Allday MJ - Search Results

1

An ATM/Chk2-mediated DNA damage-responsive signaling pathway suppresses Epstein-Barr virus transformation of primary human B cells.

Nikitin PA, Yan CM, Forte E, Bocedi A, Tourigny JP, White RE, Allday MJ, Patel A, Dave SS, Kim W, Hu K, Guo J, Tainter D, Rusyn E, Luftig MA. Nikitin PA, et al. Among authors: allday mj. Cell Host Microbe. 2010 Dec 16;8(6):510-22. doi: 10.1016/j.chom.2010.11.004. Cell Host Microbe. 2010. PMID: 21147465 Free PMC article.

2

Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection.

Price AM, Dai J, Bazot Q, Patel L, Nikitin PA, Djavadian R, Winter PS, Salinas CA, Barry AP, Wood KC, Johannsen EC, Letai A, Allday MJ, Luftig MA. Price AM, et al. Among authors: allday mj. Elife. 2017 Apr 20;6:e22509. doi: 10.7554/eLife.22509. Elife. 2017. PMID: 28425914 Free PMC article.

3

Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP.

Skalska L, White RE, Franz M, Ruhmann M, Allday MJ. Skalska L, et al. Among authors: allday mj. PLoS Pathog. 2010 Jun 10;6(6):e1000951. doi: 10.1371/journal.ppat.1000951. PLoS Pathog. 2010. PMID: 20548956 Free PMC article.

4

Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification.

White RE, Groves IJ, Turro E, Yee J, Kremmer E, Allday MJ. White RE, et al. Among authors: allday mj. PLoS One. 2010 Nov 15;5(11):e13979. doi: 10.1371/journal.pone.0013979. PLoS One. 2010. PMID: 21085583 Free PMC article.

5

Latent Epstein-Barr virus can inhibit apoptosis in B cells by blocking the induction of NOXA expression.

Yee J, White RE, Anderton E, Allday MJ. Yee J, et al. Among authors: allday mj. PLoS One. 2011;6(12):e28506. doi: 10.1371/journal.pone.0028506. Epub 2011 Dec 9. PLoS One. 2011. PMID: 22174825 Free PMC article.

6

The EBNA3 Family: Two Oncoproteins and a Tumour Suppressor that Are Central to the Biology of EBV in B Cells.

Allday MJ, Bazot Q, White RE. Allday MJ, et al. Curr Top Microbiol Immunol. 2015;391:61-117. doi: 10.1007/978-3-319-22834-1_3. Curr Top Microbiol Immunol. 2015. PMID: 26428372 Review.

7

EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.

Styles CT, Bazot Q, Parker GA, White RE, Paschos K, Allday MJ. Styles CT, et al. Among authors: allday mj. PLoS Biol. 2017 Aug 3;15(8):e2001992. doi: 10.1371/journal.pbio.2001992. eCollection 2017 Aug. PLoS Biol. 2017. PMID: 28771465 Free PMC article.

8

Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines.

Skalska L, White RE, Parker GA, Turro E, Sinclair AJ, Paschos K, Allday MJ. Skalska L, et al. Among authors: allday mj. PLoS Pathog. 2013 Feb;9(2):e1003187. doi: 10.1371/journal.ppat.1003187. Epub 2013 Feb 21. PLoS Pathog. 2013. PMID: 23436997 Free PMC article.

9

Persistent KSHV Infection Increases EBV-Associated Tumor Formation In Vivo via Enhanced EBV Lytic Gene Expression.

McHugh D, Caduff N, Barros MHM, Rämer PC, Raykova A, Murer A, Landtwing V, Quast I, Styles CT, Spohn M, Fowotade A, Delecluse HJ, Papoudou-Bai A, Lee YM, Kim JM, Middeldorp J, Schulz TF, Cesarman E, Zbinden A, Capaul R, White RE, Allday MJ, Niedobitek G, Blackbourn DJ, Grundhoff A, Münz C. McHugh D, et al. Among authors: allday mj. Cell Host Microbe. 2017 Jul 12;22(1):61-73.e7. doi: 10.1016/j.chom.2017.06.009. Cell Host Microbe. 2017. PMID: 28704654 Free article.

10

Epstein-barr virus latency in B cells leads to epigenetic repression and CpG methylation of the tumour suppressor gene Bim.

Paschos K, Smith P, Anderton E, Middeldorp JM, White RE, Allday MJ. Paschos K, et al. Among authors: allday mj. PLoS Pathog. 2009 Jun;5(6):e1000492. doi: 10.1371/journal.ppat.1000492. Epub 2009 Jun 26. PLoS Pathog. 2009. PMID: 19557159 Free PMC article.

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