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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1950 1
1952 1
1964 1
1993 1
1994 14
1995 12
1996 9
1997 17
1998 29
1999 21
2000 26
2001 28
2002 34
2003 25
2004 25
2005 29
2006 31
2007 34
2008 40
2009 29
2010 37
2011 35
2012 38
2013 44
2014 43
2015 42
2016 38
2017 39
2018 36
2019 15
2020 0
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702 results
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Page 1
Lipid binding promotes the open conformation and tumor-suppressive activity of neurofibromin 2.
Chinthalapudi K, et al. Nat Commun 2018. PMID 29626191 Free PMC article.
Neurofibromatosis type 2 (NF2) is a tumor-forming disease of the nervous system caused by deletion or by loss-of-function mutations in NF2, encoding the tumor suppressing protein neurofibromin 2 (also known as schwannomin or merlin). Neurofibromin 2 is a member of the ezrin, radixin, moesin (ERM) family of proteins regulating the cytoskeleton and cell signaling. ...
Neurofibromatosis type 2 (NF2) is a tumor-forming disease of the nervous system caused by deletion or by loss-of-function muta
Role of Merlin/NF2 inactivation in tumor biology
Petrilli AM and Fernández-Valle C. Oncogene 2016 - Review. PMID 25893302 Free PMC article.
Merlin (Moesin-ezrin-radixin-like protein, also known as schwannomin) is a tumor suppressor protein encoded by the neurofibromatosis type 2 gene NF2. Loss of function mutations or deletions in NF2 cause neurofibromatosis type 2 (NF2), a multiple tumor forming disease of the nervous system. ...
Merlin (Moesin-ezrin-radixin-like protein, also known as schwannomin) is a tumor suppressor protein encoded by the neurofibromatosis
NF2/Merlin Inactivation and Potential Therapeutic Targets in Mesothelioma
Sato T and Sekido Y. Int J Mol Sci 2018 - Review. PMID 29587439 Free PMC article.
The neurofibromatosis type 2 (NF2) gene encodes merlin, a tumor suppressor protein frequently inactivated in schwannoma, meningioma, and malignant mesothelioma (MM). ...
The neurofibromatosis type 2 (NF2) gene encodes merlin, a tumor suppressor protein frequently inactivated in schwannoma, menin …
Neurofibromatosis type 2.
Asthagiri AR, et al. Lancet 2009 - Review. PMID 19476995 Free PMC article.
Half of patients inherit a germline mutation from an affected parent and the remainder acquire a de novo mutation for neurofibromatosis type 2. ...We review the molecular pathogenesis, genetics, clinical findings, and management strategies for neurofibromatosis type 2....
Half of patients inherit a germline mutation from an affected parent and the remainder acquire a de novo mutation for neurofibromatosis
Genetic landscape of meningioma
Yuzawa S, et al. Brain Tumor Pathol 2016 - Review. PMID 27624470
Characterization of Hippo Pathway Components by Gene Inactivation
Plouffe SW, et al. Mol Cell 2016. PMID 27912098 Free PMC article.
By this approach, we provide an overview of the functional importance of many Hippo pathway components and demonstrate NF2 and RHOA as important regulators of YAP/TAZ and TAOK1/3 as direct kinases for LATS1/2....
By this approach, we provide an overview of the functional importance of many Hippo pathway components and demonstrate NF2 and RHOA as impor …
Genetic and pharmacological disruption of the TEAD-YAP complex suppresses the oncogenic activity of YAP
Liu-Chittenden Y, et al. Genes Dev 2012. PMID 22677547 Free PMC article.
We show that a dominant-negative TEAD molecule does not perturb normal liver growth but potently suppresses hepatomegaly/tumorigenesis resulting from YAP overexpression or Neurofibromin 2 (NF2)/Merlin inactivation. ...
We show that a dominant-negative TEAD molecule does not perturb normal liver growth but potently suppresses hepatomegaly/tumorigenesis resul …
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