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2000 1
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2010 4
2011 6
2012 1
2013 2
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2016 5
2017 3
2018 2
2020 1
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Borrelia-primed and -infected mice deficient of interleukin-17 develop arthritis after neutralization of gamma-interferon.
Kuo J, Warner TF, Schell RF. Kuo J, et al. Pathog Dis. 2017 Mar 1;75(2). doi: 10.1093/femspd/ftx014. Pathog Dis. 2017. PMID: 28175297
The immune mechanisms responsible for development of Lyme arthritis are partially understood with interleukin-17 (IL-17) and gamma-interferon (IFN-γ) playing a generally accepted role. ...We also found higher levels of tumor necrosis factor alpha (TNF- …
The immune mechanisms responsible for development of Lyme arthritis are partially understood with interleukin-17 (IL- …
CD4+ cell-derived interleukin-17 in a model of dysregulated, Borrelia-induced arthritis.
Hansen ES, Johnson ME, Schell RF, Nardelli DT. Hansen ES, et al. Pathog Dis. 2016 Oct;74(7):ftw084. doi: 10.1093/femspd/ftw084. Epub 2016 Aug 21. Pathog Dis. 2016. PMID: 27549424
Here, we hypothesized that a murine model combining the dysregulated immune response of an environment lacking interleukin-10 (IL-10) with a robust T-cell-driven inflammatory response would reflect arthritis associated with the production of IL-17 by C …
Here, we hypothesized that a murine model combining the dysregulated immune response of an environment lacking interleukin-10 (IL
Arthritis is developed in Borrelia-primed and -infected mice deficient of interleukin-17.
Kuo J, Warner TF, Munson EL, Nardelli DT, Schell RF. Kuo J, et al. Pathog Dis. 2016 Oct;74(7):ftw077. doi: 10.1093/femspd/ftw077. Epub 2016 Aug 4. Pathog Dis. 2016. PMID: 27493062
Interleukin-17 (IL-17) has been shown to participate in the development of Lyme arthritis in experimental mice. For example, neutralization of IL-17 with antibodies inhibits induction of arthritis in Borrelia-primed and -infected C
Interleukin-17 (IL-17) has been shown to participate in the development of Lyme arthritis in experimental mice.
Interleukin-10 (IL-10) inhibits Borrelia burgdorferi-induced IL-17 production and attenuates IL-17-mediated Lyme arthritis.
Hansen ES, Medić V, Kuo J, Warner TF, Schell RF, Nardelli DT. Hansen ES, et al. Infect Immun. 2013 Dec;81(12):4421-30. doi: 10.1128/IAI.01129-13. Epub 2013 Sep 16. Infect Immun. 2013. PMID: 24042116 Free PMC article.
Previous studies have shown that cells and cytokines associated with interleukin-17 (IL-17)-driven inflammation are involved in the arthritic response to Borrelia burgdorferi infection. Here, we report that IL-17 is a contributing …
Previous studies have shown that cells and cytokines associated with interleukin-17 (IL-17)-driven inflammation …
Interleukin-35 enhances Lyme arthritis in Borrelia-vaccinated and -infected mice.
Kuo J, Nardelli DT, Warner TF, Callister SM, Schell RF. Kuo J, et al. Clin Vaccine Immunol. 2011 Jul;18(7):1125-32. doi: 10.1128/CVI.00052-11. Epub 2011 May 25. Clin Vaccine Immunol. 2011. PMID: 21613457 Free PMC article.
Interleukin-35 (IL-35) has been reported to inhibit the production of interleukin-17 (IL-17) as a means of preventing arthritis and other inflammatory diseases. ...We hypothesized that IL-35 would reduce the severity of arthritis b
Interleukin-35 (IL-35) has been reported to inhibit the production of interleukin-17 (IL-17) as a
Role of interleukin-23 (IL-23) receptor signaling for IL-17 responses in human Lyme disease.
