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1986 1
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Assessment of the genetic variance of late-onset Alzheimer's disease.
Ridge PG, Hoyt KB, Boehme K, Mukherjee S, Crane PK, Haines JL, Mayeux R, Farrer LA, Pericak-Vance MA, Schellenberg GD, Kauwe JSK; Alzheimer's Disease Genetics Consortium (ADGC). Ridge PG, et al. Neurobiol Aging. 2016 May;41:200.e13-200.e20. doi: 10.1016/j.neurobiolaging.2016.02.024. Epub 2016 Mar 3. Neurobiol Aging. 2016. PMID: 27036079 Free PMC article.
Alzheimer's disease (AD) is a complex genetic disorder with no effective treatments. ...Of the unexplained genetic variance, approximately 41% is explained by unknown SNPs in regions adjacent to known AD SNPs, and the remaining unexplaine
Alzheimer's disease (AD) is a complex genetic disorder with no effective treatments. ...Of the unexplained ge
Genetic analyses of early-onset Alzheimer's disease using next generation sequencing.
Giau VV, Bagyinszky E, Yang YS, Youn YC, An SSA, Kim SY. Giau VV, et al. Sci Rep. 2019 Jun 10;9(1):8368. doi: 10.1038/s41598-019-44848-2. Sci Rep. 2019. PMID: 31182772 Free PMC article.
Alzheimer's disease (AD) is the most common type of neurodegenerative dementia, but the cause of AD remained poorly understood. ...Additionally, 67 missense mutations in susceptibility genes for late-onset AD were identified, which may be involv
Alzheimer's disease (AD) is the most common type of neurodegenerative dementia, but the cause of AD remained poorly und
Functional annotation of genomic variants in studies of late-onset Alzheimer's disease.
Butkiewicz M, Blue EE, Leung YY, Jian X, Marcora E, Renton AE, Kuzma A, Wang LS, Koboldt DC, Haines JL, Bush WS. Butkiewicz M, et al. Bioinformatics. 2018 Aug 15;34(16):2724-2731. doi: 10.1093/bioinformatics/bty177. Bioinformatics. 2018. PMID: 29590295 Free PMC article.
Computational predictions of variant function are routinely incorporated into gene-based analyses of rare-variants, though to date most studies use limited information for assessing variant function that is often agnostic of the disease being studied. RESULTS: In th …
Computational predictions of variant function are routinely incorporated into gene-based analyses of rare-variants, though to date most stud …
Variants in the ATP-binding cassette transporter (ABCA7), apolipoprotein E 4,and the risk of late-onset Alzheimer disease in African Americans.
Reitz C, Jun G, Naj A, Rajbhandary R, Vardarajan BN, Wang LS, Valladares O, Lin CF, Larson EB, Graff-Radford NR, Evans D, De Jager PL, Crane PK, Buxbaum JD, Murrell JR, Raj T, Ertekin-Taner N, Logue M, Baldwin CT, Green RC, Barnes LL, Cantwell LB, Fallin MD, Go RC, Griffith P, Obisesan TO, Manly JJ, Lunetta KL, Kamboh MI, Lopez OL, Bennett DA, Hendrie H, Hall KS, Goate AM, Byrd GS, Kukull WA, Foroud TM, Haines JL, Farrer LA, Pericak-Vance MA, Schellenberg GD, Mayeux R; Alzheimer Disease Genetics Consortium. Reitz C, et al. JAMA. 2013 Apr 10;309(14):1483-92. doi: 10.1001/jama.2013.2973. JAMA. 2013. PMID: 23571587 Free PMC article.
IMPORTANCE: Genetic variants associated with susceptibility to late-onset Alzheimer disease are known for individuals of European ancestry, but whether the same or different variants account for the genetic risk of Alzheimer dis
IMPORTANCE: Genetic variants associated with susceptibility to late-onset Alzheimer disease are known for …
Lack of genetic support for shared aetiology of Coronary Artery Disease and Late-onset Alzheimer's disease.
Grace C, Clarke R, Goel A, Farrall M, Watkins H, Hopewell JC. Grace C, et al. Sci Rep. 2018 May 8;8(1):7102. doi: 10.1038/s41598-018-25460-2. Sci Rep. 2018. PMID: 29740026 Free PMC article.
