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No evidence for prooxidative effects of homocysteine in vascular endothelial cells.
Frank J, Beck SC, Flaccus A, Biesalski HK. Frank J, et al. Eur J Nutr. 2007 Aug;46(5):286-92. doi: 10.1007/s00394-007-0663-x. Epub 2007 Jun 28. Eur J Nutr. 2007. PMID: 17599238
However, our results demonstrate that HCys had neither prooxidative nor cytotoxic effects on endothelial cells. Only a moderate time- and concentration-dependent increase in monocyte adhesion up to 28.3 +/- 5.5% was achieved relative to control after 4 …
However, our results demonstrate that HCys had neither prooxidative nor cytotoxic effects on endothelial cells. …
Vascular incompetence in dialysis patients--protein-bound uremic toxins and endothelial dysfunction.
Jourde-Chiche N, Dou L, Cerini C, Dignat-George F, Brunet P. Jourde-Chiche N, et al. Semin Dial. 2011 May-Jun;24(3):327-37. doi: 10.1111/j.1525-139X.2011.00925.x. Semin Dial. 2011. PMID: 21682773 Review.
Patients with CKD display impaired endothelium-dependent vasodilatation, elevated soluble biomarkers of endothelial dysfunction, and increased oxidative stress. They also present an imbalance between circulating endothelial populations reflecting endothelial …
Patients with CKD display impaired endothelium-dependent vasodilatation, elevated soluble biomarkers of endothelial dysfunction, and …
Coronary artery disease--free radical damage, antioxidant protection and the role of homocysteine.
Maxwell SR. Maxwell SR. Basic Res Cardiol. 2000;95 Suppl 1:I65-71. doi: 10.1007/s003950070012. Basic Res Cardiol. 2000. PMID: 11192356 Review.
This may facilitate the development of vascular disease because of (i) increased oxidation of low-density lipoprotein (LDL) particles which increases their propensity to deposition in the vascular wall, (ii) inactivation of endothelium-derived nitric oxide, and (iii …
This may facilitate the development of vascular disease because of (i) increased oxidation of low-density lipoprotein (LDL) particles …