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Biomolecular Condensates in Myeloid Leukemia: What Do They Tell Us?
Jevtic Z, Allram M, Grebien F, Schwaller J. Jevtic Z, et al. Hemasphere. 2023 Jun 27;7(7):e923. doi: 10.1097/HS9.0000000000000923. eCollection 2023 Jul. Hemasphere. 2023. PMID: 37388925 Free PMC article. Review.
As these compartments, generally referred to as onco-condensates, are specific to tumor cells and are tightly linked to disease development, the mechanisms of their formation and maintenance have been intensively studied. Here we review the proposed leukemogenic and tumor- …
As these compartments, generally referred to as onco-condensates, are specific to tumor cells and are tightly linked to disease development, …
Acute leukemia in early childhood.
Emerenciano M, Koifman S, Pombo-de-Oliveira MS. Emerenciano M, et al. Braz J Med Biol Res. 2007 Jun;40(6):749-60. doi: 10.1590/s0100-879x2007000600002. Braz J Med Biol Res. 2007. PMID: 17581672 Free article. Review.
A molecular study on a Brazilian cohort (age range 0-23 months) has detected TEL/AML1+ve (N = 9), E2A/PBX1+ve (N = 4), PML/RARA+ve (N = 4), and AML1/ETO+ve (N = 2) cases. Undoubtedly, the great majority of genetic events occurring in these patients arise prenatally. ...The …
A molecular study on a Brazilian cohort (age range 0-23 months) has detected TEL/AML1+ve (N = 9), E2A/PBX1+ve (N = 4), PML/RARA+ve (N …
Acute myeloid leukemia in children and adolescents: identification of new molecular targets brings promise of new therapies.
Kolb EA, Meshinchi S. Kolb EA, et al. Hematology Am Soc Hematol Educ Program. 2015;2015:507-13. doi: 10.1182/asheducation-2015.1.507. Hematology Am Soc Hematol Educ Program. 2015. PMID: 26637766 Review.
In all other patients, Ras pathway mutations, KMT2A and other methyltransferase mutations, FLT3 mutations, and KIT mutations are all relatively common in childhood AML and all are potentially "druggable". The focus of this review is on those therapies likely to be c …
In all other patients, Ras pathway mutations, KMT2A and other methyltransferase mutations, FLT3 mutations, and KIT mutations are all …
Molecular profiling of adult acute myeloid and lymphoid leukemia in a major referral center in Lebanon: a 10-year experience report and review of the literature.
Assaf N, El-Cheikh J, Bazarbachi A, Salem Z, Farra C, Chakhachiro Z, Nassif S, Zaatari G, Mahfouz R. Assaf N, et al. Mol Biol Rep. 2019 Apr;46(2):2003-2011. doi: 10.1007/s11033-019-04649-2. Epub 2019 Jan 30. Mol Biol Rep. 2019. PMID: 30701458
Genetic profiling of AML cases in our CAP accredited molecular diagnostics laboratory consists of a validated lab developed RT-PCR for the detection of RUNX1/RUNX1T1, CBFB/MYH11, KMT2A/MLLT3, PML-RARA, and BCR-ABL and mutations in the FLT3 receptor, NPM1, c-kit and …
Genetic profiling of AML cases in our CAP accredited molecular diagnostics laboratory consists of a validated lab developed RT-PCR for the d …
A Highly Complex Hyperdiploid Karyotype in a Patient with MDS: A Case Report and Review of the Literature.
Tirado CA, Hurtado R, King J, Eastwood K, Guardiola MT, Rao A. Tirado CA, et al. J Assoc Genet Technol. 2023;49(2):79-87. J Assoc Genet Technol. 2023. PMID: 37269317
The cytogenetic analysis correlates with the concurrent FISH study which was positive for additional signals of EVI1(3q26.2), TAS2R1 (5p15.31), EGR1 (5q31.2), RELN (7q22), TES (7q31) RUNX1T1 (8q21.3), ABL1 (9q34), KMT2A (11q23), PML (15q24.1), CBFB (16q22), RARA (17 …
The cytogenetic analysis correlates with the concurrent FISH study which was positive for additional signals of EVI1(3q26.2), TAS2R1 (5p15.3 …
[Molecular mechanisms in the development of acute myeloid leukemia].
Ichikawa M, Kurokawa M. Ichikawa M, et al. Nihon Rinsho. 2009 Oct;67(10):1889-93. Nihon Rinsho. 2009. PMID: 19860185 Review. Japanese.
Molecular pathogenesis of AML has been disclosed through analyses of such gene aberrations, including AML1 and MLL abnormalities, PML-RARA chimeric gene, activating mutations of FLT3, and EVI-1 abnormalities. Through prediction of prognosis and targeted therapy, this knowl …
Molecular pathogenesis of AML has been disclosed through analyses of such gene aberrations, including AML1 and MLL abnormalities, PML-RAR …