Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

Filters

My Custom Filters

Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1990 1
1999 1
2000 1
2001 2
2002 1
2003 2
2004 1
2005 2
2006 3
2007 1
2008 2
2010 2
2025 0

Publication date

Text availability

Article attribute

Article type

Additional filters

Article Language

Species

Sex

Age

Other

Search Results

18 results

Results by year

Filters applied: . Clear all
Page 1
Calmodulin kinase II activity is required for normal atrioventricular nodal conduction.
Khoo MS, Kannankeril PJ, Li J, Zhang R, Kupershmidt S, Zhang W, Atkinson JB, Colbran RJ, Roden DM, Anderson ME. Khoo MS, et al. Among authors: colbran rj. Heart Rhythm. 2005 Jun;2(6):634-40. doi: 10.1016/j.hrthm.2005.03.019. Heart Rhythm. 2005. PMID: 15922273
OBJECTIVES: The purpose of this study was to test the potential role of CaMKII in regulating AV nodal conduction in heart. METHODS: We developed a novel mouse with genetic CaMKII inhibition by cardiac-specific expression of autocamtide 3 inhibitory peptide (AC3-I) mimickin …
OBJECTIVES: The purpose of this study was to test the potential role of CaMKII in regulating AV nodal conduction in heart. METHODS: W …
Tissue-specific variation of Ube3a protein expression in rodents and in a mouse model of Angelman syndrome.
Gustin RM, Bichell TJ, Bubser M, Daily J, Filonova I, Mrelashvili D, Deutch AY, Colbran RJ, Weeber EJ, Haas KF. Gustin RM, et al. Among authors: colbran rj. Neurobiol Dis. 2010 Sep;39(3):283-91. doi: 10.1016/j.nbd.2010.04.012. Epub 2010 Apr 25. Neurobiol Dis. 2010. PMID: 20423730 Free PMC article.
Immunoblot and immunohistochemical analyses revealed a marked loss of Ube3a protein in hippocampus, hypothalamus, olfactory bulb, cerebral cortex, striatum, thalamus, midbrain, and cerebellum in AS mice relative to WT littermates. Also, Ube3a expression in heart and liver …
Immunoblot and immunohistochemical analyses revealed a marked loss of Ube3a protein in hippocampus, hypothalamus, olfactory bulb, cerebral c …
Calmodulin kinase II inhibition protects against structural heart disease.
Zhang R, Khoo MS, Wu Y, Yang Y, Grueter CE, Ni G, Price EE Jr, Thiel W, Guatimosim S, Song LS, Madu EC, Shah AN, Vishnivetskaya TA, Atkinson JB, Gurevich VV, Salama G, Lederer WJ, Colbran RJ, Anderson ME. Zhang R, et al. Among authors: colbran rj. Nat Med. 2005 Apr;11(4):409-17. doi: 10.1038/nm1215. Epub 2005 Mar 27. Nat Med. 2005. PMID: 15793582
These findings mark CaMKII as a determinant of clinically important heart disease phenotypes, and suggest CaMKII inhibition can be a highly selective approach for targeting adverse myocardial remodeling linked to betaAR signaling....
These findings mark CaMKII as a determinant of clinically important heart disease phenotypes, and suggest CaMKII inhibition can be a …
A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation.
Erickson JR, Joiner ML, Guan X, Kutschke W, Yang J, Oddis CV, Bartlett RK, Lowe JS, O'Donnell SE, Aykin-Burns N, Zimmerman MC, Zimmerman K, Ham AJ, Weiss RM, Spitz DR, Shea MA, Colbran RJ, Mohler PJ, Anderson ME. Erickson JR, et al. Among authors: colbran rj. Cell. 2008 May 2;133(3):462-74. doi: 10.1016/j.cell.2008.02.048. Cell. 2008. PMID: 18455987 Free PMC article.
CaV1.2 beta-subunit coordinates CaMKII-triggered cardiomyocyte death and afterdepolarizations.
Koval OM, Guan X, Wu Y, Joiner ML, Gao Z, Chen B, Grumbach IM, Luczak ED, Colbran RJ, Song LS, Hund TJ, Mohler PJ, Anderson ME. Koval OM, et al. Among authors: colbran rj. Proc Natl Acad Sci U S A. 2010 Mar 16;107(11):4996-5000. doi: 10.1073/pnas.0913760107. Epub 2010 Mar 1. Proc Natl Acad Sci U S A. 2010. PMID: 20194790 Free PMC article.
Excessive activation of calmodulin kinase II (CaMKII) causes arrhythmias and heart failure, but the cellular mechanisms for CaMKII-targeted proteins causing disordered cell membrane excitability and myocardial dysfunction remain uncertain. ...
Excessive activation of calmodulin kinase II (CaMKII) causes arrhythmias and heart failure, but the cellular mechanisms for CaMKII-ta …
Death, cardiac dysfunction, and arrhythmias are increased by calmodulin kinase II in calcineurin cardiomyopathy.
Khoo MS, Li J, Singh MV, Yang Y, Kannankeril P, Wu Y, Grueter CE, Guan X, Oddis CV, Zhang R, Mendes L, Ni G, Madu EC, Yang J, Bass M, Gomez RJ, Wadzinski BE, Olson EN, Colbran RJ, Anderson ME. Khoo MS, et al. Among authors: colbran rj. Circulation. 2006 Sep 26;114(13):1352-9. doi: 10.1161/CIRCULATIONAHA.106.644583. Epub 2006 Sep 18. Circulation. 2006. PMID: 16982937
METHODS AND RESULTS: We found that activity and expression of a second Ca2+-activated signaling molecule, calmodulin kinase II (CaMKII), were increased in hearts from CAN transgenic mice and that CaMKII-inhibitory drugs improved LV function and suppressed arrhythmias. ...
METHODS AND RESULTS: We found that activity and expression of a second Ca2+-activated signaling molecule, calmodulin kinase II (CaMKII), wer …
18 results