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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1997 2
1998 2
1999 2
2001 1
2003 1
2006 2
2007 3
2008 2
2009 1
2010 2
2011 6
2013 2
2014 3
2015 1
2016 1
2017 1
2018 2
2019 3
2020 3
2023 3
2024 0

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37 results

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Page 1
ILT4 reprograms glucose metabolism to promote tumor progression in triple-negative breast cancer.
Zhang H, Gao A, Liu Q, Zhang F, Wang S, Chen X, Shi W, Zhang Y, Liu Q, Zheng Y, Sun Y. Zhang H, et al. J Cell Sci. 2023 Sep 15;136(18):jcs260964. doi: 10.1242/jcs.260964. Epub 2023 Sep 18. J Cell Sci. 2023. PMID: 37622462
Further mechanistic analysis revealed that ILT4 reprogrammed aerobic glycolysis of tumor cells via AKT-mTOR signaling-mediated glucose transporter 3 (GLUT3; also known as SLC2A3) and pyruvate kinase muscle 2 (PKM2, an isoform encoded by PKM) overexpression. ILT4 inh …
Further mechanistic analysis revealed that ILT4 reprogrammed aerobic glycolysis of tumor cells via AKT-mTOR signaling-mediated glucose trans …
Combined deletion of Glut1 and Glut3 impairs lung adenocarcinoma growth.
Contat C, Ancey PB, Zangger N, Sabatino S, Pascual J, Escrig S, Jensen L, Goepfert C, Lanz B, Lepore M, Gruetter R, Rossier A, Berezowska S, Neppl C, Zlobec I, Clerc-Rosset S, Knott GW, Rathmell JC, Abel ED, Meibom A, Meylan E. Contat C, et al. Elife. 2020 Jun 23;9:e53618. doi: 10.7554/eLife.53618. Elife. 2020. PMID: 32571479 Free PMC article.
In a genetically-engineered mouse model of lung adenocarcinoma, we show that the deletion of only one highly expressed glucose transporter, Glut1 or Glut3, in cancer cells does not impair tumor growth, whereas their combined loss diminishes tumor development. (18)F-FDG-PET …
In a genetically-engineered mouse model of lung adenocarcinoma, we show that the deletion of only one highly expressed glucose transporter, …
Correlations between class I glucose transporter expression patterns and clinical outcomes in non-small cell lung cancer.
Nakanishi Y, Iwai M, Hirotani Y, Kato R, Tanino T, Nishimaki-Watanabe H, Nozaki F, Ohni S, Tang X, Masuda S, Sasaki-Fukatsu K. Nakanishi Y, et al. Thorac Cancer. 2023 Sep;14(27):2761-2769. doi: 10.1111/1759-7714.15060. Epub 2023 Aug 7. Thorac Cancer. 2023. PMID: 37549925 Free PMC article.
SLC2A4 expression correlated with lower IL8 expression. GLUT3 and GLUT4 expressions were localized in the tumor cytoplasm. CONCLUSIONS: In lung ADC, single SLC2A3 expression correlated with poor prognosis, whereas single SLC2A4 expression correlated with better prog …
SLC2A4 expression correlated with lower IL8 expression. GLUT3 and GLUT4 expressions were localized in the tumor cytoplasm. CONCLUSION …
Glucose transporter expression in developing fetal lungs and lung neoplasms.
Ito T, Noguchi Y, Udaka N, Kitamura H, Satoh S. Ito T, et al. Histol Histopathol. 1999 Jul;14(3):895-904. doi: 10.14670/HH-14.895. Histol Histopathol. 1999. PMID: 10425560 Review.
A few cases of lung carcinoma show positive staining for GLUT3 and GLUT4. Thus, expression of some facilitative glucose transporter isoforms is detected in developing fetal epithelium and in lung carcinomas. ...
A few cases of lung carcinoma show positive staining for GLUT3 and GLUT4. Thus, expression of some facilitative glucose transporter i …
Identification of Proteomic Features To Distinguish Benign Pulmonary Nodules from Lung Adenocarcinoma.
Codreanu SG, Hoeksema MD, Slebos RJC, Zimmerman LJ, Rahman SMJ, Li M, Chen SC, Chen H, Eisenberg R, Liebler DC, Massion PP. Codreanu SG, et al. J Proteome Res. 2017 Sep 1;16(9):3266-3276. doi: 10.1021/acs.jproteome.7b00245. Epub 2017 Aug 8. J Proteome Res. 2017. PMID: 28731711 Free PMC article.
