Cited In for PMID: 36934777

Year	Number of Results
2023	4
2024	6
2025	7
2026	0

1

Reticulocyte mitochondrial retention increases reactive oxygen species and oxygen consumption in mouse models of sickle cell disease and phlebotomy-induced anemia.

Gallivan A, Alejandro M, Kanu A, Zekaryas N, Horneman H, Hong LK, Vinchinsky E, Lavelle D, Diamond AM, Molokie RE, Ramasamy J, Rivers A. Gallivan A, et al. Exp Hematol. 2023 Jun;122:55-62. doi: 10.1016/j.exphem.2023.02.005. Epub 2023 Mar 16. Exp Hematol. 2023. PMID: 36934777 Free article.

2

Cell-free mitochondrial DNA as a pro-inflammatory agent in blood circulation: mechanisms, therapeutic implications, and clinical challenges in immune dysregulation.

Zhao Y, Wu C, Liang X, Yang M. Zhao Y, et al. Front Immunol. 2025 Oct 1;16:1640748. doi: 10.3389/fimmu.2025.1640748. eCollection 2025. Front Immunol. 2025. PMID: 41103421 Free PMC article. Review.

3

Mitochondrial retention and autophagy dysregulation drive oxidative stress in sickle cell disease erythrocytes.

Ramasamy J, Punathil Kannan PK, Dhavamani S, Palanimuthu S, Molokie R, Rivers A. Ramasamy J, et al. Exp Hematol. 2025 Dec;152:105269. doi: 10.1016/j.exphem.2025.105269. Epub 2025 Oct 2. Exp Hematol. 2025. PMID: 41046099 Free PMC article.

4

Dual Assessment of the RBC Fraction Abnormally Retaining Mitochondria and of the RBC Mitochondrial Load Provides a New Clinical Parameter for Sickle Cell Disease Patients.

Soupene E, Horneman H, Alejandro M, Mohammed K, Nyansa Ansong-Ansongton YO, Rivers A. Soupene E, et al. EJHaem. 2025 Aug 26;6(4):e70130. doi: 10.1002/jha2.70130. eCollection 2025 Aug. EJHaem. 2025. PMID: 40873469 Free PMC article.

5

Innovations in Drug Discovery for Sickle Cell Disease Targeting Oxidative Stress and NRF2 Activation-A Short Review.

Starlard-Davenport A, Palani CD, Zhu X, Pace BS. Starlard-Davenport A, et al. Int J Mol Sci. 2025 Apr 28;26(9):4192. doi: 10.3390/ijms26094192. Int J Mol Sci. 2025. PMID: 40362428 Free PMC article. Review.

6

The involvement of mitochondria in erythrocyte pathology and diseases: from mechanisms to therapeutic strategies.

Lin D, Yang H, Liang X, Yang M, Zhao Y. Lin D, et al. Clin Exp Med. 2025 May 9;25(1):144. doi: 10.1007/s10238-024-01555-1. Clin Exp Med. 2025. PMID: 40343592 Free PMC article. Review.

7

Are Mitochondria a Potential Target for Treating β-Thalassemia?

Di Pierro E, Di Stefano V, Migone De Amicis M, Graziadei G. Di Pierro E, et al. J Clin Med. 2025 Feb 8;14(4):1095. doi: 10.3390/jcm14041095. J Clin Med. 2025. PMID: 40004626 Free PMC article. Review.

8

Transsulfuration pathway activation attenuates oxidative stress and ferroptosis in sickle primary erythroblasts and transgenic mice.

Xi C, Pang J, Xue W, Cui Y, Jiang N, Zhi W, Shi H, Horuzsko A, Pace BS, Zhu X. Xi C, et al. Commun Biol. 2025 Jan 6;8(1):15. doi: 10.1038/s42003-024-07424-7. Commun Biol. 2025. PMID: 39762627 Free PMC article.

9

Mitochondrial retention in mature red blood cells from patients with sickle cell disease is associated with stress erythropoiesis but not with proinflammatory state.

Romana M, Laurance S, Hardy-Dessources MD, Claer L, Ravion S, Dorgham K, Garnier Y, Kuznicki L, Tarer V, Tressières B, Lefevre SD, Baccini V, Ostuni MA, Le Van Kim C, Etienne-Julan M. Romana M, et al. Hemasphere. 2024 Oct 28;8(11):e70030. doi: 10.1002/hem3.70030. eCollection 2024 Nov. Hemasphere. 2024. PMID: 39473454 Free PMC article. No abstract available.

10

Ferroptosis as an emerging target in sickle cell disease.

Fortuna V, Lima J, Oliveira GF, Oliveira YS, Getachew B, Nekhai S, Aschner M, Tizabi Y. Fortuna V, et al. Curr Res Toxicol. 2024 Jun 18;7:100181. doi: 10.1016/j.crtox.2024.100181. eCollection 2024. Curr Res Toxicol. 2024. PMID: 39021403 Free PMC article.

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