Skip to main page content
Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

My NCBI Filters
Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1985 1
1997 1
2001 1
2002 1
2005 1
2006 1
2007 1
2008 1
2009 1
2010 2
2011 2
2012 4
2013 1
2014 1
2015 2
2016 5
2017 3
2018 1
2019 5
2020 4
Text availability
Article attribute
Article type
Publication date

Search Results

34 results
Results by year

Citations

1 article found by citation matching

Search results

Filters applied: . Clear all
Page 1
Infiltrating monocytes in liver injury and repair.
Brempelis KJ, Crispe IN. Brempelis KJ, et al. Clin Transl Immunology. 2016 Nov 4;5(11):e113. doi: 10.1038/cti.2016.62. eCollection 2016 Nov. Clin Transl Immunology. 2016. PMID: 27990288 Free PMC article. Review.
Bone marrow-derived monocytes infiltrate the damaged liver tissue and are a critical component of the innate immune response that may drive injury resolution or host death in the short term or chronic inflammation, fibrosis and hepatocellular carcinoma …
Bone marrow-derived monocytes infiltrate the damaged liver tissue and are a critical component of the innate immune res …
Targeting hepatic macrophages to treat liver diseases.
Tacke F. Tacke F. J Hepatol. 2017 Jun;66(6):1300-1312. doi: 10.1016/j.jhep.2017.02.026. Epub 2017 Mar 4. J Hepatol. 2017. PMID: 28267621 Review.
This fosters the infiltration of monocytes into the liver, which give rise to large numbers of inflammatory monocyte-derived macrophages. ...If liver injury ceases, specific molecular signals trigger hepatic macrophages to switch their ph …
This fosters the infiltration of monocytes into the liver, which give rise to large numbers of inflammatory monocyte
Role of macrophages in experimental liver injury and repair in mice.
Dong X, Liu J, Xu Y, Cao H. Dong X, et al. Exp Ther Med. 2019 May;17(5):3835-3847. doi: 10.3892/etm.2019.7450. Epub 2019 Mar 27. Exp Ther Med. 2019. PMID: 31007731 Free PMC article. Review.
Subsequently, inflammatory monocytes accumulate in the liver via chemokine-chemokine receptor interactions, resulting in massive inflammatory MoMo infiltration. ...The present review primarily focuses on the diverse functions of macrophages in experimental …
Subsequently, inflammatory monocytes accumulate in the liver via chemokine-chemokine receptor interactions, resulting in massi …
Inflammatory pathways in alcoholic steatohepatitis.
Gao B, Ahmad MF, Nagy LE, Tsukamoto H. Gao B, et al. J Hepatol. 2019 Feb;70(2):249-259. doi: 10.1016/j.jhep.2018.10.023. J Hepatol. 2019. PMID: 30658726 Free PMC article. Review.
Herein, we review the roles of multiple cell types that are involved in inflammation in ASH, including resident macrophages and infiltrating monocytes, as well as other cell types in the innate and adaptive immune system. ...We highlight the concept that inflammatio …
Herein, we review the roles of multiple cell types that are involved in inflammation in ASH, including resident macrophages and infiltrat
Chemokine (C-C motif) receptor 2-positive monocytes aggravate the early phase of acetaminophen-induced acute liver injury.
Mossanen JC, Krenkel O, Ergen C, Govaere O, Liepelt A, Puengel T, Heymann F, Kalthoff S, Lefebvre E, Eulberg D, Luedde T, Marx G, Strassburg CP, Roskams T, Trautwein C, Tacke F. Mossanen JC, et al. Hepatology. 2016 Nov;64(5):1667-1682. doi: 10.1002/hep.28682. Epub 2016 Jul 22. Hepatology. 2016. PMID: 27302828
Influx of MoMFs is dependent on the chemokine receptor, chemokine (C-C motif) receptor 2 (CCR2), given that Ccr2(-/-) mice display reduced infiltration of monocytes and attenuated liver injury post-APAP overdose at early time points. ...CCR2(+) MoMFs e …
Influx of MoMFs is dependent on the chemokine receptor, chemokine (C-C motif) receptor 2 (CCR2), given that Ccr2(-/-) mice display reduced …
