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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1939 1
1941 1
1942 2
1945 2
1946 17
1947 5
1948 16
1949 19
1950 34
1951 31
1952 25
1953 28
1954 19
1955 31
1956 54
1957 58
1958 52
1959 86
1960 61
1961 91
1962 106
1963 141
1964 157
1965 153
1966 126
1967 190
1968 213
1969 210
1970 282
1971 212
1972 237
1973 262
1974 255
1975 246
1976 304
1977 262
1978 280
1979 336
1980 335
1981 323
1982 360
1983 400
1984 404
1985 470
1986 521
1987 516
1988 614
1989 654
1990 784
1991 835
1992 917
1993 867
1994 956
1995 1038
1996 1050
1997 1040
1998 1026
1999 1096
2000 1187
2001 1242
2002 1282
2003 1265
2004 1300
2005 1426
2006 1440
2007 1486
2008 1535
2009 1577
2010 1810
2011 1872
2012 2021
2013 2121
2014 2249
2015 2330
2016 2377
2017 2312
2018 2465
2019 2474
2020 1846
2021 1
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52,351 results
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Page 1
Cystic fibrosis.
Elborn JS. Elborn JS. Lancet. 2016 Nov 19;388(10059):2519-2531. doi: 10.1016/S0140-6736(16)00576-6. Epub 2016 Apr 29. Lancet. 2016. PMID: 27140670 Review.
Cystic fibrosis is a common life-limiting autosomal recessive genetic disorder, with highest prevalence in Europe, North America, and Australia. ...Cystic fibrosis affects several body systems, and morbidity and mortality is mostly caused by bronchiect …
Cystic fibrosis is a common life-limiting autosomal recessive genetic disorder, with highest prevalence in Europe, North Ameri …
Cystic fibrosis year in review 2016.
Savant AP, McColley SA. Savant AP, et al. Pediatr Pulmonol. 2017 Aug;52(8):1092-1102. doi: 10.1002/ppul.23747. Epub 2017 Jun 13. Pediatr Pulmonol. 2017. PMID: 28608632 Review.
In this article, we highlight cystic fibrosis (CF) research and case reports published in Pediatric Pulmonology during 2016. ...
In this article, we highlight cystic fibrosis (CF) research and case reports published in Pediatric Pulmonology during 2016. . …
Cystic fibrosis from the gastroenterologist's perspective.
Ooi CY, Durie PR. Ooi CY, et al. Nat Rev Gastroenterol Hepatol. 2016 Mar;13(3):175-85. doi: 10.1038/nrgastro.2015.226. Epub 2016 Jan 21. Nat Rev Gastroenterol Hepatol. 2016. PMID: 26790364 Review.
Cystic fibrosis is a life-limiting, recessive disease caused by mutations in the cystic fibrosis transmembrane conductance regulator (CFTR) gene. ...Manifestations are either a direct consequence of the primary defect in cystic fibrosis o …
Cystic fibrosis is a life-limiting, recessive disease caused by mutations in the cystic fibrosis transmembrane c …
Cystic fibrosis: a clinical view.
Castellani C, Assael BM. Castellani C, et al. Cell Mol Life Sci. 2017 Jan;74(1):129-140. doi: 10.1007/s00018-016-2393-9. Epub 2016 Oct 5. Cell Mol Life Sci. 2017. PMID: 27709245 Review.
Cystic fibrosis (CF), a monogenic disease caused by mutations in the CFTR gene on chromosome 7, is complex and greatly variable in clinical expression. ...
Cystic fibrosis (CF), a monogenic disease caused by mutations in the CFTR gene on chromosome 7, is complex and greatly variabl …
[Historical compilation of cystic fibrosis].
Navarro S. Navarro S. Gastroenterol Hepatol. 2016 Jan;39(1):36-42. doi: 10.1016/j.gastrohep.2015.04.012. Epub 2015 Jun 10. Gastroenterol Hepatol. 2016. PMID: 26070393 Review. Spanish.
Cystic fibrosis is the most common life-shortening recessively inherited disorder in the Caucasian population. The genetic mutation that most frequently provokes cystic fibrosis (ΔF508) appeared at least 53,000years ago. ...
Cystic fibrosis is the most common life-shortening recessively inherited disorder in the Caucasian population. The genetic mut …
Cystic fibrosis.
O'Sullivan BP, Freedman SD. O'Sullivan BP, et al. Lancet. 2009 May 30;373(9678):1891-904. doi: 10.1016/S0140-6736(09)60327-5. Epub 2009 May 4. Lancet. 2009. PMID: 19403164 Review.
Researchers now have a more complete understanding of the molecular-biological defect that underlies cystic fibrosis, which is leading to new approaches to treatment. ...We review clinical care for cystic fibrosis and discuss recent advances in the und …
Researchers now have a more complete understanding of the molecular-biological defect that underlies cystic fibrosis, which is …
The cystic fibrosis transmembrane conductance regulator (CFTR) and its stability.
Meng X, Clews J, Kargas V, Wang X, Ford RC. Meng X, et al. Cell Mol Life Sci. 2017 Jan;74(1):23-38. doi: 10.1007/s00018-016-2386-8. Epub 2016 Oct 12. Cell Mol Life Sci. 2017. PMID: 27734094 Free PMC article. Review.
The cystic fibrosis transmembrane conductance regulator (CFTR) is responsible for the disease cystic fibrosis (CF). It is a membrane protein belonging to the ABC transporter family functioning as a chloride/anion channel in epithelial cells around the …
The cystic fibrosis transmembrane conductance regulator (CFTR) is responsible for the disease cystic fibrosis (C …
Cystic fibrosis.
Ratjen F, Döring G. Ratjen F, et al. Lancet. 2003 Feb 22;361(9358):681-9. doi: 10.1016/S0140-6736(03)12567-6. Lancet. 2003. PMID: 12606185 Review.
Cystic fibrosis is the most common autosomal recessive disorder in white people, with a frequency of about 1 in 2500 livebirths. ...Gene replacement therapy is still far from being used in patients with cystic fibrosis, mostly because of difficulties o …
Cystic fibrosis is the most common autosomal recessive disorder in white people, with a frequency of about 1 in 2500 livebirth …
The structural basis of cystic fibrosis.
Meng X, Clews J, Martin ER, Ciuta AD, Ford RC. Meng X, et al. Biochem Soc Trans. 2018 Oct 19;46(5):1093-1098. doi: 10.1042/BST20180296. Epub 2018 Aug 28. Biochem Soc Trans. 2018. PMID: 30154098 Review.
Mutations in CFTR cause the inherited disease cystic fibrosis (CF), the most common inherited condition in humans of European descent [Science (1989) 245, 1066-1073; Pflugers Arch. (2007) 453, 555-567]. ...
Mutations in CFTR cause the inherited disease cystic fibrosis (CF), the most common inherited condition in humans of European …
Cystic fibrosis.
Paranjape SM, Mogayzel PJ Jr. Paranjape SM, et al. Pediatr Rev. 2014 May;35(5):194-205. doi: 10.1542/pir.35-5-194. Pediatr Rev. 2014. PMID: 24790073 No abstract available.
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