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Year Number of Results
1986 1
1991 5
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1993 2
1994 5
1995 7
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1997 12
1998 8
1999 11
2000 8
2001 8
2002 7
2003 10
2004 2
2005 3
2006 10
2007 5
2008 1
2009 6
2010 4
2011 3
2012 2
2013 4
2014 1
2015 3
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2023 5
2024 2

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152 results

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Page 1
Apparent mineralocorticoid excess.
Funder JW. Funder JW. J Steroid Biochem Mol Biol. 2017 Jan;165(Pt A):151-153. doi: 10.1016/j.jsbmb.2016.03.010. Epub 2016 Mar 5. J Steroid Biochem Mol Biol. 2017. PMID: 26956190 Review.
Apparent mineralocorticoid excess is a syndrome reflecting the absent or impaired activity of the enzyme 11beta-hydroxysteroid dehydrogenase Type 2. ...Treatment is symptomatic in the mild form - correction of hypertension and hypokalemia - but needs t
Apparent mineralocorticoid excess is a syndrome reflecting the absent or impaired activity of the enzyme 11beta-
Low-Renin Hypertension.
Athimulam S, Lazik N, Bancos I. Athimulam S, et al. Endocrinol Metab Clin North Am. 2019 Dec;48(4):701-715. doi: 10.1016/j.ecl.2019.08.003. Epub 2019 Sep 25. Endocrinol Metab Clin North Am. 2019. PMID: 31655771 Review.
Primary hyperaldosteronism presents with low renin and aldosterone excess. Low-renin, low-aldosterone hypertension represents a wide spectrum of disorders that includes essential low-renin hypertension, hereditary forms of hypertension, and hypertension secondary to endoge …
Primary hyperaldosteronism presents with low renin and aldosterone excess. Low-renin, low-aldosterone hypertension represents a wide …
Classic and Nonclassic Apparent Mineralocorticoid Excess Syndrome.
Carvajal CA, Tapia-Castillo A, Vecchiola A, Baudrand R, Fardella CE. Carvajal CA, et al. J Clin Endocrinol Metab. 2020 Apr 1;105(4):dgz315. doi: 10.1210/clinem/dgz315. J Clin Endocrinol Metab. 2020. PMID: 31909799 Review.
Subtypes of essential hypertension characterized by low renin levels allowed the identification of 2 different clinical entities: aldosterone-mediated mineralocorticoid receptor (MR) activation and cortisol-mediated MR activation. EVIDENCE ACQUISITION: This review is based …
Subtypes of essential hypertension characterized by low renin levels allowed the identification of 2 different clinical entities: aldosteron …
Apparent mineralocorticoid excess.
Wilson RC, Nimkarn S, New MI. Wilson RC, et al. Trends Endocrinol Metab. 2001 Apr;12(3):104-11. doi: 10.1016/s1043-2760(00)00356-8. Trends Endocrinol Metab. 2001. PMID: 11306334 Review.
Apparent mineralocorticoid excess (AME) is a potentially fatal genetic disorder causing severe juvenile hypertension, pre- and postnatal growth failure, hypokalemia and low to undetectable levels of renin and aldosterone. ...The 11 beta-HSD2 enzyme is respons
Apparent mineralocorticoid excess (AME) is a potentially fatal genetic disorder causing severe juvenile hypertension, p
Apparent mineralocorticoid excess: comprehensive overview of molecular genetics.
Lu YT, Zhang D, Zhang QY, Zhou ZM, Yang KQ, Zhou XL, Peng F. Lu YT, et al. J Transl Med. 2022 Nov 3;20(1):500. doi: 10.1186/s12967-022-03698-9. J Transl Med. 2022. PMID: 36329487 Free PMC article. Review.
Apparent mineralocorticoid excess is an autosomal recessive form of monogenic disease characterized by juvenile resistant low-renin hypertension, marked hypokalemic alkalosis, low aldosterone levels, and high ratios of cortisol to cortisone metabolites. ...In
Apparent mineralocorticoid excess is an autosomal recessive form of monogenic disease characterized by juvenile resista
Apparent mineralocorticoid excess: type I and type II.
Mantero F, Palermo M, Petrelli MD, Tedde R, Stewart PM, Shackleton CH. Mantero F, et al. Steroids. 1996 Apr;61(4):193-6. doi: 10.1016/0039-128x(96)00012-8. Steroids. 1996. PMID: 8732999 Review.
The syndrome of apparent mineralocorticoid excess (AME) is a heritable form of hypertension due to an inborn error of cortisol metabolism and is characterized by hypokalemia and low renin levels despite subnormal or normal levels of aldosterone and oth …
The syndrome of apparent mineralocorticoid excess (AME) is a heritable form of hypertension due to an inborn err …
The Dehydrogenase Hypothesis.
Woods C, Tomlinson JW. Woods C, et al. Adv Exp Med Biol. 2015;872:353-80. doi: 10.1007/978-1-4939-2895-8_16. Adv Exp Med Biol. 2015. PMID: 26216003 Review.
This inactivates cortisol to cortisone and offers protection for the mineralocorticoid receptor form occupation and activation by cortisol. ...Mutations within the gene encoding 11beta (Beta)-HSD2 cause the Syndrome of Apparent Mineralocorticoid Exc
This inactivates cortisol to cortisone and offers protection for the mineralocorticoid receptor form occupation and activation by cor …
Pathophysiologic approach in genetic hypokalemia: An update.
Blanchard A. Blanchard A. Ann Endocrinol (Paris). 2023 Apr;84(2):298-307. doi: 10.1016/j.ando.2022.11.005. Epub 2023 Jan 10. Ann Endocrinol (Paris). 2023. PMID: 36639120 Review.
Genetic hypertensive hypokalemia is rare. It includes familial hyperaldosteronism, Liddle syndrome, apparent mineralocorticoid excess,11beta hydroxylase deficiency and Geller syndrome. ...Due to the severity of hypokalemic metabolic alkalosis, t …
Genetic hypertensive hypokalemia is rare. It includes familial hyperaldosteronism, Liddle syndrome, apparent mineralocortic
Mineralocorticoid and apparent mineralocorticoid syndromes of secondary hypertension.
Ardhanari S, Kannuswamy R, Chaudhary K, Lockette W, Whaley-Connell A. Ardhanari S, et al. Adv Chronic Kidney Dis. 2015 May;22(3):185-95. doi: 10.1053/j.ackd.2015.03.002. Adv Chronic Kidney Dis. 2015. PMID: 25908467 Review.
Inappropriate excess levels of aldosterone contribute significantly to the cardiorenal metabolic syndrome and target organ injury that include atherosclerosis, myocardial hypertrophy, fibrosis, heart failure, and kidney disease. The importance of understanding the r …
Inappropriate excess levels of aldosterone contribute significantly to the cardiorenal metabolic syndrome and target organ inj …
Hyperaldosteronism in pregnancy.
Escher G. Escher G. Ther Adv Cardiovasc Dis. 2009 Apr;3(2):123-32. doi: 10.1177/1753944708100180. Epub 2009 Jan 26. Ther Adv Cardiovasc Dis. 2009. PMID: 19171690 Free article. Review.
Features of hyperaldosteronism are also found in patients with apparent mineralocorticoid excess (AME), in which glucocorticoids exacerbate activation of the mineralocorticoid receptor (MR) because of a defect in the 11beta-hydroxysteroid dehydrogenase …
Features of hyperaldosteronism are also found in patients with apparent mineralocorticoid excess (AME), in which glucoc …
152 results