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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1999 2
2000 2
2001 1
2002 1
2003 1
2004 5
2005 10
2006 15
2007 14
2008 8
2009 11
2010 8
2011 7
2012 7
2013 4
2015 3
2016 6
2017 3
2018 5
2019 2
2020 12
2021 1
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Similar articles for PMID: 23226049

115 results
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The molecular mechanisms and pharmacotherapy of ATP-sensitive potassium channel gene mutations underlying neonatal diabetes.
Lang V, Light PE. Lang V, et al. Pharmgenomics Pers Med. 2010;3:145-61. doi: 10.2147/PGPM.S6969. Epub 2010 Nov 24. Pharmgenomics Pers Med. 2010. PMID: 23226049 Free PMC article.
Diabetes and hypoglycaemia in young children and mutations in the Kir6.2 subunit of the potassium channel: therapeutic consequences.
Flechtner I, de Lonlay P, Polak M. Flechtner I, et al. Diabetes Metab. 2006 Dec;32(6):569-80. doi: 10.1016/S1262-3636(07)70311-7. Diabetes Metab. 2006. PMID: 17296510 Review.
Update of mutations in the genes encoding the pancreatic beta-cell K(ATP) channel subunits Kir6.2 (KCNJ11) and sulfonylurea receptor 1 (ABCC8) in diabetes mellitus and hyperinsulinism.
Flanagan SE, Clauin S, Bellanné-Chantelot C, de Lonlay P, Harries LW, Gloyn AL, Ellard S. Flanagan SE, et al. Hum Mutat. 2009 Feb;30(2):170-80. doi: 10.1002/humu.20838. Hum Mutat. 2009. PMID: 18767144 Review.
Transient Neonatal Diabetes Mellitus in a Very Preterm Infant due to ABCC8 Mutation.
Piccini B, Coviello C, Drovandi L, Rosangela A, Monzali F, Casalini E, Giglio S, Toni S, Dani C. Piccini B, et al. AJP Rep. 2018 Jan;8(1):e39-e42. doi: 10.1055/s-0038-1636427. Epub 2018 Mar 7. AJP Rep. 2018. PMID: 29527407 Free PMC article.
Diabetes in very young children and mutations in the insulin-secreting cell potassium channel genes: therapeutic consequences.
Flechtner I, Vaxillaire M, Cavé H, Scharfmann R, Froguel P, Polak M. Flechtner I, et al. Endocr Dev. 2007;12:86-98. doi: 10.1159/000109636. Endocr Dev. 2007. PMID: 17923772
Mutations in the genes encoding the pancreatic beta-cell KATP channel subunits Kir6.2 (KCNJ11) and SUR1 (ABCC8) in diabetes mellitus and hyperinsulinism.
Gloyn AL, Siddiqui J, Ellard S. Gloyn AL, et al. Hum Mutat. 2006 Mar;27(3):220-31. doi: 10.1002/humu.20292. Hum Mutat. 2006. PMID: 16416420 Review.
ABCC8 (SUR1) and KCNJ11 (KIR6.2) mutations in persistent hyperinsulinemic hypoglycemia of infancy and evaluation of different therapeutic measures.
Darendeliler F, Fournet JC, Baş F, Junien C, Gross MS, Bundak R, Saka N, Günöz H. Darendeliler F, et al. J Pediatr Endocrinol Metab. 2002 Jul-Aug;15(7):993-1000. doi: 10.1515/jpem.2002.15.7.993. J Pediatr Endocrinol Metab. 2002. PMID: 12199344
A rare mutation in ABCC8/SUR1 leading to altered ATP-sensitive K+ channel activity and beta-cell glucose sensing is associated with type 2 diabetes in adults.
Tarasov AI, Nicolson TJ, Riveline JP, Taneja TK, Baldwin SA, Baldwin JM, Charpentier G, Gautier JF, Froguel P, Vaxillaire M, Rutter GA. Tarasov AI, et al. Diabetes. 2008 Jun;57(6):1595-604. doi: 10.2337/db07-1547. Epub 2008 Mar 17. Diabetes. 2008. PMID: 18346985 Free PMC article.
Mutations in ATP-sensitive K+ channel genes cause transient neonatal diabetes and permanent diabetes in childhood or adulthood.
Flanagan SE, Patch AM, Mackay DJ, Edghill EL, Gloyn AL, Robinson D, Shield JP, Temple K, Ellard S, Hattersley AT. Flanagan SE, et al. Diabetes. 2007 Jul;56(7):1930-7. doi: 10.2337/db07-0043. Epub 2007 Apr 19. Diabetes. 2007. PMID: 17446535 Free article.
A heterozygous activating mutation in the sulphonylurea receptor SUR1 (ABCC8) causes neonatal diabetes.
Proks P, Arnold AL, Bruining J, Girard C, Flanagan SE, Larkin B, Colclough K, Hattersley AT, Ashcroft FM, Ellard S. Proks P, et al. Hum Mol Genet. 2006 Jun 1;15(11):1793-800. doi: 10.1093/hmg/ddl101. Epub 2006 Apr 13. Hum Mol Genet. 2006. PMID: 16613899
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