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Year Number of Results
1989 4
1990 3
1991 4
1992 19
1993 27
1994 38
1995 40
1996 49
1997 61
1998 66
1999 67
2000 106
2001 115
2002 107
2003 137
2004 154
2005 193
2006 200
2007 212
2008 243
2009 271
2010 371
2011 378
2012 443
2013 458
2014 512
2015 517
2016 558
2017 621
2018 620
2019 682
2020 802
2021 830
2022 798
2023 848
2024 970
2025 1362
2026 464

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11,826 results

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Page 1
Interaction between Abeta and Tau in the Pathogenesis of Alzheimer's Disease.
Zhang H, Wei W, Zhao M, Ma L, Jiang X, Pei H, Cao Y, Li H. Zhang H, et al. Int J Biol Sci. 2021 May 27;17(9):2181-2192. doi: 10.7150/ijbs.57078. eCollection 2021. Int J Biol Sci. 2021. PMID: 34239348 Free PMC article. Review.
Extracellular neuritic plaques composed of amyloid-beta (Abeta) protein and intracellular neurofibrillary tangles containing phosphorylated tau protein are the two hallmark proteins of Alzheimer's disease (AD), and the separate neurotoxicity of …
Extracellular neuritic plaques composed of amyloid-beta (Abeta) protein and intracellular neurofibrillary tangles containing p …
The Cellular Phase of Alzheimer's Disease.
De Strooper B, Karran E. De Strooper B, et al. Cell. 2016 Feb 11;164(4):603-15. doi: 10.1016/j.cell.2015.12.056. Cell. 2016. PMID: 26871627 Free article. Review.
The amyloid hypothesis for Alzheimer's disease (AD) posits a neuron-centric, linear cascade initiated by Abeta and leading to dementia. ...
The amyloid hypothesis for Alzheimer's disease (AD) posits a neuron-centric, linear cascade initiated by Abeta a …
Amyloidosis in Alzheimer's Disease: Pathogeny, Etiology, and Related Therapeutic Directions.
Ma C, Hong F, Yang S. Ma C, et al. Molecules. 2022 Feb 11;27(4):1210. doi: 10.3390/molecules27041210. Molecules. 2022. PMID: 35209007 Free PMC article. Review.
The amyloid hypothesis of Alzheimer's disease has long been the predominant theory, suggesting that Alzheimer's disease is caused by the accumulation of amyloid beta protein (Abeta) in the brain, leading to neuronal t …
The amyloid hypothesis of Alzheimer's disease has long been the predominant theory, suggesting that Alzheimer
Amyloid Precursor Protein and Alzheimer's Disease.
Orobets KS, Karamyshev AL. Orobets KS, et al. Int J Mol Sci. 2023 Sep 30;24(19):14794. doi: 10.3390/ijms241914794. Int J Mol Sci. 2023. PMID: 37834241 Free PMC article. Review.
Alzheimer's disease (AD) is one of the most common neurodegenerative disorders associated with age or inherited mutations. ...AD progression is linked to the accumulation of cytotoxic amyloid beta (Abeta) and hyperphosphorylated tau protein comb
Alzheimer's disease (AD) is one of the most common neurodegenerative disorders associated with age or inherited mutatio
Apoptosis in Alzheimer's disease: insight into the signaling pathways and therapeutic avenues.
Kumari S, Dhapola R, Reddy DH. Kumari S, et al. Apoptosis. 2023 Aug;28(7-8):943-957. doi: 10.1007/s10495-023-01848-y. Epub 2023 Apr 26. Apoptosis. 2023. PMID: 37186274 Review.
Alzheimer's disease (AD) is characterized by the accumulation of hyperphosphorylated tau and amyloid-beta (Abeta) protein resulting in synaptic loss and apoptosis. ...Drugs like filgrastim, epigallocatechin gallate, curcumin, nicergoline and min
Alzheimer's disease (AD) is characterized by the accumulation of hyperphosphorylated tau and amyloid-beta
Molecular and cellular mechanisms underlying the pathogenesis of Alzheimer's disease.
Guo T, Zhang D, Zeng Y, Huang TY, Xu H, Zhao Y. Guo T, et al. Mol Neurodegener. 2020 Jul 16;15(1):40. doi: 10.1186/s13024-020-00391-7. Mol Neurodegener. 2020. PMID: 32677986 Free PMC article. Review.
Alzheimer's disease (AD) is the most common neurodegenerative disorder seen in age-dependent dementia. ...Studies within the last few decades provide growing evidence for a central role of amyloid beta (Abeta) and tau, as well as glial contribut
Alzheimer's disease (AD) is the most common neurodegenerative disorder seen in age-dependent dementia. ...Studies withi
The neuroimmune axis of Alzheimer's disease.
Jorfi M, Maaser-Hecker A, Tanzi RE. Jorfi M, et al. Genome Med. 2023 Jan 26;15(1):6. doi: 10.1186/s13073-023-01155-w. Genome Med. 2023. PMID: 36703235 Free PMC article. Review.
Alzheimer's disease (AD) is a genetically complex and heterogeneous disorder with multifaceted neuropathological features, including beta-amyloid plaques, neurofibrillary tangles, and neuroinflammation. ...We emphasize the role of peripheral imm
Alzheimer's disease (AD) is a genetically complex and heterogeneous disorder with multifaceted neuropathological featur
The role of CREB and BDNF in neurobiology and treatment of Alzheimer's disease.
Amidfar M, de Oliveira J, Kucharska E, Budni J, Kim YK. Amidfar M, et al. Life Sci. 2020 Sep 15;257:118020. doi: 10.1016/j.lfs.2020.118020. Epub 2020 Jun 27. Life Sci. 2020. PMID: 32603820 Review.
Alzheimer's disease (AD) is the most common form of dementia worldwide. beta-amyloid peptide (Abeta) is currently assumed to be the main cause of synaptic dysfunction and cognitive impairments in AD, but the molecular signaling pathways u
Alzheimer's disease (AD) is the most common form of dementia worldwide. beta-amyloid peptide (Abeta) is c
Anti-amyloid-beta Antibodies and Anti-tau Therapies for Alzheimer's Disease: Recent Advances and Perspectives.
Suzuki N, Hatta T, Ito M, Kusakabe KI. Suzuki N, et al. Chem Pharm Bull (Tokyo). 2024;72(7):602-609. doi: 10.1248/cpb.c24-00069. Chem Pharm Bull (Tokyo). 2024. PMID: 38945936 Free article. Review.
Amyloid-beta (Abeta) plaques and neurofibrillary tangles containing phosphorylated tau protein are major hallmarks of Alzheimer's disease (AD). ...Recently, substantial breakthroughs have been achieved in the clinical development of Abeta antibo
Amyloid-beta (Abeta) plaques and neurofibrillary tangles containing phosphorylated tau protein are major hallmarks of Alzhe
Microglia in Alzheimer's Disease.
Süß P, Schlachetzki JCM. Süß P, et al. Curr Alzheimer Res. 2020;17(1):29-43. doi: 10.2174/1567205017666200212155234. Curr Alzheimer Res. 2020. PMID: 32048973 Review.
Alzheimer's Disease (AD) is the most frequent neurodegenerative disorder. Although proteinaceous aggregates of extracellular Amyloid-beta (Abeta) and intracellular hyperphosphorylated microtubule- associated tau have long been identified as char
Alzheimer's Disease (AD) is the most frequent neurodegenerative disorder. Although proteinaceous aggregates of extracel
11,826 results
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