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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1991 1
1998 1
2000 1
2003 1
2004 1
2005 2
2006 3
2007 1
2008 7
2009 3
2010 3
2011 6
2012 4
2013 10
2014 20
2015 20
2016 5
2017 5
2018 3
2019 4
2020 11
2021 0
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Similar articles for PMID: 21289217

97 results
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Distinct macrophage phenotypes contribute to kidney injury and repair.
Lee S, Huen S, Nishio H, Nishio S, Lee HK, Choi BS, Ruhrberg C, Cantley LG. Lee S, et al. J Am Soc Nephrol. 2011 Feb;22(2):317-26. doi: 10.1681/ASN.2009060615. J Am Soc Nephrol. 2011. PMID: 21289217 Free PMC article.
GM-CSF Promotes Macrophage Alternative Activation after Renal Ischemia/Reperfusion Injury.
Huen SC, Huynh L, Marlier A, Lee Y, Moeckel GW, Cantley LG. Huen SC, et al. J Am Soc Nephrol. 2015 Jun;26(6):1334-45. doi: 10.1681/ASN.2014060612. Epub 2014 Nov 11. J Am Soc Nephrol. 2015. PMID: 25388222 Free PMC article.
Mesenchymal stem cells ameliorate rhabdomyolysis-induced acute kidney injury via the activation of M2 macrophages.
Geng Y, Zhang L, Fu B, Zhang J, Hong Q, Hu J, Li D, Luo C, Cui S, Zhu F, Chen X. Geng Y, et al. Stem Cell Res Ther. 2014 Jun 24;5(3):80. doi: 10.1186/scrt469. Stem Cell Res Ther. 2014. PMID: 24961539 Free PMC article.
Netrin-1-treated macrophages protect the kidney against ischemia-reperfusion injury and suppress inflammation by inducing M2 polarization.
Ranganathan PV, Jayakumar C, Ramesh G. Ranganathan PV, et al. Am J Physiol Renal Physiol. 2013 Apr 1;304(7):F948-57. doi: 10.1152/ajprenal.00580.2012. Epub 2013 Feb 13. Am J Physiol Renal Physiol. 2013. PMID: 23408164 Free PMC article.
Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy.
Lech M, Gröbmayr R, Ryu M, Lorenz G, Hartter I, Mulay SR, Susanti HE, Kobayashi KS, Flavell RA, Anders HJ. Lech M, et al. J Am Soc Nephrol. 2014 Feb;25(2):292-304. doi: 10.1681/ASN.2013020152. Epub 2013 Dec 5. J Am Soc Nephrol. 2014. PMID: 24309188 Free PMC article.
Macrophages are involved in the protective role of human umbilical cord-derived stromal cells in renal ischemia-reperfusion injury.
Li W, Zhang Q, Wang M, Wu H, Mao F, Zhang B, Ji R, Gao S, Sun Z, Zhu W, Qian H, Chen Y, Xu W. Li W, et al. Stem Cell Res. 2013 May;10(3):405-16. doi: 10.1016/j.scr.2013.01.005. Epub 2013 Jan 29. Stem Cell Res. 2013. PMID: 23466564 Free article.
Treatment with Recombinant Trichinella spiralis Cathepsin B-like Protein Ameliorates Intestinal Ischemia/Reperfusion Injury in Mice by Promoting a Switch from M1 to M2 Macrophages.
Liu WF, Wen SH, Zhan JH, Li YS, Shen JT, Yang WJ, Zhou XW, Liu KX. Liu WF, et al. J Immunol. 2015 Jul 1;195(1):317-28. doi: 10.4049/jimmunol.1401864. Epub 2015 May 18. J Immunol. 2015. PMID: 25987744 Free article.
Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells.
Duffield JS, Park KM, Hsiao LL, Kelley VR, Scadden DT, Ichimura T, Bonventre JV. Duffield JS, et al. J Clin Invest. 2005 Jul;115(7):1743-55. doi: 10.1172/JCI22593. Epub 2005 Jun 2. J Clin Invest. 2005. PMID: 16007251 Free PMC article.
Blockade of interleukin-6 signaling inhibits the classic pathway and promotes an alternative pathway of macrophage activation after spinal cord injury in mice.
Guerrero AR, Uchida K, Nakajima H, Watanabe S, Nakamura M, Johnson WE, Baba H. Guerrero AR, et al. J Neuroinflammation. 2012 Feb 27;9:40. doi: 10.1186/1742-2094-9-40. J Neuroinflammation. 2012. PMID: 22369693 Free PMC article.
Failed renoprotection by alternatively activated bone marrow macrophages is due to a proliferation-dependent phenotype switch in vivo.
Cao Q, Wang Y, Zheng D, Sun Y, Wang C, Wang XM, Lee VW, Wang Y, Zheng G, Tan TK, Wang YM, Alexander SI, Harris DC. Cao Q, et al. Kidney Int. 2014 Apr;85(4):794-806. doi: 10.1038/ki.2013.341. Epub 2013 Sep 18. Kidney Int. 2014. PMID: 24048378 Free article.
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