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1998 1
2000 3
2001 5
2002 2
2004 1
2021 0
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Page 1
Genes and cellular requirements for autoimmune diabetes susceptibility in nonobese diabetic mice.
Serreze DV, Leiter EH. Serreze DV, et al. Curr Dir Autoimmun. 2001;4:31-67. doi: 10.1159/000060527. Curr Dir Autoimmun. 2001. PMID: 11569409 Review. No abstract available.
B cell selection defects underlie the development of diabetogenic APCs in nonobese diabetic mice.
Silveira PA, Dombrowsky J, Johnson E, Chapman HD, Nemazee D, Serreze DV. Silveira PA, et al. J Immunol. 2004 Apr 15;172(8):5086-94. doi: 10.4049/jimmunol.172.8.5086. J Immunol. 2004. PMID: 15067092 Free PMC article.
New mouse model to study islet transplantation in insulin-dependent diabetes mellitus.
Mathews CE, Langley SH, Leiter EH. Mathews CE, et al. Transplantation. 2002 Apr 27;73(8):1333-6. doi: 10.1097/00007890-200204270-00024. Transplantation. 2002. PMID: 11981430
Unusual resistance of ALR/Lt mouse beta cells to autoimmune destruction: role for beta cell-expressed resistance determinants.
Mathews CE, Graser RT, Savinov A, Serreze DV, Leiter EH. Mathews CE, et al. Proc Natl Acad Sci U S A. 2001 Jan 2;98(1):235-40. doi: 10.1073/pnas.98.1.235. Proc Natl Acad Sci U S A. 2001. PMID: 11136257 Free PMC article.
Acceleration of type 1 diabetes by a coxsackievirus infection requires a preexisting critical mass of autoreactive T-cells in pancreatic islets.
Serreze DV, Ottendorfer EW, Ellis TM, Gauntt CJ, Atkinson MA. Serreze DV, et al. Diabetes. 2000 May;49(5):708-11. doi: 10.2337/diabetes.49.5.708. Diabetes. 2000. PMID: 10905477
The preferential ability of B lymphocytes to act as diabetogenic APC in NOD mice depends on expression of self-antigen-specific immunoglobulin receptors.
Silveira PA, Johnson E, Chapman HD, Bui T, Tisch RM, Serreze DV. Silveira PA, et al. Eur J Immunol. 2002 Dec;32(12):3657-66. doi: 10.1002/1521-4141(200212)32:12<3657::AID-IMMU3657>3.0.CO;2-E. Eur J Immunol. 2002. PMID: 12516557 Free article.
Th1 to Th2 cytokine shifts in nonobese diabetic mice: sometimes an outcome, rather than the cause, of diabetes resistance elicited by immunostimulation.
Serreze DV, Chapman HD, Post CM, Johnson EA, Suarez-Pinzon WL, Rabinovitch A. Serreze DV, et al. J Immunol. 2001 Jan 15;166(2):1352-9. doi: 10.4049/jimmunol.166.2.1352. J Immunol. 2001. PMID: 11145720 Free article.
Inhibition of autoimmune diabetes in nonobese diabetic mice by transgenic restoration of H2-E MHC class II expression: additive, but unequal, involvement of multiple APC subtypes.
Johnson EA, Silveira P, Chapman HD, Leiter EH, Serreze DV. Johnson EA, et al. J Immunol. 2001 Aug 15;167(4):2404-10. doi: 10.4049/jimmunol.167.4.2404. J Immunol. 2001. PMID: 11490031 Free article.
Identification of a CD8 T cell that can independently mediate autoimmune diabetes development in the complete absence of CD4 T cell helper functions.
Graser RT, DiLorenzo TP, Wang F, Christianson GJ, Chapman HD, Roopenian DC, Nathenson SG, Serreze DV. Graser RT, et al. J Immunol. 2000 Apr 1;164(7):3913-8. doi: 10.4049/jimmunol.164.7.3913. J Immunol. 2000. PMID: 10725754 Free article.
Subcongenic analysis of the Idd13 locus in NOD/Lt mice: evidence for several susceptibility genes including a possible diabetogenic role for beta 2-microglobulin.
Serreze DV, Bridgett M, Chapman HD, Chen E, Richard SD, Leiter EH. Serreze DV, et al. J Immunol. 1998 Feb 1;160(3):1472-8. J Immunol. 1998. PMID: 9570569 Free article.
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