Oosting M, ter Hofstede H, van de Veerdonk FL, Sturm P, Kullberg BJ, van der Meer JW, Netea MG, Joosten LA. Oosting M, et al. Infect Immun. 2011 Nov;79(11):4681-7. doi: 10.1128/IAI.05242-11. Epub 2011 Sep 6. Infect Immun. 2011. PMID: 21896776 Free PMC article.
Interleukin-23 (IL-23) is known to play a crucial role in the development and maintenance of T helper 17 cells. It has been previously demonstrated that IL-17 is involved in experimental Lyme arthritis, caused by Borrelia burgdorferi bact
Interleukin-23 (IL-23) is known to play a crucial role in the development and maintenance of T helper 17 cells. It has
Borrelia species induce inflammasome activation and IL-17 production through a caspase-1-dependent mechanism.
Oosting M, van de Veerdonk FL, Kanneganti TD, Sturm P, Verschueren I, Berende A, van der Meer JW, Kullberg BJ, Netea MG, Joosten LA. Oosting M, et al. Eur J Immunol. 2011 Jan;41(1):172-81. doi: 10.1002/eji.201040385. Epub 2010 Dec 9. Eur J Immunol. 2011. PMID: 21182088 Free PMC article.
Caspase-1-deficient cells showed significantly less IFN-γ and IL-17 production after Borrelia stimulation. A lack of IL-1β was responsible for the defective IL-17 production, whereas IL-18 was crucial for the IFN-γ production. Casp …
Caspase-1-deficient cells showed significantly less IFN-γ and IL-17 production after Borrelia stimulation. A lack of …
Inhibition of interleukin-17 prevents the development of arthritis in vaccinated mice challenged with Borrelia burgdorferi.
Burchill MA, Nardelli DT, England DM, DeCoster DJ, Christopherson JA, Callister SM, Schell RF. Burchill MA, et al. Infect Immun. 2003 Jun;71(6):3437-42. doi: 10.1128/iai.71.6.3437-3442.2003. Infect Immun. 2003. PMID: 12761128 Free PMC article.
The immune response underlying the development of the severe destructive arthritis involves interleukin-17 (IL-17). Treatment of vaccinated IFN-gamma(0) mice challenged with B. burgdorferi with anti-IL-17 antibody delayed the onset of swe …
The immune response underlying the development of the severe destructive arthritis involves interleukin-17 (IL-17
Anti-interleukin-15 prevents arthritis in Borrelia-vaccinated and -infected mice.
Amlong CA, Nardelli DT, Peterson SH, Warner TF, Callister SM, Schell RF. Amlong CA, et al. Clin Vaccine Immunol. 2006 Feb;13(2):289-96. doi: 10.1128/CVI.13.2.289-296.2006. Clin Vaccine Immunol. 2006. PMID: 16467340 Free PMC article.
We showed previously that interleukin-17 (IL-17) plays a significant role in the induction of arthritis associated with Borrelia vaccination and challenge. ...These results suggest that IL-15 plays a major role in orchestrating IL- …
We showed previously that interleukin-17 (IL-17) plays a significant role in the induction of arthritis associat …
Early production of IL-22 but not IL-17 by peripheral blood mononuclear cells exposed to live Borrelia burgdorferi: the role of monocytes and interleukin-1.
Bachmann M, Horn K, Rudloff I, Goren I, Holdener M, Christen U, Darsow N, Hunfeld KP, Koehl U, Kind P, Pfeilschifter J, Kraiczy P, Mühl H. Bachmann M, et al. PLoS Pathog. 2010 Oct 14;6(10):e1001144. doi: 10.1371/journal.ppat.1001144. PLoS Pathog. 2010. PMID: 20976193 Free PMC article.
Since Th17 cytokines are connected to host defense against extracellular bacteria, we focused on interleukin (IL)-17 and IL-22. Here, we report that, despite induction of inflammatory cytokines including IL-23, IL-17 remained barel …
Since Th17 cytokines are connected to host defense against extracellular bacteria, we focused on interleukin (IL)-17 an …
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