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1996 1
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2015 3
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2017 4
2018 1
2019 3
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[Transglutaminase and neurodegenerative diseases].
Liu Z, Zeng J, Zeng S, Tang B, Wang J. Liu Z, et al. Zhonghua Yi Xue Yi Chuan Xue Za Zhi. 2015 Aug;32(4):562-6. doi: 10.3760/cma.j.issn.1003-9406.2015.04.025. Zhonghua Yi Xue Yi Chuan Xue Za Zhi. 2015. PMID: 26252108 Review. Chinese.
Notably, neurodegenerative diseases such as Huntington's disease, spinocerebellar ataxia, Alzheimer's disease and Parkinson's disease, have a close connection with TGase's role in the human body....
Notably, neurodegenerative diseases such as Huntington's disease, spinocerebellar ataxia, Alzheimer's disease and Parkinson's disease …
[Gluten Ataxia: Anti-Transglutaminase-6 Antibody as a New Biomarker].
Sato K, Nanri K. Sato K, et al. Brain Nerve. 2017 Aug;69(8):933-940. doi: 10.11477/mf.1416200845. Brain Nerve. 2017. PMID: 28819077 Review. Japanese.
Gluten ataxia is defined as an anti-gliadin antibody positive sporadic ataxia. ...In patients with gluten ataxia, sensory disturbance is generally considered to be so mild that it contributes minimally to ataxia. ...
Gluten ataxia is defined as an anti-gliadin antibody positive sporadic ataxia. ...In patients with gluten ataxia, senso …
Transglutaminase diseases: from biochemistry to the bedside.
Lorand L, Iismaa SE. Lorand L, et al. FASEB J. 2019 Jan;33(1):3-12. doi: 10.1096/fj.201801544R. FASEB J. 2019. PMID: 30593123
Loss of TG function is associated with numerous orphan diseases that affect a relatively small number of individuals: loss of FXIIIa (transamidase-activated form) crosslinking leads to defects in blood coagulation in FXIII deficiency; loss of TG1 and TG5 cross linking leads to de …
Loss of TG function is associated with numerous orphan diseases that affect a relatively small number of individuals: loss of FXIIIa (transa …
Transglutaminase 6 antibodies in gluten neuropathy.
Zis P, Rao DG, Sarrigiannis PG, Aeschlimann P, Aeschlimann DP, Sanders D, Grünewald RA, Hadjivassiliou M. Zis P, et al. Dig Liver Dis. 2017 Nov;49(11):1196-1200. doi: 10.1016/j.dld.2017.08.019. Epub 2017 Aug 10. Dig Liver Dis. 2017. PMID: 28886934
BACKGROUND: TG6 antibodies have been shown to be a marker of gluten ataxia but their presence in the context of other neurological manifestations of gluten sensitivity has not been explored. ...
BACKGROUND: TG6 antibodies have been shown to be a marker of gluten ataxia but their presence in the context of other neurological ma …
Autoantibody targeting of brain and intestinal transglutaminase in gluten ataxia.
Hadjivassiliou M, Mäki M, Sanders DS, Williamson CA, Grünewald RA, Woodroofe NM, Korponay-Szabó IR. Hadjivassiliou M, et al. Neurology. 2006 Feb 14;66(3):373-7. doi: 10.1212/01.wnl.0000196480.55601.3a. Neurology. 2006. PMID: 16476935
OBJECTIVE: To investigate the presence of autoantibody deposition against type 2 tissue transglutaminase (TG2; a reliable marker of the whole spectrum of gluten sensitivity) in the jejunal tissue and brain of patients with gluten ataxia and in control subjects. METHODS: Th …
OBJECTIVE: To investigate the presence of autoantibody deposition against type 2 tissue transglutaminase (TG2; a reliable marker of the whol …
Transglutaminase 6 antibodies in the diagnosis of gluten ataxia.
