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1996 1
1997 2
1998 1
2001 1
2021 0
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Page 1
Molecular mechanisms of thiamine utilization.
Singleton CK, Martin PR. Singleton CK, et al. Curr Mol Med. 2001 May;1(2):197-207. doi: 10.2174/1566524013363870. Curr Mol Med. 2001. PMID: 11899071 Review.
A transketolase assembly defect in a Wernicke-Korsakoff syndrome patient.
Wang JJ, Martin PR, Singleton CK. Wang JJ, et al. Alcohol Clin Exp Res. 1997 Jun;21(4):576-80. Alcohol Clin Exp Res. 1997. PMID: 9194907
Conserved residues are functionally distinct within transketolases of different species.
Singleton CK, Wang JJ, Shan L, Martin PR. Singleton CK, et al. Biochemistry. 1996 Dec 10;35(49):15865-9. doi: 10.1021/bi9616920. Biochemistry. 1996. PMID: 8961951
Thiamine deficiency decreases steady-state transketolase and pyruvate dehydrogenase but not alpha-ketoglutarate dehydrogenase mRNA levels in three human cell types.
Pekovich SR, Martin PR, Singleton CK. Pekovich SR, et al. J Nutr. 1998 Apr;128(4):683-7. doi: 10.1093/jn/128.4.683. J Nutr. 1998. PMID: 9521628
Aspartate 155 of human transketolase is essential for thiamine diphosphate-magnesium binding, and cofactor binding is required for dimer formation.
Wang JJ, Martin PR, Singleton CK. Wang JJ, et al. Biochim Biophys Acta. 1997 Sep 5;1341(2):165-72. doi: 10.1016/s0167-4838(97)00067-8. Biochim Biophys Acta. 1997. PMID: 9357955
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