Epidemiological studies suggest a positive association between coronary artery disease (CAD) and late-onset Alzheimer's disease (LOAD). This large-scale genetic study brings together 'big data' resources to examine the causal impac …
Epidemiological studies suggest a positive association between coronary artery disease (CAD) and late-onset Alzheime
Genetic perturbations of disease risk genes in mice capture transcriptomic signatures of late-onset Alzheimer's disease.
Pandey RS, Graham L, Uyar A, Preuss C, Howell GR, Carter GW. Pandey RS, et al. Mol Neurodegener. 2019 Dec 26;14(1):50. doi: 10.1186/s13024-019-0351-3. Mol Neurodegener. 2019. PMID: 31878951 Free PMC article.
BACKGROUND: New genetic and genomic resources have identified multiple genetic risk factors for late-onset Alzheimer's disease (LOAD) and characterized this common dementia at the molecular level. Experimental studies in model orga …
BACKGROUND: New genetic and genomic resources have identified multiple genetic risk factors for late-onset Al
The role of ABCA7 in Alzheimer's disease: evidence from genomics, transcriptomics and methylomics.
De Roeck A, Van Broeckhoven C, Sleegers K. De Roeck A, et al. Acta Neuropathol. 2019 Aug;138(2):201-220. doi: 10.1007/s00401-019-01994-1. Epub 2019 Mar 22. Acta Neuropathol. 2019. PMID: 30903345 Free PMC article. Review.
Genome-wide association studies (GWAS) originally identified ATP-binding cassette, sub-family A, member 7 (ABCA7), as a novel risk gene of Alzheimer's disease (AD). Since then, accumulating evidence from in vitro, in vivo, and human-based studies has corrobor …
Genome-wide association studies (GWAS) originally identified ATP-binding cassette, sub-family A, member 7 (ABCA7), as a novel risk gene of …
Association of HMGCR polymorphism with late-onset Alzheimer's disease in Han Chinese.
Chang XL, Tan L, Tan MS, Wang HF, Tan CC, Zhang W, Zheng ZJ, Kong LL, Wang ZX, Jiang T, Yu JT, Tan L. Chang XL, et al. Oncotarget. 2016 Apr 19;7(16):22746-51. doi: 10.18632/oncotarget.8176. Oncotarget. 2016. PMID: 27009838 Free PMC article.
The 3-hydroxy-3-methylglutaryl-CoA reductase (HMGCR) acts as a potential genetic modifier for Alzheimer's disease (AD). Previous reports identified that HMGCR rs3846662 polymorphism is associated with biosynthesis of cholesterol in AD pathology. In ord …
The 3-hydroxy-3-methylglutaryl-CoA reductase (HMGCR) acts as a potential genetic modifier for Alzheimer's disease
Late Onset Alzheimer's Disease Risk Variants in Cognitive Decline: The PATH Through Life Study.
Andrews SJ, Das D, Anstey KJ, Easteal S. Andrews SJ, et al. J Alzheimers Dis. 2017;57(2):423-436. doi: 10.3233/JAD-160774. J Alzheimers Dis. 2017. PMID: 28269768
Recent genome wide association studies have identified a number of single nucleotide polymorphisms associated with late onset Alzheimer's disease (LOAD). We examined the associations of 24 LOAD risk loci, individually and collectively as a ge
Recent genome wide association studies have identified a number of single nucleotide polymorphisms associated with late onset
Analysis of the Putative Role of CR1 in Alzheimer's Disease: Genetic Association, Expression and Function.
Fonseca MI, Chu S, Pierce AL, Brubaker WD, Hauhart RE, Mastroeni D, Clarke EV, Rogers J, Atkinson JP, Tenner AJ. Fonseca MI, et al. PLoS One. 2016 Feb 25;11(2):e0149792. doi: 10.1371/journal.pone.0149792. eCollection 2016. PLoS One. 2016. PMID: 26914463 Free PMC article.
Chronic activation of the complement system and induced inflammation are associated with neuropathology in Alzheimer's disease (AD). Recent large genome wide association studies (GWAS) have identified single nucleotide polymorphisms (SNPs) in the C3b/C4b rece …
Chronic activation of the complement system and induced inflammation are associated with neuropathology in Alzheimer's dise
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