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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1947 1
1949 1
1951 4
1952 13
1953 21
1954 12
1955 5
1956 11
1957 19
1958 5
1959 8
1960 17
1961 10
1962 8
1964 4
1965 22
1966 54
1967 71
1968 95
1969 108
1970 120
1971 118
1972 124
1973 118
1974 133
1975 126
1976 130
1977 125
1978 137
1979 153
1980 147
1981 138
1982 163
1983 156
1984 176
1985 178
1986 169
1987 203
1988 170
1989 187
1990 218
1991 217
1992 201
1993 236
1994 204
1995 201
1996 213
1997 191
1998 243
1999 214
2000 235
2001 257
2002 287
2003 295
2004 291
2005 328
2006 343
2007 354
2008 425
2009 395
2010 434
2011 462
2012 436
2013 433
2014 448
2015 494
2016 461
2017 434
2018 468
2019 217
2020 2
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12,311 results
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Page 1
How I treat hypoxia in adults with hemoglobinopathies and hemolytic disorders.
Machogu EM and Machado RF. Blood 2018 - Review. PMID 30206115 Free PMC article.
Hemoglobinopathies are caused by genetic mutations that result in abnormal hemoglobin molecules, resulting in hemolytic anemia. Chronic complications involving the lung parenchyma, vasculature, and cardiac function in hemoglobinopathies result in impaired gas exchange, resulting in tissue hypoxia. ...
Hemoglobinopathies are caused by genetic mutations that result in abnormal hemoglobin molecules, resulting in hemolytic anemia
Sickle cell disease: Clinical presentation and management of a global health challenge
Houwing ME, et al. Blood Rev 2019 - Review. PMID 31128863
This review provides an extensive summary of the pathophysiology and management of sickle cell disease and encompasses the characteristics, complications and current and future treatment options of the disease....
This review provides an extensive summary of the pathophysiology and management of sickle cell disease and encompasses the characteristics, …
Haemoglobinopathies and the rheumatologist
Hughes M, et al. Rheumatology (Oxford) 2016 - Review. PMID 27018056
Musculoskeletal (MSK) complications are common in patients with haemoglobinopathies and may affect the whole of the MSK system, in addition to bone, which is the primary site of the disease. ...Rheumatologists need to be aware of the range of MSK complications, treatment challenges and the need for such patients to be managed as part of a dedicated multidisciplinary team alongside haematology....
Musculoskeletal (MSK) complications are common in patients with haemoglobinopathies and may affect the whole of the MSK system, in ad …
[Cardiac complications of sickle cell disease in children].
Astadicko I, et al. Rev Med Liege 2018. PMID 30431243 French. Free article.
of the hemoglobin. The main clinical manifestations related to the major forms of SCD (SS-, SC-, and S-thalassemia) are chronic hemolysis, susceptibility to infections and vasoconstrictive crisis causing micro-emboli and/or infarction responsible for acute or chronic organ lesions. ...In children, cardiac complications of SCD are not sufficiently identified. They principally consist in dilated or restrictive cardiomyopathy and pulmonary arterial hypertension. ...
of the hemoglobin. The main clinical manifestations related to the major forms of SCD (SS-, SC-, and S-thalassemia) are chronic hemol …
Thalassemia and malignancy: An emerging concern?
Hodroj MH, et al. Blood Rev 2019 - Review. PMID 31253373
The thalassemias constitute a variable group of anemias that result from autosomal recessive inherited defects in the production of hemoglobin. ...
The thalassemias constitute a variable group of anemias that result from autosomal recessive inherited defects in the production of hemog
Genetic modifiers of severity in sickle cell disease
Chang AK, et al. Clin Hemorheol Microcirc 2018. PMID 29614629
While the effects of higher fetal hemoglobin (HbF) levels, UGTA1A polymorphisms, alpha-thalassemia and G6PD deficiency on SCD has been extensively studied, these variables do not explain all of the clinical heterogeneity of SCD. It is not known why some patients develop certain complications, and it is difficult to predict which complications a particular patient will experience. ...
While the effects of higher fetal hemoglobin (HbF) levels, UGTA1A polymorphisms, alpha-thalassemia and G6PD deficiency on SCD has bee …
Treatment of dental complications in sickle cell disease.
Mulimani P, et al. Cochrane Database Syst Rev 2016 - Review. PMID 27103509
Updated
Management of dental complications in people with sickle cell disease requires special consideration for three main reasons. ...Finally, the treatment of these oral complications must be adapted to the systemic condition and special needs of these individuals, in order not to exacerbate or deteriorate their general health.Guidelines for the treatment of dental complications in this population who require special care are unclear and even unavailable in many aspects. ...
Management of dental complications in people with sickle cell disease requires special consideration for three main reasons. ...Final …
Rare Association of Hb D-Los Angeles (HBB: c.364G>C) with Hb H Disease: Diagnosis and Clinical Implications
Zioga A, et al. Hemoglobin 2018. PMID 30626242
Heterozygous or homozygous inheritance does not cause significant medical problems, whereas association with other hemoglobinopathies, especially β-thalassemia (β-thal) and sickle cell disease, changes the phenotype. ...
Heterozygous or homozygous inheritance does not cause significant medical problems, whereas association with other hemoglobinopathies
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