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Interleukin-1 upregulates anaphylatoxin receptors on mononuclear cells.
Takabayashi T, Shimizu S, Clark BD, Beinborn M, Burke JF, Gelfand JA. Takabayashi T, et al. Surgery. 2004 May;135(5):544-54. doi: 10.1016/j.surg.2003.09.010. Surgery. 2004. PMID: 15118592
We investigated the effects of IL-1 on anaphylatoxin receptor expression in monocytes. METHODS: A human monocytic cell line, MONO-MAC-6, was used. ...IL-1 receptor antagonist blocked the effects of IL-1alpha upregulation of anaphylatoxin rece
We investigated the effects of IL-1 on anaphylatoxin receptor expression in monocytes. METHODS: A human monocytic cell line, M …
C5a/C5aR pathway is essential for up-regulating SphK1 expression through p38-MAPK activation in acute liver failure.
Lei YC, Lu CL, Chen L, Ge K, Yang LL, Li W, Wu YH. Lei YC, et al. World J Gastroenterol. 2016 Dec 14;22(46):10148-10157. doi: 10.3748/wjg.v22.i46.10148. World J Gastroenterol. 2016. PMID: 28028363 Free PMC article.
Expression of C5aR, sphingosine kinase 1 (SphK1), p38-MAPK and p-p38-MAPK in liver tissue, peripheral blood mononuclear cells (PBMCs) and peritoneal exudative macrophages (PEMs) of mice or RAW 264.7 cells was analyzed by western blotting. C5aR mRNA levels wer …
Expression of C5aR, sphingosine kinase 1 (SphK1), p38-MAPK and p-p38-MAPK in liver tissue, peripheral blood mononuclear cells
The priming effects of the products of stimulated mononuclear cells on the response of neutrophils to C5a des arg.
Crouch S, Fletcher J. Crouch S, et al. Br J Haematol. 1991 Feb;77(2):158-64. doi: 10.1111/j.1365-2141.1991.tb07971.x. Br J Haematol. 1991. PMID: 2004016
This effect could not be reproduced with recombinant human forms of interleukin 1 beta (Il-1 beta) or granulocyte-macrophage-colony stimulating factor (GM-CSF), although, with the latter, there was some effect when the preincubation stage was carried out for 60 min …
This effect could not be reproduced with recombinant human forms of interleukin 1 beta (Il-1 beta) or granulocyte-macrophage-c …
Increased Complement 3a Receptor is Associated with Behcet's disease and Vogt-Koyanagi-Harada disease.
Wang C, Cao S, Zhang D, Li H, Kijlstra A, Yang P. Wang C, et al. Sci Rep. 2017 Nov 14;7(1):15579. doi: 10.1038/s41598-017-15740-8. Sci Rep. 2017. PMID: 29138505 Free PMC article.
Behcet's disease (BD) and Vogt-Koyanagi-Harada disease (VKH) are systemic and recurrent autoimmune diseases associated with abnormal T cell immune response. Complement 3a receptor (C3aR) and complement 5a receptor (C5aR) have been reported to be involved in T cell m …
Behcet's disease (BD) and Vogt-Koyanagi-Harada disease (VKH) are systemic and recurrent autoimmune diseases associated with abnormal T cell …
Complement gene expression is regulated by pro-inflammatory cytokines and the anaphylatoxin C3a in human tenocytes.
Busch C, Girke G, Kohl B, Stoll C, Lemke M, Krasnici S, Ertel W, Silawal S, John T, Schulze-Tanzil G. Busch C, et al. Mol Immunol. 2013 Apr;53(4):363-73. doi: 10.1016/j.molimm.2012.09.001. Epub 2012 Oct 13. Mol Immunol. 2013. PMID: 23070120
The expression and regulation of complement factors by cytokines or anaphylatoxins are completely unclear in tendon. Hence, the gene expression of the anaphylatoxin receptors C3aR, C5aR and cytoprotective complement regulatory proteins (CRPs) was analysed in …
The expression and regulation of complement factors by cytokines or anaphylatoxins are completely unclear in tendon. Hence, the gene …