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Interleukin-1 upregulates anaphylatoxin receptors on mononuclear cells.
Takabayashi T, Shimizu S, Clark BD, Beinborn M, Burke JF, Gelfand JA. Takabayashi T, et al. Surgery. 2004 May;135(5):544-54. doi: 10.1016/j.surg.2003.09.010. Surgery. 2004. PMID: 15118592
We investigated the effects of IL-1 on anaphylatoxin receptor expression in monocytes. METHODS: A human monocytic cell line, MONO-MAC-6, was used. ...IL-1 receptor antagonist blocked the effects of IL-1alpha upregulation of anap
We investigated the effects of IL-1 on anaphylatoxin receptor expression in monocytes. METHODS: A human monocytic ce
C5a/C5aR pathway is essential for up-regulating SphK1 expression through p38-MAPK activation in acute liver failure.
Lei YC, Lu CL, Chen L, Ge K, Yang LL, Li W, Wu YH. Lei YC, et al. World J Gastroenterol. 2016 Dec 14;22(46):10148-10157. doi: 10.3748/wjg.v22.i46.10148. World J Gastroenterol. 2016. PMID: 28028363 Free PMC article.
Serum complement 5 (C5), C5a, tumor necrosis factor-alpha (TNF-alpha), interleukin (IL)-1beta, IL-6, high-mobility group protein B1 (HMGB1) and sphingosine-1-phosphate levels were detected by enzyme-linked immunosorbant assay. Hepatic morphological changes at 36 h a …
Serum complement 5 (C5), C5a, tumor necrosis factor-alpha (TNF-alpha), interleukin (IL)-1beta, IL-6, high-mobility group protein B1 ( …
The priming effects of the products of stimulated mononuclear cells on the response of neutrophils to C5a des arg.
Crouch S, Fletcher J. Crouch S, et al. Br J Haematol. 1991 Feb;77(2):158-64. doi: 10.1111/j.1365-2141.1991.tb07971.x. Br J Haematol. 1991. PMID: 2004016
Certain recombinant human cytokines have been shown to enhance polymorphonuclear leucocyte (PMN) responses to subsequent stimulation. Mononuclear cells (MNC) from normal healthy individuals were stimulated for 5 h with 1 micrograms/ml bacterial lipopolysaccha …
Certain recombinant human cytokines have been shown to enhance polymorphonuclear leucocyte (PMN) responses to subsequent stimulation. Mon
Increased Complement 3a Receptor is Associated with Behcet's disease and Vogt-Koyanagi-Harada disease.
Wang C, Cao S, Zhang D, Li H, Kijlstra A, Yang P. Wang C, et al. Sci Rep. 2017 Nov 14;7(1):15579. doi: 10.1038/s41598-017-15740-8. Sci Rep. 2017. PMID: 29138505 Free PMC article.
Behcet's disease (BD) and Vogt-Koyanagi-Harada disease (VKH) are systemic and recurrent autoimmune diseases associated with abnormal T cell immune response. Complement 3a receptor (C3aR) and complement 5a receptor (C5aR) have been reported to be involv …
Behcet's disease (BD) and Vogt-Koyanagi-Harada disease (VKH) are systemic and recurrent autoimmune diseases associated with abnormal …
Complement gene expression is regulated by pro-inflammatory cytokines and the anaphylatoxin C3a in human tenocytes.
Busch C, Girke G, Kohl B, Stoll C, Lemke M, Krasnici S, Ertel W, Silawal S, John T, Schulze-Tanzil G. Busch C, et al. Mol Immunol. 2013 Apr;53(4):363-73. doi: 10.1016/j.molimm.2012.09.001. Epub 2012 Oct 13. Mol Immunol. 2013. PMID: 23070120
The expression and regulation of complement factors by cytokines or anaphylatoxins are completely unclear in tendon. ...Time-dependent regulation of complement by cytokines and the anaphylatoxin C3a was assessed in cultured tenocytes. ...
The expression and regulation of complement factors by cytokines or anaphylatoxins are completely unclear in tendon. ...Time-d …
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