Involvement of JAM-A in mononuclear cell recruitment on inflamed or atherosclerotic endothelium: inhibition by soluble JAM-A.
Arterioscler Thromb Vasc Biol. 2005.
PMID: 15681301
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Involvement of JAM-A in mononuclear cell recruitment on inflamed or atherosclerotic endothelium: inhibition by soluble JAM-A.
Ostermann G, Fraemohs L, Baltus T, Schober A, Lietz M, Zernecke A, Liehn EA, Weber C.
Ostermann G, et al.
Arterioscler Thromb Vasc Biol. 2005 Apr;25(4):729-35. doi: 10.1161/01.ATV.0000157154.14474.3b. Epub 2005 Jan 27.
Arterioscler Thromb Vasc Biol. 2005.
PMID: 15681301
It is unknown whether these functions can be inhibited by soluble forms of JAM-A, whether JAM-A is expressed on atherosclerotic endothelium, and whether it participates in atherogenic recruitment of mononuclear …
It is unknown whether these functions can be inhibited by soluble forms of JAM-A, whether JAM-A is …
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Platelets and their chemokines in atherosclerosis-clinical applications.
von Hundelshausen P, Schmitt MM.
von Hundelshausen P, et al.
Front Physiol. 2014 Aug 8;5:294. doi: 10.3389/fphys.2014.00294. eCollection 2014.
Front Physiol. 2014.
PMID: 25152735
Free PMC article.
Review.
CCL5 is released and deposited on endothelium by activated platelets thereby triggering atherogenic monocyte recruitment, which can be attenuated by blocking the corresponding chemokine receptor CCR5. ...Platelet surface molecules GPIIb/IIIa, GP1balpha, P-selectin, …
CCL5 is released and deposited on endothelium by activated platelets thereby triggering atherogenic monocyte recruitment, whic …
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