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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1964 1
1965 8
1966 17
1967 21
1968 12
1969 21
1970 21
1971 28
1972 29
1973 29
1974 28
1975 19
1976 28
1977 15
1978 23
1979 13
1980 18
1981 15
1982 16
1983 16
1984 28
1985 36
1986 21
1987 27
1988 25
1989 37
1990 36
1991 38
1992 60
1993 48
1994 42
1995 46
1996 48
1997 24
1998 24
1999 32
2000 25
2001 30
2002 27
2003 17
2004 16
2005 25
2006 35
2007 37
2008 23
2009 25
2010 34
2011 25
2012 22
2013 20
2014 28
2015 19
2016 14
2017 9
2018 9
2019 6
2020 0
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1,374 results
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Page 1
[Risk factors for adverse course of gastric and duodenal peptic ulcer].
Komar OM, et al. Wiad Lek 2018. PMID 29602925 Russian.
The aim of the study is to assess the influence of the main risk factors on the course of gastric and duodenal peptic ulcer. ...The risk of peptic ulcer in hereditary tainted young men of the working age with parental lineage (+2.3) and in males with 0(I) Rh+ blood type (+1.4) was proved. ...
The aim of the study is to assess the influence of the main risk factors on the course of gastric and duodenal peptic ulcer. . …
Dragstedt, gastric acid and duodenal ulcer.
Hobsley M. Yale J Biol Med 1994 - Review. PMID 7502527 Free PMC article.
Dragstedt believed that basal hypersecretion of gastric acid was the root cause of duodenal ulcer, that the hypersecretion was due to an increased vagal stimulation, and that vagotomy would therefore cure duodenal ulcer. He introduced vagotomy and demonstrated that the operation was successful in curing most patients of their duodenal ulcers. This article reviews how further research in the succeeding half century has demonstrated that it is the effect of vagotomy on stimulated, rather than upon basal secretion that cures duodenal ulcer and that the apparent basal hypersecretion of patients with duodenal ulcer is due to an increased parietal cell mass. ...
Dragstedt believed that basal hypersecretion of gastric acid was the root cause of duodenal ulcer, that the hypersecretion was …
Time trends in epidemiology of peptic ulcer disease in India over two decades.
Dutta AK, et al. Indian J Gastroenterol 2012. PMID 22766645
The decline was more marked for duodenal ulcer, and the ratio of duodenal to gastric ulcer declined from 2.7 in 1988 to 1.1 in 2008. CONCLUSIONS: The epidemiology of PUD in India may have changed in the past two decades with the incidence of duodenal ulcer declining more rapidly than that of gastric ulcer....
The decline was more marked for duodenal ulcer, and the ratio of duodenal to gastric ulcer declined from 2.7 in …
The incidence of gastric ulcer and duodenal ulcer in north Norway. A prospective epidemiological study.
Ostensen H, et al. Scand J Gastroenterol 1985. PMID 3992175
From a prospective epidemiological study of peptic ulcer disease in the northern part of Norway, incidence rates for gastric and duodenal ulcer are presented. ...The ratio of gastric ulcer to duodenal ulcer is still 1 to 1.1 for both sexes, 1 to 0.9 in women, and 1 to 1.14 in men in the northern part of Norway. ...
From a prospective epidemiological study of peptic ulcer disease in the northern part of Norway, incidence rates for gastric a …
Duodenal ulcer in childhood.
Rosenlund ML and Koop CE. Pediatrics 1970. PMID 5308689
Are duodenal ulcer seasonal fluctuations paralleled by seasonal changes in 24-hour gastric acidity and Helicobacter pylori infection?
Savarino V, et al. J Clin Gastroenterol 1996. PMID 8724253
The percentage of H. pylori-positive duodenal ulcer patients was then calculated for each season. The calendar fluctuation of duodenal ulcer occurrence showed an evident increase (p < 0.001) in fall (October-December) and in winter (January-March) compared with spring (April-June) and summer (July-September). ...We conclude that there was no parallel circannual fluctuation of duodenal ulcer, gastric acidity, and H. pylori infection in the restricted sample of patients we studied. ...
The percentage of H. pylori-positive duodenal ulcer patients was then calculated for each season. The calendar fluctuation of …
Occupational mortality from gastric and duodenal ulcer.
Sonnenberg A and Sonnenberg GS. Br J Ind Med 1986. PMID 3947561 Free PMC article.
Married women displayed a gradient of gastric ulcer mortality increasing from social class 1 to social class 5 but gave no evidence of social class correlation with respect to duodenal ulcer. ...In duodenal ulcer the varying extent of energy expenditure among different occupations may be responsible for the different risk of contracting duodenal ulcer and dying from it. ...
Married women displayed a gradient of gastric ulcer mortality increasing from social class 1 to social class 5 but gave no evidence o …
Duodenal ulcer in Johannesburg.
Cooke SA. Br Med J 1978. PMID 647234 Free PMC article.
Prevalence of non-Helicobacter pylori duodenal ulcer in Karachi, Pakistan.
Yakoob J, et al. World J Gastroenterol 2005. PMID 15962375 Free PMC article.
AIM: To determine the prevalence of non-Helicobacter pylori (H pylori)-related duodenal ulcer in patients with acid-peptic diseases. ...In the presence of co-morbids, non-H pylori and non-NSAID duodenal ulcer is likely to be present....
AIM: To determine the prevalence of non-Helicobacter pylori (H pylori)-related duodenal ulcer in patients with acid-peptic dis …
Relationship between the birth cohort pattern of Helicobacter pylori infection and the epidemiology of duodenal ulcer.
Harvey RF, et al. QJM 2002. PMID 12145391
BACKGROUND: Helicobacter-pylori-related duodenal ulcer (DU) is an important cause of dyspepsia. AIM: To determine the relationship between the pattern of H. pylori infection and the epidemiology of duodenal ulcer in a single population. ...DISCUSSION: Duodenal ulcer prevalence (as judged by the rate of referral of duodenal ulcer patients for endoscopy) is determined principally by the distribution of H. pylori infection in the local population. ...
BACKGROUND: Helicobacter-pylori-related duodenal ulcer (DU) is an important cause of dyspepsia. AIM: To determine the relation …
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