Combination of sivelestat and N-acetylcysteine alleviates the inflammatory response and exceeds standard treatment for acetaminophen-induced liver injury.
Raevens S, Van Campenhout S, Debacker PJ, Lefere S, Verhelst X, Geerts A, Van Vlierberghe H, Colle I, Devisscher L. Raevens S, et al. J Leukoc Biol. 2020 Feb;107(2):341-355. doi: 10.1002/JLB.5A1119-279R. Epub 2019 Dec 16. J Leukoc Biol. 2020. PMID: 31841237
Hepatocyte death during acetaminophen (APAP) intoxication elicits a reactive inflammatory response, with hepatic recruitment of neutrophils and monocytes, which further aggravates liver injury. Neutrophil elastase (NE), secreted by activated neutrophils, carr …
Hepatocyte death during acetaminophen (APAP) intoxication elicits a reactive inflammatory response, with hepatic recruitment of neutrophils …
Identification and characterization of infiltrating macrophages in acetaminophen-induced liver injury.
Holt MP, Cheng L, Ju C. Holt MP, et al. J Leukoc Biol. 2008 Dec;84(6):1410-21. doi: 10.1189/jlb.0308173. Epub 2008 Aug 19. J Leukoc Biol. 2008. PMID: 18713872 Free PMC article.
The role of macrophages in the pathogenesis of acetaminophen (APAP)-induced liver injury remains controversial, as it has been demonstrated that these cells display pro-toxicant and hepato-protective functions. ...The data demonstrated that the IMs were derived from …
The role of macrophages in the pathogenesis of acetaminophen (APAP)-induced liver injury remains controversial, as it has been …
Serelaxin induces Notch1 signaling and alleviates hepatocellular damage in orthotopic liver transplantation.
Kageyama S, Nakamura K, Ke B, Busuttil RW, Kupiec-Weglinski JW. Kageyama S, et al. Am J Transplant. 2018 Jul;18(7):1755-1763. doi: 10.1111/ajt.14706. Epub 2018 Mar 23. Am J Transplant. 2018. PMID: 29464890 Free PMC article.
Liver ischemia-reperfusion injury (IRI) represents a risk factor for early graft dysfunction and an obstacle to expanding donor pool in orthotopic liver transplantation (OLT). ...Our study documents the efficacy of SER-Notch1 signaling in mouse OLT and highli
Liver ischemia-reperfusion injury (IRI) represents a risk factor for early graft dysfunction and an obstacle to expanding dono
Prevention of severe toxic liver injury and oxidative stress in MCP-1-deficient mice.
Zamara E, Galastri S, Aleffi S, Petrai I, Aragno M, Mastrocola R, Novo E, Bertolani C, Milani S, Vizzutti F, Vercelli A, Pinzani M, Laffi G, LaVilla G, Parola M, Marra F. Zamara E, et al. J Hepatol. 2007 Feb;46(2):230-8. doi: 10.1016/j.jhep.2006.09.007. Epub 2006 Oct 23. J Hepatol. 2007. PMID: 17125873
BACKGROUND/AIMS: Administration of carbon tetrachloride determines liver injury, inflammation and oxidative stress, but the molecular mechanisms of damage are only partially understood. ...CONCLUSIONS: Lack of MCP-1 affords protection from damage and development of …
BACKGROUND/AIMS: Administration of carbon tetrachloride determines liver injury, inflammation and oxidative stress, but the mo …
Hepatic Macrophages in Liver Injury.
Shan Z, Ju C. Shan Z, et al. Front Immunol. 2020 Apr 17;11:322. doi: 10.3389/fimmu.2020.00322. eCollection 2020. Front Immunol. 2020. PMID: 32362892 Free PMC article. Review.
Ample evidence suggests that hepatic macrophages play key roles in the injury and repair mechanisms during liver disease progression. There are two major populations of hepatic macrophages: the liver resident Kupffer cells and the monocyte-deriv …
Ample evidence suggests that hepatic macrophages play key roles in the injury and repair mechanisms during liver diseas …
34 results
You have reached the last page of results. A maximum of 10,000 results are available.
Jump to page
Feedback