Hadjivassiliou M, Aeschlimann P, Sanders DS, Mäki M, Kaukinen K, Grünewald RA, Bandmann O, Woodroofe N, Haddock G, Aeschlimann DP. Hadjivassiliou M, et al. Neurology. 2013 May 7;80(19):1740-5. doi: 10.1212/WNL.0b013e3182919070. Epub 2013 Apr 10. Neurology. 2013. PMID: 23576621
The groups included patients with idiopathic sporadic ataxia, GA, and CD, and neurology and healthy controls. All were tested for TG6 antibodies. ...Forty-two percent of patients with GA had enteropathy as did 51% of patients with ataxia and TG6 antibodies. Five of …
The groups included patients with idiopathic sporadic ataxia, GA, and CD, and neurology and healthy controls. All were tested for TG6 …
Transglutaminase-dependent formation of protein aggregates as possible biochemical mechanism for polyglutamine diseases.
Violante V, Luongo A, Pepe I, Annunziata S, Gentile V. Violante V, et al. Brain Res Bull. 2001 Oct-Nov 1;56(3-4):169-72. doi: 10.1016/s0361-9230(01)00576-7. Brain Res Bull. 2001. PMID: 11719247 Review.
Recently, the transglutaminase activity has been hypothesized to be involved in the pathogenetic mechanisms responsible for the formation of cellular inclusions present in Huntington disease and in all the other polyglutamine (polyQ) diseases hitherto identified, such as spinobul …
Recently, the transglutaminase activity has been hypothesized to be involved in the pathogenetic mechanisms responsible for the formation of …
Anti transglutaminase antibodies cause ataxia in mice.
Boscolo S, Lorenzon A, Sblattero D, Florian F, Stebel M, Marzari R, Not T, Aeschlimann D, Ventura A, Hadjivassiliou M, Tongiorgi E. Boscolo S, et al. PLoS One. 2010 Mar 15;5(3):e9698. doi: 10.1371/journal.pone.0009698. PLoS One. 2010. PMID: 20300628 Free PMC article.
To determine the role of anti-TG2 antibodies in ataxia, two anti-TG2 single chain variable fragments (scFv), isolated from a phage-display IgA antibody library, were characterized by immunohistochemistry and ELISA, and injected in mice to study their effects on motor coord …
To determine the role of anti-TG2 antibodies in ataxia, two anti-TG2 single chain variable fragments (scFv), isolated from a phage-di …
Transglutaminase-6 is an autoantigen in progressive multiple sclerosis and is upregulated in reactive astrocytes.
Cristofanilli M, Gratch D, Pagano B, McDermott K, Huang J, Jian J, Bates D, Sadiq SA. Cristofanilli M, et al. Mult Scler. 2017 Nov;23(13):1707-1715. doi: 10.1177/1352458516684022. Epub 2016 Dec 22. Mult Scler. 2017. PMID: 28273770
It has been proposed as an autoimmune target in cerebral palsy, gluten-sensitive cerebellar ataxia, and schizophrenia. OBJECTIVE: To investigate TGM6 involvement in multiple sclerosis (MS). ...
It has been proposed as an autoimmune target in cerebral palsy, gluten-sensitive cerebellar ataxia, and schizophrenia. OBJECTIVE: To …
Transglutaminase 6 interacts with polyQ proteins and promotes the formation of polyQ aggregates.
Guan WJ, Xia KD, Ma YT, Liu YT, Shi YT, Jiang H, Shen L, Xia K, Li JD, Tang BS, Wang JL. Guan WJ, et al. Biochem Biophys Res Commun. 2013 Jul 19;437(1):94-100. doi: 10.1016/j.bbrc.2013.06.044. Epub 2013 Jun 22. Biochem Biophys Res Commun. 2013. PMID: 23800413
We previously identified the transglutaminase 6 gene to be causative of spinocerebellar ataxia type 35 (SCA35), and we found that SCA35-associated mutants exhibited reduced transglutaminase activity. ...
We previously identified the transglutaminase 6 gene to be causative of spinocerebellar ataxia type 35 (SCA35), and we found that SCA …
28 results