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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1951 1
1952 7
1953 2
1954 4
1955 1
1956 3
1957 5
1959 1
1962 1
1964 2
1965 22
1966 43
1967 68
1968 83
1969 74
1970 83
1971 100
1972 117
1973 105
1974 129
1975 103
1976 122
1977 98
1978 65
1979 72
1980 88
1981 89
1982 71
1983 73
1984 84
1985 117
1986 113
1987 144
1988 157
1989 191
1990 196
1991 199
1992 220
1993 227
1994 398
1995 642
1996 616
1997 665
1998 698
1999 720
2000 748
2001 739
2002 737
2003 785
2004 742
2005 817
2006 825
2007 902
2008 958
2009 1046
2010 1013
2011 1092
2012 1131
2013 1148
2014 1108
2015 1092
2016 1053
2017 1091
2018 999
2019 455
2020 10
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23,146 results
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Page 1
Skeletal muscle fiber type-selective effects of acute exercise on insulin-stimulated glucose uptake in insulin-resistant, high-fat-fed rats.
Pataky MW, et al. Am J Physiol Endocrinol Metab 2019. PMID 30753114 Free PMC article.
Insulin-stimulated glucose uptake (GU) by skeletal muscle is enhanced several hours after acute exercise in rats with normal or reduced insulin sensitivity. Skeletal muscle is composed of multiple fiber types, but exercise's effect on fiber type-specific insulin-stimulated GU in insulin-resistant muscle was previously unknown. ...
Insulin-stimulated glucose uptake (GU) by skeletal muscle is enhanced several hours after acute exercise in rats with normal o …
Fibroblast growth factor 23 does not directly influence skeletal muscle cell proliferation and differentiation or ex vivo muscle contractility.
Avin KG, et al. Am J Physiol Endocrinol Metab 2018. PMID 29558205 Free PMC article.
Skeletal muscle dysfunction accompanies the clinical disorders of chronic kidney disease (CKD) and hereditary hypophosphatemic rickets. ...FGF23 has been shown to play a direct role in cardiac muscle dysfunction; however, it is unknown whether FGF23 signaling can also directly induce skeletal muscle dysfunction. ...
Skeletal muscle dysfunction accompanies the clinical disorders of chronic kidney disease (CKD) and hereditary hypophosphatemic
CK-2127107 amplifies skeletal muscle response to nerve activation in humans.
Andrews JA, et al. Muscle Nerve 2018 - Clinical Trial. PMID 29150952 Free PMC article.
INTRODUCTION: Three studies evaluated safety, tolerability, pharmacokinetics, and pharmacodynamics of CK-2127107 (CK-107), a next-generation fast skeletal muscle troponin activator (FSTA), in healthy participants. ...Muscle Nerve 57: 729-734, 2018....
INTRODUCTION: Three studies evaluated safety, tolerability, pharmacokinetics, and pharmacodynamics of CK-2127107 (CK-107), a next-generation …
Is there enough evidence to use botulinum toxin injections for bruxism management? A systematic literature review
De la Torre Canales G, et al. Clin Oral Investig 2017 - Review. PMID 28255752
OBJECTIVE: The objective of the study was to conduct a systematic review of the literature assessing the effects of botulinum toxin (BoNT-A) injections in the management of bruxism. ...Nonetheless, there is no consensus about its effects in this disorder....
OBJECTIVE: The objective of the study was to conduct a systematic review of the literature assessing the effects of botulinum toxin ( …
Effects of yessotoxin (YTX) on the skeletal muscle: an update.
Tubaro A, et al. Food Addit Contam Part A Chem Anal Control Expo Risk Assess 2008. PMID 19238620
The aim of this study was to investigate the possible effects of YTX on the other contractile striated tissue, the skeletal muscle, in vitro and in vivo. ...The observation that effects were detected in vitro but not in vivo supports the hypothesis of a low YTX bioavailability to skeletal muscle after oral intake. ...
The aim of this study was to investigate the possible effects of YTX on the other contractile striated tissue, the skeletal
β-hydroxy-β-methyl butyrate promotes leucine metabolism and improves muscle fibre composition in growing pigs.
Duan YH, et al. J Anim Physiol Anim Nutr (Berl) 2018. PMID 30009416
The aim of this study was to investigate the effects of excess leucine (Leu) vs. its metabolites α-ketoisocaproate (KIC) and β-hydroxy-β-methyl butyrate (HMB) on Leu metabolism, muscle fibre composition and muscle growth in growing pigs. ...These findings demonstrated that doubling dietary Leu content exerted growth-depressing effects in growing pigs; dietary KIC supplementation induced muscular branched-chain amino acid imbalance and promoted muscle toward a more glycolytic phenotype; while dietary HMB supplementation promoted the generation of more oxidative muscle types and increased muscle growth specially in oxidative skeletal muscle, and these effects of HMB might be associated with the AMPKα-Sirt1-PGC-1α axis and mitochondrial biogenesis....
The aim of this study was to investigate the effects of excess leucine (Leu) vs. its metabolites α-ketoisocaproate (KIC) and β-hydrox …
Arginine promotes skeletal muscle fiber type transformation from fast-twitch to slow-twitch via Sirt1/AMPK pathway.
Chen X, et al. J Nutr Biochem 2018. PMID 30236872
However, inhibition of AMPK activity by compound C significantly attenuated the effects of arginine on slow MyHC and fast MyHC expressions in C2C12 myotubes. ...Together, our findings indicated that arginine promotes skeletal muscle fiber type switching from fast-twitch to slow-twitch via Sirt1/AMPK pathway....
However, inhibition of AMPK activity by compound C significantly attenuated the effects of arginine on slow MyHC and fast MyHC expres …
Effects of myotoxins on skeletal muscle fibers.
Khan MA. Prog Neurobiol 1995 - Review. PMID 8532852
Despite their chemical diversity and mechanism(s) of action, it is evident from the data discussed here that remarkably different toxic agents exert quite similar effects and induce toxic myopathies. The latter include preferential involvement of slow-twitch red muscle, mitochondrial derangement, denervation-like alterations, formation of membranous whorls, tubular aggregates, autophagic vacuoles and axonal sprouts. ...
Despite their chemical diversity and mechanism(s) of action, it is evident from the data discussed here that remarkably different toxic agen …
Desensitizing mouse cardiac troponin C to calcium converts slow muscle towards a fast muscle phenotype.
Tikunova S, et al. J Physiol 2018. PMID 29992562 Free PMC article.
KEY POINTS: The Ca(2+) -desensitizing D73N mutation in slow skeletal/cardiac troponin C caused dilatated cardiomyopathy in mice, but the consequences of this mutation in skeletal muscle were not known. ...Effects of the D73N mutation on physiological properties of mouse soleus muscle, in which slow-twitch fibres are prevalent, were examined. ...
KEY POINTS: The Ca(2+) -desensitizing D73N mutation in slow skeletal/cardiac troponin C caused dilatated cardiomyopathy in mice, but …
23,146 results
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