aggregated lipoproteins, damaged or senescent cells, and related debris. We previously reported that enrichment of human monocyte/macrophages with unesterified cholesterol (UC) trig …
OBJECTIVE: Cholesterol enrichment occurs in vivo when phagocytes ingest retained and aggregated lipoproteins, damaged or senescent cells
Effect of porcine endothelial tissue factor pathway inhibitor on human coagulation factors.
Kopp CW, Siegel JB, Hancock WW, Anrather J, Winkler H, Geczy CL, Kaczmarek E, Bach FH, Robson SC. Kopp CW, et al. Transplantation. 1997 Mar 15;63(5):749-58. doi: 10.1097/00007890-199703150-00023. Transplantation. 1997. PMID: 9075849
BACKGROUND: Delayed xenograft rejection (DXR) is characterized by inflammation and vascular thrombosis. Activation of coagulation may occur as a result of tissue factor (TF) expression on both activated donor endothelial cells (EC) and recipient infilt …
BACKGROUND: Delayed xenograft rejection (DXR) is characterized by inflammation and vascular thrombosis. Activation of coagulation may occur …
Induction of tissue factor production but not the upregulation of adhesion molecule expression by ceramide in human vascular endothelial cells.
Hirokawa M, Kitabayashi A, Kuroki J, Miura AB. Hirokawa M, et al. Tohoku J Exp Med. 2000 Jul;191(3):167-76. doi: 10.1620/tjem.191.167. Tohoku J Exp Med. 2000. PMID: 10997557 Free article.
We have investigated the role for the sphingomyelin/ceramide pathway in the TNF-alpha-induced upregulation of adhesion molecule expression and tissue factor production of human endothelial cells. TNF-alpha stimulated human umbilical vascular …
We have investigated the role for the sphingomyelin/ceramide pathway in the TNF-alpha-induced upregulation of adhesion molecule expre …
Estrogenic and anti-inflammatory effects of pseudoprotodioscin in atherosclerosis-prone mice: Insights into endothelial cells and perivascular adipose tissues.
Sun B, Yang D, Yin YZ, Xiao J. Sun B, et al. Eur J Pharmacol. 2020 Feb 15;869:172887. doi: 10.1016/j.ejphar.2019.172887. Epub 2019 Dec 23. Eur J Pharmacol. 2020. PMID: 31877277
Molecular docking analysis suggested a selective interaction of PPD with ERalpha. Upon PPD treatment, ERalpha and endothelial nitric oxide synthase (eNOS) protein levels were increased, whereas cell adhesion molecule and monocyte chemoattractant protein-1 (MC …
Molecular docking analysis suggested a selective interaction of PPD with ERalpha. Upon PPD treatment, ERalpha and endothelial nitric …
Curcumin (Diferuloylmethane) inhibition of tumor necrosis factor (TNF)-mediated adhesion of monocytes to endothelial cells by suppression of cell surface expression of adhesion molecules and of nuclear factor-kappaB activation.
Kumar A, Dhawan S, Hardegen NJ, Aggarwal BB. Kumar A, et al. Biochem Pharmacol. 1998 Mar 15;55(6):775-83. doi: 10.1016/s0006-2952(97)00557-1. Biochem Pharmacol. 1998. PMID: 9586949
In the present study, we investigated the effect of curcumin, an antiinflammatory agent, on the adhesion of monocytes to human umbilical vein endothelial cells (EC). Treatment of EC with tumor necrosis factor (TNF) for 6 hr augmented the adhe
In the present study, we investigated the effect of curcumin, an antiinflammatory agent, on the adhesion of monocytes to human …
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