Black EJ - Search Results

1

Chk1 is required for spindle checkpoint function.

Zachos G, Black EJ, Walker M, Scott MT, Vagnarelli P, Earnshaw WC, Gillespie DA. Zachos G, et al. Among authors: black ej. Dev Cell. 2007 Feb;12(2):247-60. doi: 10.1016/j.devcel.2007.01.003. Dev Cell. 2007. PMID: 17276342 Free PMC article.

2

An oncogenic mutation uncouples the v-Jun oncoprotein from positive regulation by the SAPK/JNK pathway in vivo.

May GH, Funk M, Black EJ, Clark W, Hussain S, Woodgett JR, Gillespie DA. May GH, et al. Among authors: black ej. Curr Biol. 1998 Jan 15;8(2):117-20. doi: 10.1016/s0960-9822(98)70043-0. Curr Biol. 1998. PMID: 9427647

3

Cdk-mediated phosphorylation of Chk1 is required for efficient activation and full checkpoint proficiency in response to DNA damage.

Xu N, Libertini S, Black EJ, Lao Y, Hegarat N, Walker M, Gillespie DA. Xu N, et al. Among authors: black ej. Oncogene. 2012 Mar 1;31(9):1086-94. doi: 10.1038/onc.2011.310. Epub 2011 Jul 18. Oncogene. 2012. PMID: 21765472

4

Chk1 C-terminal regulatory phosphorylation mediates checkpoint activation by de-repression of Chk1 catalytic activity.

Walker M, Black EJ, Oehler V, Gillespie DA, Scott MT. Walker M, et al. Among authors: black ej. Oncogene. 2009 Jun 18;28(24):2314-23. doi: 10.1038/onc.2009.102. Epub 2009 May 4. Oncogene. 2009. PMID: 19421147 Free PMC article.

5

Chk2 is required for optimal mitotic delay in response to irradiation-induced DNA damage incurred in G2 phase.

Rainey MD, Black EJ, Zachos G, Gillespie DA. Rainey MD, et al. Among authors: black ej. Oncogene. 2008 Feb 7;27(7):896-906. doi: 10.1038/sj.onc.1210702. Epub 2007 Aug 6. Oncogene. 2008. PMID: 17684483

6

v-Jun overrides the mitogen dependence of S-phase entry by deregulating retinoblastoma protein phosphorylation and E2F-pocket protein interactions as a consequence of enhanced cyclin E-cdk2 catalytic activity.

Clark W, Black EJ, MacLaren A, Kruse U, LaThangue N, Vogt PK, Gillespie DA. Clark W, et al. Among authors: black ej. Mol Cell Biol. 2000 Apr;20(7):2529-42. doi: 10.1128/mcb.20.7.2529-2542.2000. Mol Cell Biol. 2000. PMID: 10713176 Free PMC article.

7

Transient deactivation of ERK signalling is sufficient for stable entry into G0 in primary avian fibroblasts.

Black EJ, Clark W, Gillespie DA. Black EJ, et al. Curr Biol. 2000 Sep 21;10(18):1119-22. doi: 10.1016/s0960-9822(00)00699-0. Curr Biol. 2000. PMID: 10996792

8

Phosphorylation at serine 331 is required for Aurora B activation.

Petsalaki E, Akoumianaki T, Black EJ, Gillespie DA, Zachos G. Petsalaki E, et al. Among authors: black ej. J Cell Biol. 2011 Oct 31;195(3):449-66. doi: 10.1083/jcb.201104023. Epub 2011 Oct 24. J Cell Biol. 2011. PMID: 22024163 Free PMC article.

9

Vertebrate cells genetically deficient for Cdc14A or Cdc14B retain DNA damage checkpoint proficiency but are impaired in DNA repair.

Mocciaro A, Berdougo E, Zeng K, Black E, Vagnarelli P, Earnshaw W, Gillespie D, Jallepalli P, Schiebel E. Mocciaro A, et al. J Cell Biol. 2010 May 17;189(4):631-9. doi: 10.1083/jcb.200910057. J Cell Biol. 2010. PMID: 20479464 Free PMC article.

10

Cell transformation by v-Jun deactivates ERK MAP kinase signalling.

Black EJ, Walker M, Clark W, MacLaren A, Gillespie DA. Black EJ, et al. Oncogene. 2002 Sep 19;21(42):6540-8. doi: 10.1038/sj.onc.1205851. Oncogene. 2002. PMID: 12226757

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