Abell AN - Search Results

1

Constitutive activation of G protein-coupled receptors as a result of selective substitution of a conserved leucine residue in transmembrane helix III.

Tao YX, Abell AN, Liu X, Nakamura K, Segaloff DL. Tao YX, et al. Among authors: abell an. Mol Endocrinol. 2000 Aug;14(8):1272-82. doi: 10.1210/mend.14.8.0503. Mol Endocrinol. 2000. PMID: 10935550

2

Evidence for the direct involvement of transmembrane region 6 of the lutropin/choriogonadotropin receptor in activating Gs.

Abell AN, Segaloff DL. Abell AN, et al. J Biol Chem. 1997 Jun 6;272(23):14586-91. doi: 10.1074/jbc.272.23.14586. J Biol Chem. 1997. PMID: 9169417 Free article.

3

A unique constitutively activating mutation in third transmembrane helix of luteinizing hormone receptor causes sporadic male gonadotropin-independent precocious puberty.

Latronico AC, Abell AN, Arnhold IJ, Liu X, Lins TS, Brito VN, Billerbeck AE, Segaloff DL, Mendonca BB. Latronico AC, et al. Among authors: abell an. J Clin Endocrinol Metab. 1998 Jul;83(7):2435-40. doi: 10.1210/jcem.83.7.4968. J Clin Endocrinol Metab. 1998. PMID: 9661624

4

Certain activating mutations within helix 6 of the human luteinizing hormone receptor may be explained by alterations that allow transmembrane regions to activate Gs.

Abell AN, McCormick DJ, Segaloff DL. Abell AN, et al. Mol Endocrinol. 1998 Dec;12(12):1857-69. doi: 10.1210/mend.12.12.0202. Mol Endocrinol. 1998. PMID: 9849960

5

Deletions of portions of the extracellular loops of the lutropin/choriogonadotropin receptor decrease the binding affinity for ovine luteinizing hormone, but not human choriogonadotropin, by preventing the formation of mature cell surface receptor.

Abell A, Liu X, Segaloff DL. Abell A, et al. J Biol Chem. 1996 Feb 23;271(8):4518-27. doi: 10.1074/jbc.271.8.4518. J Biol Chem. 1996. PMID: 8626807 Free article.

6

Mutations that induce constitutive activation and mutations that impair signal transduction modulate the basal and/or agonist-stimulated internalization of the Lutropin/Choriogonadotropin receptor.

Min KS, Liu X, Fabritz J, Jaquette J, Abell AN, Ascoli M. Min KS, et al. Among authors: abell an. J Biol Chem. 1998 Dec 25;273(52):34911-9. doi: 10.1074/jbc.273.52.34911. J Biol Chem. 1998. PMID: 9857020 Free article.

7

Wiring diagrams of MAPK regulation by MEKK1, 2, and 3.

Uhlik MT, Abell AN, Cuevas BD, Nakamura K, Johnson GL. Uhlik MT, et al. Among authors: abell an. Biochem Cell Biol. 2004 Dec;82(6):658-63. doi: 10.1139/o04-114. Biochem Cell Biol. 2004. PMID: 15674433 Review.

8

Ablation of MEKK4 kinase activity causes neurulation and skeletal patterning defects in the mouse embryo.

Abell AN, Rivera-Perez JA, Cuevas BD, Uhlik MT, Sather S, Johnson NL, Minton SK, Lauder JM, Winter-Vann AM, Nakamura K, Magnuson T, Vaillancourt RR, Heasley LE, Johnson GL. Abell AN, et al. Mol Cell Biol. 2005 Oct;25(20):8948-59. doi: 10.1128/MCB.25.20.8948-8959.2005. Mol Cell Biol. 2005. PMID: 16199873 Free PMC article.

9

Defining the expressed breast cancer kinome.

Midland AA, Whittle MC, Duncan JS, Abell AN, Nakamura K, Zawistowski JS, Carey LA, Earp HS 3rd, Graves LM, Gomez SM, Johnson GL. Midland AA, et al. Among authors: abell an. Cell Res. 2012 Apr;22(4):620-3. doi: 10.1038/cr.2012.25. Epub 2012 Feb 7. Cell Res. 2012. PMID: 22310242 Free PMC article. No abstract available.

10

Dynamic reprogramming of the kinome in response to targeted MEK inhibition in triple-negative breast cancer.

Duncan JS, Whittle MC, Nakamura K, Abell AN, Midland AA, Zawistowski JS, Johnson NL, Granger DA, Jordan NV, Darr DB, Usary J, Kuan PF, Smalley DM, Major B, He X, Hoadley KA, Zhou B, Sharpless NE, Perou CM, Kim WY, Gomez SM, Chen X, Jin J, Frye SV, Earp HS, Graves LM, Johnson GL. Duncan JS, et al. Among authors: abell an. Cell. 2012 Apr 13;149(2):307-21. doi: 10.1016/j.cell.2012.02.053. Cell. 2012. PMID: 22500798 Free PMC article.

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