Monocyte function and trafficking in cardiovascular disease.
Thromb Haemost. 2012.
PMID: 22918193
Review.
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Monocyte function and trafficking in cardiovascular disease.
Thromb Haemost. 2012 Nov;108(5):804-11. doi: 10.1160/TH12-04-0276. Epub 2012 Aug 23.
Thromb Haemost. 2012.
PMID: 22918193
Review.
Despite their role in immune defense and tissue repair mechanisms, monocytes are also involved in several pathological conditions such as autoimmune and cardiovascular diseases as well as cancer. ...This review focuses on recent advances on the role of mon …
Despite their role in immune defense and tissue repair mechanisms, monocytes are also involved in several pathological conditions suc …
Aging disrupts circadian gene regulation and function in macrophages.
Nat Immunol. 2022 Feb;23(2):229-236. doi: 10.1038/s41590-021-01083-0. Epub 2021 Dec 23.
Nat Immunol. 2022.
PMID: 34949832
Free PMC article.
Aging is characterized by an increased vulnerability to infection and the development of inflammatory diseases, such as atherosclerosis, frailty, cancer and neurodegeneration. Here, we find that aging is associated with the loss of diurnally rhythmic innate immune response …
Aging is characterized by an increased vulnerability to infection and the development of inflammatory diseases, such as atheroscleros …
CCL17 Aggravates Myocardial Injury by Suppressing Recruitment of Regulatory T Cells.
Circulation. 2022 Mar 8;145(10):765-782. doi: 10.1161/CIRCULATIONAHA.121.055888. Epub 2022 Feb 3.
Circulation. 2022.
PMID: 35113652
Free PMC article.
Elucidation of the effector mechanisms that mediate adverse effects of CCR2(+) monocytes, macrophages, and dendritic cells will yield important insights into therapeutic strategies to suppress myocardial inflammation. ...We used Ccl17 knockout mice, flow cytometry, RNA seq …
Elucidation of the effector mechanisms that mediate adverse effects of CCR2(+) monocytes, macrophages, and dendritic cells will yield …
TREM-1 orchestrates angiotensin II-induced monocyte trafficking and promotes experimental abdominal aortic aneurysm.
J Clin Invest. 2021 Jan 19;131(2):e142468. doi: 10.1172/JCI142468.
J Clin Invest. 2021.
PMID: 33258804
Free PMC article.
The triggering receptor expressed on myeloid cells 1 (TREM-1) drives inflammatory responses in several cardiovascular diseases but its role in abdominal aortic aneurysm (AAA) remains unknown. ...Trem1 gene deletion attenuated the inflammatory response in the aorta, …
The triggering receptor expressed on myeloid cells 1 (TREM-1) drives inflammatory responses in several cardiovascular diseases …
Monocyte Recruitment, Specification, and Function in Atherosclerosis.
Cells. 2020 Dec 24;10(1):15. doi: 10.3390/cells10010015.
Cells. 2020.
PMID: 33374145
Free PMC article.
Review.
Yet, many questions regarding tissue-specific regulation of monocyte-to-macrophage differentiation and the contribution of recruited monocytes at stages of atherosclerotic disease progression remain unknown. In this review, we highlight recent advances regard …
Yet, many questions regarding tissue-specific regulation of monocyte-to-macrophage differentiation and the contribution of recruited …
S100A9 Links Inflammation and Repair in Myocardial Infarction.
Circ Res. 2020 Aug 14;127(5):664-676. doi: 10.1161/CIRCRESAHA.120.315865. Epub 2020 May 21.
Circ Res. 2020.
PMID: 32434457
Monocyte trafficking from the spleen to the myocardium and subsequent phenotype switching to reparatory Ly6C(lo)MerTK(hi) macrophages was also impaired, leading to inefficient efferocytosis, accumulation of apoptotic cardiomyocytes, and a larger myocardial scar. The …
Monocyte trafficking from the spleen to the myocardium and subsequent phenotype switching to reparatory Ly6C(lo)MerTK(hi) macr …
Monocyte and Macrophage Dynamics in the Cardiovascular System: JACC Macrophage in CVD Series (Part 3).
J Am Coll Cardiol. 2018 Oct 30;72(18):2198-2212. doi: 10.1016/j.jacc.2018.08.2150.
J Am Coll Cardiol. 2018.
PMID: 30360828
Free PMC article.
Review.
In parallel, progressive advances in imaging and cell labeling techniques have opened new opportunities for in vivo imaging of monocyte/macrophage trafficking in atherosclerotic lesions and at the systemic level. In this Part 3 of a 4-part review series covering the …
In parallel, progressive advances in imaging and cell labeling techniques have opened new opportunities for in vivo imaging of monocyte …
Monocyte trafficking in acute and chronic inflammation.
Trends Immunol. 2011 Oct;32(10):470-7. doi: 10.1016/j.it.2011.05.001. Epub 2011 Jun 12.
Trends Immunol. 2011.
PMID: 21664185
Free PMC article.
Review.
These mechanisms will be compared to monocyte trafficking during chronic inflammation in experimental models of atherosclerosis. Recent developments suggest that monocyte trafficking shares common themes in diverse inflammatory diseases; however …
These mechanisms will be compared to monocyte trafficking during chronic inflammation in experimental models of atherosclerosi …
Methods to Study Monocyte and Macrophage Trafficking in Atherosclerosis Progression and Resolution.
Methods Mol Biol. 2019;1951:153-165. doi: 10.1007/978-1-4939-9130-3_12.
Methods Mol Biol. 2019.
PMID: 30825151
Free PMC article.
Monocytes are circulating cells imperative to the response against pathogens. ...There are two major subsets of monocytes, "inflammatory" and "nonclassical," which are believed to have distinct functions. ...
Monocytes are circulating cells imperative to the response against pathogens. ...There are two major subsets of monocytes, "in …
Trafficking of Mononuclear Phagocytes in Healthy Arteries and Atherosclerosis.
Front Immunol. 2021 Oct 25;12:718432. doi: 10.3389/fimmu.2021.718432. eCollection 2021.
Front Immunol. 2021.
PMID: 34759917
Free PMC article.
Review.
Monocytes and macrophages play essential roles in all stages of atherosclerosis - from early precursor lesions to advanced stages of the disease. ...Furthermore, we discuss the functional and ontogenetic composition of the aortic pool of mononuclear phagocyte …
Monocytes and macrophages play essential roles in all stages of atherosclerosis - from early precursor lesions to advanced stages of …
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