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Role of Muller cells in retinal degenerations.
Bringmann A, Reichenbach A. Bringmann A, et al. Front Biosci. 2001 Oct 1;6:E72-92. doi: 10.2741/bringman. Front Biosci. 2001. PMID: 11578954 Review.
This morphological relationship is reflected by a multitude of functional interactions between retinal neurons and Muller cells, including extracellular ion homeostasis and glutamate recycling by Muller cells. Virtually every disease of the retina is associated with a
This morphological relationship is reflected by a multitude of functional interactions between retinal neurons and Muller cells, incl …
Mammalian retinal glial (Müller) cells express large-conductance Ca(2+)-activated K+ channels that are modulated by Mg2+ and pH and activated by protein kinase A.
Bringmann A, Faude F, Reichenbach A. Bringmann A, et al. Glia. 1997 Apr;19(4):311-23. doi: 10.1002/(sici)1098-1136(199704)19:4<311::aid-glia4>3.0.co;2-#. Glia. 1997. PMID: 9097075
The maxi-K+ channels in Müller cells of both species display a single channel conductance of 175 pS in cell-attached and inside-out patches (125/110 mM K+). ...Phosphorylation of the channel after cytosolic addition of the catalytic subunit of a cAMP dependent prote …
The maxi-K+ channels in Müller cells of both species display a single channel conductance of 175 pS in cell-attached and inside-out p …
The Müller (glial) cell in normal and diseased retina: a case for single-cell electrophysiology.
Reichenbach A, Faude F, Enzmann V, Bringmann A, Pannicke T, Francke M, Biedermann B, Kuhrt H, Stolzenburg JU, Skatchkov SN, Heinemann U, Wiedemann P, Reichelt W. Reichenbach A, et al. Ophthalmic Res. 1997;29(5):326-40. doi: 10.1159/000268031. Ophthalmic Res. 1997. PMID: 9323724
Heterogenous expression of Ca2+-dependent K+ currents by Müller glial cells.
Bringmann A, Reichenbach A. Bringmann A, et al. Neuroreport. 1997 Dec 22;8(18):3841-5. doi: 10.1097/00001756-199712220-00001. Neuroreport. 1997. PMID: 9462452
In whole-cell records, phloretin increased the amplitude of an outward current which was blockable by iberiotoxin (40 nM) and TEA (1 mM), and, thus, was identified as a BKCa current. ...As glutamate is a major neurotransmitter of the retina, and the membrane propert …
In whole-cell records, phloretin increased the amplitude of an outward current which was blockable by iberiotoxin (40 nM) and TEA (1 mM), an …
Spermine/spermidine is expressed by retinal glial (Müller) cells and controls distinct K+ channels of their membrane.
Biedermann B, Skatchkov SN, Brunk I, Bringmann A, Pannicke T, Bernstein HG, Faude F, Germer A, Veh R, Reichenbach A. Biedermann B, et al. Glia. 1998 Jul;23(3):209-20. doi: 10.1002/(sici)1098-1136(199807)23:3<209::aid-glia4>3.0.co;2-#. Glia. 1998. PMID: 9633806
Alterations of potassium channel activity in retinal Müller glial cells induced by arachidonic acid.
Bringmann A, Skatchkov SN, Biedermann B, Faude F, Reichenbach A. Bringmann A, et al. Neuroscience. 1998 Oct;86(4):1291-306. doi: 10.1016/s0306-4522(98)00079-7. Neuroscience. 1998. PMID: 9697134
Arachidonic acid reduced the peak amplitude of the transient (A-type) outward K+ currents in a dose-dependent and reversible manner, with a 50% reduction achieved by 4.1 microM arachidonic acid. ...In cell-attached recordings with 3 mM K+ in the bath and 130 …
Arachidonic acid reduced the peak amplitude of the transient (A-type) outward K+ currents in a dose-dependent and reversible m …
Müller (glial) cell development in vivo and in retinal explant cultures: morphology and electrophysiology, and the effects of elevated ammonia.
Bringmann A, Kuhrt H, Germer A, Biedermann B, Reichenbach A. Bringmann A, et al. J Hirnforsch. 1998;39(2):193-206. J Hirnforsch. 1998. PMID: 10022343
We used such cultures as a model for the (ammonia-induced) hepatic retinopathy, earlier observed in humans with chronical liver failure, and ascribed to a breakdown of Müller (glial) cell function. ...Four days of elevated ammonia in the culture medium caused a
We used such cultures as a model for the (ammonia-induced) hepatic retinopathy, earlier observed in humans with chronical liver failu …
Protein kinases A and C are opponents in modulating glial Ca2+ -activated K+ channels.
Schopf S, Bringmann A, Reichenbach A. Schopf S, et al. Neuroreport. 1999 Apr 26;10(6):1323-7. doi: 10.1097/00001756-199904260-00031. Neuroreport. 1999. PMID: 10363947
Extracellular exposure to dibutyryl-cAMP, known to stimulate the protein kinase A, increased the open probability of the channels. ...As glial BK channels are modulated in an opposite manner by protein kinases A and C, they may act as a cellular focus of inte …
Extracellular exposure to dibutyryl-cAMP, known to stimulate the protein kinase A, increased the open probability of the channels. .. …
Human Müller glial cells: altered potassium channel activity in proliferative vitreoretinopathy.
Bringmann A, Francke M, Pannicke T, Biedermann B, Faude F, Enzmann V, Wiedemann P, Reichelt W, Reichenbach A. Bringmann A, et al. Invest Ophthalmol Vis Sci. 1999 Dec;40(13):3316-23. Invest Ophthalmol Vis Sci. 1999. PMID: 10586958
RESULTS: Cells from patients displayed a less negative mean membrane potential (-52.8 mV) than cells from healthy donors (-80.6 mV). ...At the resting membrane potential, single BK channels displayed a higher mean activity (open probability, Po, and channel current …
RESULTS: Cells from patients displayed a less negative mean membrane potential (-52.8 mV) than cells from healthy donors (-80.6 mV). …
Role of glial K(+) channels in ontogeny and gliosis: a hypothesis based upon studies on Müller cells.
Bringmann A, Francke M, Pannicke T, Biedermann B, Kodal H, Faude F, Reichelt W, Reichenbach A. Bringmann A, et al. Glia. 2000 Jan 1;29(1):35-44. doi: 10.1002/(sici)1098-1136(20000101)29:1<35::aid-glia4>3.0.co;2-a. Glia. 2000. PMID: 10594921 Review.
The K(IR) channels mediate spatial buffering K(+) currents and maintain a stable hyperpolarized membrane potential necessary for various glial-neuronal interactions. ...It is concluded that in respect to their K(+) current pattern, mature Müller cells pass through a
The K(IR) channels mediate spatial buffering K(+) currents and maintain a stable hyperpolarized membrane potential necessary for vari …
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