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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1990 1
1992 3
1993 5
1994 3
1995 1
1996 3
1997 11
1998 5
1999 6
2000 6
2001 5
2002 4
2003 4
2004 5
2005 4
2006 6
2007 13
2008 12
2009 13
2010 12
2011 18
2012 15
2013 17
2014 19
2015 20
2016 21
2017 17
2018 19
2019 23
2020 21
2021 21
2022 27
2023 13

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327 results

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Page 1
Metabolic phenotype of bladder cancer.
Massari F, Ciccarese C, Santoni M, Iacovelli R, Mazzucchelli R, Piva F, Scarpelli M, Berardi R, Tortora G, Lopez-Beltran A, Cheng L, Montironi R. Massari F, et al. Cancer Treat Rev. 2016 Apr;45:46-57. doi: 10.1016/j.ctrv.2016.03.005. Epub 2016 Mar 8. Cancer Treat Rev. 2016. PMID: 26975021 Free article. Review.
Therefore, the high glycolytic flux depends on the overexpression of glycolysis-related genes (SRC-3, glucose transporter type 1 [GLUT1], GLUT3, lactic dehydrogenase A [LDHA], LDHB, hexokinase 1 [HK1], HK2, pyruvate kinase type M [PKM], and hypoxia-inducible factor 1-alpha …
Therefore, the high glycolytic flux depends on the overexpression of glycolysis-related genes (SRC-3, glucose transporter type 1 [GLUT1], …
AMPK modulates Hippo pathway activity to regulate energy homeostasis.
Wang W, Xiao ZD, Li X, Aziz KE, Gan B, Johnson RL, Chen J. Wang W, et al. Nat Cell Biol. 2015 Apr;17(4):490-9. doi: 10.1038/ncb3113. Epub 2015 Mar 9. Nat Cell Biol. 2015. PMID: 25751139 Free PMC article.
We show that both the Hippo pathway and AMP-activated protein kinase (AMPK) were activated during glucose starvation, resulting in phosphorylation of YAP and contributing to its inactivation. We also identified glucose-transporter 3 (GLUT3) as a YAP-regulated gene involved …
We show that both the Hippo pathway and AMP-activated protein kinase (AMPK) were activated during glucose starvation, resulting in phosphory …
Cystine transporter regulation of pentose phosphate pathway dependency and disulfide stress exposes a targetable metabolic vulnerability in cancer.
Liu X, Olszewski K, Zhang Y, Lim EW, Shi J, Zhang X, Zhang J, Lee H, Koppula P, Lei G, Zhuang L, You MJ, Fang B, Li W, Metallo CM, Poyurovsky MV, Gan B. Liu X, et al. Nat Cell Biol. 2020 Apr;22(4):476-486. doi: 10.1038/s41556-020-0496-x. Epub 2020 Mar 30. Nat Cell Biol. 2020. PMID: 32231310 Free PMC article.
An update on the molecular biology of glioblastoma, with clinical implications and progress in its treatment.
Verdugo E, Puerto I, Medina MÁ. Verdugo E, et al. Cancer Commun (Lond). 2022 Nov;42(11):1083-1111. doi: 10.1002/cac2.12361. Epub 2022 Sep 21. Cancer Commun (Lond). 2022. PMID: 36129048 Free PMC article. Review.
GBM's aggressive invasion potential is modulated by cell-to-cell crosstalk within the tumor microenvironment and altered expressions of specific genes, such as ANXA2, GBP2, FN1, PHIP, and GLUT3. Nevertheless, the rising number of active clinical trials illustrates the effo …
GBM's aggressive invasion potential is modulated by cell-to-cell crosstalk within the tumor microenvironment and altered expressions of spec …
GLUT3 induced by AMPK/CREB1 axis is key for withstanding energy stress and augments the efficacy of current colorectal cancer therapies.
Dai W, Xu Y, Mo S, Li Q, Yu J, Wang R, Ma Y, Ni Y, Xiang W, Han L, Zhang L, Cai S, Qin J, Chen WL, Jia W, Cai G. Dai W, et al. Signal Transduct Target Ther. 2020 Sep 2;5(1):177. doi: 10.1038/s41392-020-00220-9. Signal Transduct Target Ther. 2020. PMID: 32873793 Free PMC article.
Notably, GLUT3 had a greater impact on cell growth than GLUT1 under glucose-limiting stress. ...Together, the results of this study highlight the importance of the AMPK/CREB1/GLUT3 pathway for CRC cells to withstand glucose-limiting stress and underscore the therape …
Notably, GLUT3 had a greater impact on cell growth than GLUT1 under glucose-limiting stress. ...Together, the results of this study h …
miR-3189-targeted GLUT3 repression by HDAC2 knockdown inhibits glioblastoma tumorigenesis through regulating glucose metabolism and proliferation.
Kwak S, Park SH, Kim SH, Sung GJ, Song JH, Jeong JH, Kim H, Ha CH, Kim SW, Choi KC. Kwak S, et al. J Exp Clin Cancer Res. 2022 Mar 8;41(1):87. doi: 10.1186/s13046-022-02305-5. J Exp Clin Cancer Res. 2022. PMID: 35260183 Free PMC article.
Analysis of RNA sequencing and qRT-PCR reveals that miR-3189 increases and GLUT3 decreases by HDAC2 knockdown. GBM tumorigenesis also examines by using in vivo orthotopic xenograft tumor models. The metabolism change in HDAC2 knockdown GBM cells measures by glucose uptake, …
Analysis of RNA sequencing and qRT-PCR reveals that miR-3189 increases and GLUT3 decreases by HDAC2 knockdown. GBM tumorigenesis also …
Vitamin C as a Modulator of the Response to Cancer Therapy.
Blaszczak W, Barczak W, Masternak J, Kopczyński P, Zhitkovich A, Rubiś B. Blaszczak W, et al. Molecules. 2019 Jan 28;24(3):453. doi: 10.3390/molecules24030453. Molecules. 2019. PMID: 30695991 Free PMC article. Review.
Ginsenoside Rg3 inhibits angiogenesis in gastric precancerous lesions through downregulation of Glut1 and Glut4.
Zeng Z, Nian Q, Chen N, Zhao M, Zheng Q, Zhang G, Zhao Z, Chen Y, Wang J, Zeng J, Gong D, Tang J. Zeng Z, et al. Biomed Pharmacother. 2022 Jan;145:112086. doi: 10.1016/j.biopha.2021.112086. Epub 2021 Nov 16. Biomed Pharmacother. 2022. PMID: 34799220 Free article.
Data analysis revealed that enhanced protein expression of GLUT1, GLUT3 and GLUT4 were present in both human and animal GPL specimens. ...However, the GRg3-treated rats with GPLs did not demonstrate regulatory effects on GLUT3, GLUT6, GLUT10, and GLUT12. Consistent …
Data analysis revealed that enhanced protein expression of GLUT1, GLUT3 and GLUT4 were present in both human and animal GPL specimens …
327 results