PRM analyses confirmed significant nodule-specific abundance of 10 proteins including ALOX5, ALOX5AP, CCL19, CILP1, COL5A2, ITGB2, ITGAX, PTPRE, S100A12, and SLC2A3 and significant ADC-specific abundance of CEACAM6, CRABP2, LAD1, PLOD2, and TMEM110-MUSTN1. Immunohistochemi …
PRM analyses confirmed significant nodule-specific abundance of 10 proteins including ALOX5, ALOX5AP, CCL19, CILP1, COL5A2, ITGB2, ITGAX, PT …
Metabolic heterogeneity in early-stage lung adenocarcinoma revealed by RNA-seq and scRNA-seq.
Zhang Y, Shi J, Luo J, Liu C, Zhu L. Zhang Y, et al. Clin Transl Oncol. 2023 Jun;25(6):1844-1855. doi: 10.1007/s12094-023-03082-z. Epub 2023 Jan 24. Clin Transl Oncol. 2023. PMID: 36692643
The analysis of single-nucleotide variation (SNV) data showed that the top 15 genes in the four metabolic pathways with the most mutations were TKTL2, PGK2, HK3, EHHADH, GLUD2, PKLR, TKTL1, HADHB, CPT1C, HK1, HK2, PFKL, SLC2A3, PFKFB1, and CPT1A. The IFNgamma score of clus …
The analysis of single-nucleotide variation (SNV) data showed that the top 15 genes in the four metabolic pathways with the most mutations w …
Glucose transporter 3 gene variant is associated with survival outcome of patients with non-small cell lung cancer after surgical resection.
Do SK, Choi SH, Lee SY, Choi JE, Hong MJ, Kang HG, Lee WK, Lee EB, Shin KM, Jeong JY, Lee YH, Seo H, Yoo SS, Lee J, Cha SI, Kim CH, Seok Y, Cho S, Jheon S, Park JY. Do SK, et al. Gene. 2019 Jun 30;703:58-64. doi: 10.1016/j.gene.2019.04.013. Epub 2019 Apr 4. Gene. 2019. PMID: 30954677
Four single nucleotide polymorphisms (SNPs) in GLUT3 were investigated in a total of 782 patients with NSCLC who underwent curative surgery. ...This study suggests that genetic variation in GLUT3 may be useful in predicting survival of patients with early stage NSCL …
Four single nucleotide polymorphisms (SNPs) in GLUT3 were investigated in a total of 782 patients with NSCLC who underwent curative s …
Glucose-transporter-type-I-gene amplification correlates with sialyl-Lewis-X synthesis and proliferation in lung cancer.
Ogawa J, Inoue H, Koide S. Ogawa J, et al. Int J Cancer. 1997 Apr 22;74(2):189-92. doi: 10.1002/(sici)1097-0215(19970422)74:2<189::aid-ijc9>3.0.co;2-v. Int J Cancer. 1997. PMID: 9133454
To examine the role of elevated glucose uptake in lung cancer, we performed PCR amplification of 2 facilitative glucose transporter genes (GLUT1 and GLUT3) and immunohistochemical staining for GLUT1, proliferating cell nuclear antigen (PCNA), and sialyl Lewis x (sLe(x)) on …
To examine the role of elevated glucose uptake in lung cancer, we performed PCR amplification of 2 facilitative glucose transporter genes (G …
IL-8-induced O-GlcNAc modification via GLUT3 and GFAT regulates cancer stem cell-like properties in colon and lung cancer cells.
Shimizu M, Tanaka N. Shimizu M, et al. Oncogene. 2019 Feb;38(9):1520-1533. doi: 10.1038/s41388-018-0533-4. Epub 2018 Oct 10. Oncogene. 2019. PMID: 30305725
We also showed that IL-8 stimulation of colon and lung cancer cells-induced glucose uptake and expressions of glucose transporter 3 (GLUT3) and glucosamine fructose-6-phosphate aminotransferase (GFAT), a regulator of glucose flux to the hexosamine biosynthetic pathway, res …
We also showed that IL-8 stimulation of colon and lung cancer cells-induced glucose uptake and expressions of glucose transporter 3 (GLUT
CAV1 - GLUT3 signaling is important for cellular energy and can be targeted by Atorvastatin in Non-Small Cell Lung Cancer.
Ali A, Levantini E, Fhu CW, Teo JT, Clohessy JG, Goggi JL, Wu CS, Chen L, Chin TM, Tenen DG. Ali A, et al. Theranostics. 2019 Aug 14;9(21):6157-6174. doi: 10.7150/thno.35805. eCollection 2019. Theranostics. 2019. PMID: 31534543 Free PMC article.
Subsequent interrogations demonstrate an oncogenic physical interaction between Cav1 and GLUT3, and glucose uptake found distinctly in TKI-resistant NSCLC and this may be due to changes in the physical properties of Cav1 favoring GLUT3 binding in which significantly …
Subsequent interrogations demonstrate an oncogenic physical interaction between Cav1 and GLUT3, and glucose uptake found distinctly i …
37 results