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Year Number of Results
1986 1
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2012 2675
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2025 4078

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88,659 results

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Page 1
EMT Transition States during Tumor Progression and Metastasis.
Pastushenko I, Blanpain C. Pastushenko I, et al. Trends Cell Biol. 2019 Mar;29(3):212-226. doi: 10.1016/j.tcb.2018.12.001. Epub 2018 Dec 26. Trends Cell Biol. 2019. PMID: 30594349 Review.
Epithelial-mesenchymal transition (EMT) is a process in which epithelial cells acquire mesenchymal features. ...
Epithelial-mesenchymal transition (EMT) is a process in which epithelial cells acquire mesenchymal features. ...
The molecular mechanisms and therapeutic strategies of EMT in tumor progression and metastasis.
Huang Y, Hong W, Wei X. Huang Y, et al. J Hematol Oncol. 2022 Sep 8;15(1):129. doi: 10.1186/s13045-022-01347-8. J Hematol Oncol. 2022. PMID: 36076302 Free PMC article. Review.
Epithelial-mesenchymal transition (EMT) is an essential process in normal embryonic development and tissue regeneration. However, aberrant reactivation of EMT is associated with malignant properties of tumor cells during cancer progression and metastasis, inc …
Epithelial-mesenchymal transition (EMT) is an essential process in normal embryonic development and tissue regeneration …
Dynamic EMT: a multi-tool for tumor progression.
Brabletz S, Schuhwerk H, Brabletz T, Stemmler MP. Brabletz S, et al. EMBO J. 2021 Sep 15;40(18):e108647. doi: 10.15252/embj.2021108647. Epub 2021 Aug 30. EMBO J. 2021. PMID: 34459003 Free PMC article. Review.
The process of epithelial-mesenchymal transition (EMT) is fundamental for embryonic morphogenesis. Cells undergoing it lose epithelial characteristics and integrity, acquire mesenchymal features, and become motile. ...In addition, EMT is increasingly understo …
The process of epithelial-mesenchymal transition (EMT) is fundamental for embryonic morphogenesis. Cells undergoing it …
WNT5A signaling impairs breast cancer cell migration and invasion via mechanisms independent of the epithelial-mesenchymal transition.
Prasad CP, Chaurasiya SK, Guilmain W, Andersson T. Prasad CP, et al. J Exp Clin Cancer Res. 2016 Sep 13;35(1):144. doi: 10.1186/s13046-016-0421-0. J Exp Clin Cancer Res. 2016. PMID: 27623766 Free PMC article.
Moreover, knocking down CD44 in breast cancer cells using siRNA impaired cell migration and invasion. CONCLUSIONS: WNT5A bi-directionally regulates EMT in mammary epithelial cells, thereby affecting their migration and invasion. However, the ability of …
Moreover, knocking down CD44 in breast cancer cells using siRNA impaired cell migration and invasion. CONCLUSION …
CARM1 drives triple-negative breast cancer progression by coordinating with HIF1A.
Feng D, Gao J, Liu R, Liu W, Gao T, Yang Y, Zhang D, Yang T, Yin X, Yu H, Huang W, Wang Y. Feng D, et al. Protein Cell. 2024 Oct 1;15(10):744-765. doi: 10.1093/procel/pwae010. Protein Cell. 2024. PMID: 38476024 Free PMC article.
Coactivator-associated arginine methyltransferase 1 (CARM1) promotes the development and metastasis of estrogen receptor alpha (ERalpha)-positive breast cancer. The function of CARM1 in triple-negative breast cancer (TNBC) is still unclear and requires …
Coactivator-associated arginine methyltransferase 1 (CARM1) promotes the development and metastasis of estrogen receptor alpha (ERalpha)-pos …
Curcumin inhibits the invasion and metastasis of triple negative breast cancer via Hedgehog/Gli1 signaling pathway.
Li M, Guo T, Lin J, Huang X, Ke Q, Wu Y, Fang C, Hu C. Li M, et al. J Ethnopharmacol. 2022 Jan 30;283:114689. doi: 10.1016/j.jep.2021.114689. Epub 2021 Sep 28. J Ethnopharmacol. 2022. PMID: 34592340
AIM OF THE STUDY: To explore the mechanism of curcumin and Glioma-associated oncogene homolod-1 (Gli1) on invasion and metastasis of triple negative breast cancer (TNBC) cells through the Hedgehog (Hh)/Gli signaling pathway. ...RT-qPCR and Western blot …
AIM OF THE STUDY: To explore the mechanism of curcumin and Glioma-associated oncogene homolod-1 (Gli1) on invasion and metastasis of trip …
MCT-1/miR-34a/IL-6/IL-6R signaling axis promotes EMT progression, cancer stemness and M2 macrophage polarization in triple-negative breast cancer.
Weng YS, Tseng HY, Chen YA, Shen PC, Al Haq AT, Chen LM, Tung YC, Hsu HL. Weng YS, et al. Mol Cancer. 2019 Mar 18;18(1):42. doi: 10.1186/s12943-019-0988-0. Mol Cancer. 2019. PMID: 30885232 Free PMC article.
BACKGROUND: Triple-negative breast cancer (TNBC) is a poor prognostic breast cancer with the highest mutations and limited therapeutic choices. ...Overexpressing MCT-1 perturbed the oncogenic breast epithelial acini morphogenesis and stimulated epit …
BACKGROUND: Triple-negative breast cancer (TNBC) is a poor prognostic breast cancer with the highest mutations a …
Cell stemness, epithelial-to-mesenchymal transition, and immunoevasion: Intertwined aspects in cancer metastasis.
Romano S, Tufano M, D'Arrigo P, Vigorito V, Russo S, Romano MF. Romano S, et al. Semin Cancer Biol. 2020 Feb;60:181-190. doi: 10.1016/j.semcancer.2019.08.015. Epub 2019 Aug 15. Semin Cancer Biol. 2020. PMID: 31422157 Free article. Review.
Recent advances in tumor immunology, fostered by dramatic outcomes with cancer immunotherapy, have opened new scenarios in cancer metastasis. The cancer stemness/mesenchymal phenotype and an excess of immune suppressive signals are emerging as Intertwined aspects of human …
Recent advances in tumor immunology, fostered by dramatic outcomes with cancer immunotherapy, have opened new scenarios in cancer metastasis …
EMT: An Update.
Thiery JP. Thiery JP. Methods Mol Biol. 2021;2179:35-39. doi: 10.1007/978-1-0716-0779-4_6. Methods Mol Biol. 2021. PMID: 32939712
Epithelial Mesenchymal Transition (EMT) initially discovered as a key developmental mechanism is now shown to be indirectly involved in fibrosis and is contributing to the progression of carcinomas. ...
Epithelial Mesenchymal Transition (EMT) initially discovered as a key developmental mechanism is now shown to be indire …
Targeted therapy approaches for epithelial-mesenchymal transition in triple negative breast cancer.
Haque M, Shyanti RK, Mishra MK. Haque M, et al. Front Oncol. 2024 Oct 10;14:1431418. doi: 10.3389/fonc.2024.1431418. eCollection 2024. Front Oncol. 2024. PMID: 39450256 Free PMC article. Review.
Triple-negative breast cancer (TNBC) is distinguished by negative expression of estrogen receptor (ER), progesterone receptor (PR), and human epidermal growth factor receptor 2 (HER2), making it an aggressive subtype of breast cancer and contributes to …
Triple-negative breast cancer (TNBC) is distinguished by negative expression of estrogen receptor (ER), progeste …
88,659 results
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