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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1965 2
1966 1
1967 1
1968 2
1969 1
1970 5
1971 1
1972 2
1973 3
1974 1
1975 1
1976 2
1977 4
1978 1
1979 2
1980 2
1981 1
1982 3
1983 4
1984 5
1985 4
1986 9
1987 6
1988 9
1989 17
1990 12
1991 10
1992 22
1993 19
1994 19
1995 27
1996 28
1997 25
1998 12
1999 22
2000 15
2001 9
2002 12
2003 9
2004 10
2005 6
2006 10
2007 6
2008 9
2009 2
2010 3
2011 5
2012 2
2013 4
2014 4
2015 8
2016 3
2017 1
2018 2
2019 1
2020 0
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394 results
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Page 1
Disturbance of hepatic and intestinal microcirculation in experimental liver cirrhosis.
Maksan SM, et al. World J Gastroenterol 2005. PMID 15682478 Free PMC article.
AIM: To analyze hepatic, mesenteric and mucosal microcirculation and leukocyte-endothelium interaction (LEI) in a rat model with liver cirrhosis. ...Special emphasis is given on microcirculatory and morphometric changes during cirrhotic portal hypertension. RESULTS: LEI was influenced significantly in the cirrhotic liver but not in the gut. ...
AIM: To analyze hepatic, mesenteric and mucosal microcirculation and leukocyte-endothelium interaction (LEI) in a rat model with liver
Extrahepatic angiogenesis hinders recovery of portal hypertension and collaterals in rats with cirrhosis resolution.
Hsu SJ, et al. Clin Sci (Lond) 2018. PMID 29449343
Liver cirrhosis is characterized by portal hypertension. However, the alteration of portal hypertension-related derangements during cirrhosis resolution is not well known. ...In reverse thioacetamide (rTAA) model, rats were randomly allocated into four groups with control, thioacetamide (TAA) cirrhosis and rTAA groups that discontinued TAA for 4 or 8 weeks after cirrhosis induction. ...
Liver cirrhosis is characterized by portal hypertension. However, the alteration of portal hypertension-related derangements d
Dogs with experimental cirrhosis of the liver but without intrahepatic hypertension do not retain sodium or form ascites.
Unikowsky B, et al. J Clin Invest 1983. PMID 6630520 Free PMC article.
Dogs with portal cirrhosis but without portal hypertension (end-to-side portacaval anastomosis) retain sodium and expand plasma volume before ascites formation. ...Both groups were equally cirrhotic, as judged by histological and biochemical parameters. We conclude that normalizing intrahepatic pressure by providing an outflow tract for the cirrhotic liver will abolish that component of early renal tubular sodium retention not due to portal venous hypertension or ascites sequestration....
Dogs with portal cirrhosis but without portal hypertension (end-to-side portacaval anastomosis) retain sodium and expand plasma volum …
[Endotoxemia and its compensatory mechanisms in experimental liver cirrhosis].
Ueno M. Nihon Shokakibyo Gakkai Zasshi 1990. PMID 2232302 Japanese.
Both the plasma and biliary endotoxin levels in liver cirrhosis were significantly higher, compared to those in control. The functional activities of the reticuloendothelial system, particularly of the Kupffer cells, were decreased in liver cirrhosis. These data provide evidence that in liver cirrhosis systemic endotoxemia is mainly due to a decrease of functional activities of Kupffer cells. ...
Both the plasma and biliary endotoxin levels in liver cirrhosis were significantly higher, compared to those in control. The f …
Experimental liver cirrhosis does not influence reproductive performance of female rats and development of hepatic biotransformation in their offspring.
Glöckner R and Zimmermann T. Exp Pathol 1987. PMID 3569482
At the time the body mass of cirrhotic rats was not different from that of untreated controls. Body mass gain during pregnancy, length of gestation, litter size and body mass of the newborns were not influenced by experimental cirrhosis. ...Liver mass was enhanced in TAA-offspring at birth, but not any longer in 10-d-old and older rats....
At the time the body mass of cirrhotic rats was not different from that of untreated controls. Body mass gain during pregnancy, lengt …
Effects of hepatic blood inflow occlusion on liver regeneration following partial hepatectomy in an experimental model of cirrhosis.
Yin XY, et al. Br J Surg 2000. PMID 11091238
BACKGROUND: Hepatic blood inflow occlusion during hepatectomy may influence postoperative liver regeneration. The aim of this study was to investigate the influence of hepatic blood inflow occlusion on liver regeneration following partial hepatectomy in thioacetamide-induced cirrhotic rats. ...Percentages of initial liver weight were similar in groups 1, 2 and 3 on days 1, 2 and 7 after hepatectomy. CONCLUSION: Hepatic blood inflow occlusion for up to 30 min suppressed DNA synthesis and hepatocyte proliferation at an early posthepatectomy stage and consequently delayed recovery of liver function in cirrhotic rats. ...
BACKGROUND: Hepatic blood inflow occlusion during hepatectomy may influence postoperative liver regeneration. The aim of this study w …
Pathogenesis of solute-free water retention in experimental ascitic cirrhosis: is vasopressin the only culprit?
Sansoè G, et al. Clin Sci (Lond) 2016. PMID 26519424
To test the hypothesis that water retention in experimental ascitic cirrhosis might depend primarily on adrenergic hyper-function, hormonal status, renal function and tubular free-water reabsorption (TFWR) were assessed in six groups of rats with ascitic cirrhosis: rats with cirrhosis due to 13-week CCl4 (carbon tetrachloride) administration (group G1); cirrhotic rats receiving daily diuretics (0.5 mg/kg furosemide plus 2 mg/kg K(+)-canrenoate) from the 11th to the 13th week of CCl4 (G2), diuretics associated with guanfacine oral prodrug (α2A-adrenergic receptor agonist and sympatholytic agent) at 2 (G3), 7 (G4) or 10 (G5) mg/kg, or with SSP-004240F1 (V2 receptor antagonist) at 1 mg/kg (G6). ...In ascitic cirrhosis, reduced volaemia, use of diuretics (especially furosemide) and adrenergic hyper-function cause tubular retention of water. ...
To test the hypothesis that water retention in experimental ascitic cirrhosis might depend primarily on adrenergic hyper-function, ho …
Hemodynamic Effects of the Non-Peptidic Angiotensin-(1-7) Agonist AVE0991 in Liver Cirrhosis.
Klein S, et al. PLoS One 2015. PMID 26406236 Free PMC article.
Therefore, we investigated the effects of the non-peptidic Ang-(1-7) agonist, AVE0991, in experimental cirrhosis. METHODS: Cirrhosis was induced by bile duct ligation (BDL) or carbon tetrachloride (CCl4) intoxication. ...RESULTS: In vivo, AVE0991 decreased portal pressure (PP) in both rat models of cirrhosis. Importantly, systemic effects were not observed. ...
Therefore, we investigated the effects of the non-peptidic Ang-(1-7) agonist, AVE0991, in experimental cirrhosis. METHODS: Cirrhos
Pulmonary intravascular phagocytosis in liver disease.
Chang SW and Ohara N. Clin Chest Med 1996 - Review. PMID 8665786
Pulmonary intravascular phagocytosis, the uptake of circulating particles by lung cells, has been detected in rats with chronic biliary cirrhosis and in humans with malignancy and liver diseases. ...A hypothesis is asserted that the induction of pulmonary intravascular macrophage in liver cirrhosis leads to increased susceptibility to adult respiratory distress syndrome in these patients....
Pulmonary intravascular phagocytosis, the uptake of circulating particles by lung cells, has been detected in rats with chronic biliary c
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