Brown JH - Search Results

1

CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α.

Gray CB, Suetomi T, Xiang S, Mishra S, Blackwood EA, Glembotski CC, Miyamoto S, Westenbrink BD, Brown JH. Gray CB, et al. Among authors: brown jh. J Mol Cell Cardiol. 2017 Feb;103:48-55. doi: 10.1016/j.yjmcc.2017.01.002. Epub 2017 Jan 7. J Mol Cell Cardiol. 2017. PMID: 28077321 Free PMC article.

2

Location matters: clarifying the concept of nuclear and cytosolic CaMKII subtypes.

Mishra S, Gray CB, Miyamoto S, Bers DM, Brown JH. Mishra S, et al. Among authors: brown jh. Circ Res. 2011 Dec 9;109(12):1354-62. doi: 10.1161/CIRCRESAHA.111.248401. Epub 2011 Oct 13. Circ Res. 2011. PMID: 21998325 Free PMC article.

3

The promise of CaMKII inhibition for heart disease: preventing heart failure and arrhythmias.

Westenbrink BD, Edwards AG, McCulloch AD, Brown JH. Westenbrink BD, et al. Among authors: brown jh. Expert Opin Ther Targets. 2013 Aug;17(8):889-903. doi: 10.1517/14728222.2013.809064. Epub 2013 Jun 24. Expert Opin Ther Targets. 2013. PMID: 23789646 Free PMC article. Review.

4

Induction of the matricellular protein CCN1 through RhoA and MRTF-A contributes to ischemic cardioprotection.

Zhao X, Ding EY, Yu OM, Xiang SY, Tan-Sah VP, Yung BS, Hedgpeth J, Neubig RR, Lau LF, Brown JH, Miyamoto S. Zhao X, et al. Among authors: brown jh. J Mol Cell Cardiol. 2014 Oct;75:152-61. doi: 10.1016/j.yjmcc.2014.07.017. Epub 2014 Aug 8. J Mol Cell Cardiol. 2014. PMID: 25106095 Free PMC article.

5

CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca(2+) leak and the pathophysiological response to chronic β-adrenergic stimulation.

Grimm M, Ling H, Willeford A, Pereira L, Gray CB, Erickson JR, Sarma S, Respress JL, Wehrens XH, Bers DM, Brown JH. Grimm M, et al. Among authors: brown jh. J Mol Cell Cardiol. 2015 Aug;85:282-91. doi: 10.1016/j.yjmcc.2015.06.007. Epub 2015 Jun 14. J Mol Cell Cardiol. 2015. PMID: 26080362 Free PMC article.

6

Selective coupling of the S1P₃ receptor subtype to S1P-mediated RhoA activation and cardioprotection.

Yung BS, Brand CS, Xiang SY, Gray CB, Means CK, Rosen H, Chun J, Purcell NH, Brown JH, Miyamoto S. Yung BS, et al. Among authors: brown jh. J Mol Cell Cardiol. 2017 Feb;103:1-10. doi: 10.1016/j.yjmcc.2016.12.008. Epub 2016 Dec 23. J Mol Cell Cardiol. 2017. PMID: 28017639 Free PMC article.

7

Inflammation in nonischemic heart disease: initiation by cardiomyocyte CaMKII and NLRP3 inflammasome signaling.

Suetomi T, Miyamoto S, Brown JH. Suetomi T, et al. Among authors: brown jh. Am J Physiol Heart Circ Physiol. 2019 Nov 1;317(5):H877-H890. doi: 10.1152/ajpheart.00223.2019. Epub 2019 Aug 23. Am J Physiol Heart Circ Physiol. 2019. PMID: 31441689 Free PMC article. Review.

8

SR-targeted CaMKII inhibition improves SR Ca²+ handling, but accelerates cardiac remodeling in mice overexpressing CaMKIIδC.

Huke S, Desantiago J, Kaetzel MA, Mishra S, Brown JH, Dedman JR, Bers DM. Huke S, et al. Among authors: brown jh. J Mol Cell Cardiol. 2011 Jan;50(1):230-8. doi: 10.1016/j.yjmcc.2010.10.014. Epub 2010 Oct 21. J Mol Cell Cardiol. 2011. PMID: 20971119 Free PMC article.

9

Cardiomyocyte calcium and calcium/calmodulin-dependent protein kinase II: friends or foes?

Zhang T, Miyamoto S, Brown JH. Zhang T, et al. Among authors: brown jh. Recent Prog Horm Res. 2004;59:141-68. doi: 10.1210/rp.59.1.141. Recent Prog Horm Res. 2004. PMID: 14749501 Review.

10

Galphaq expression activates EGFR and induces Akt mediated cardiomyocyte survival: dissociation from Galphaq mediated hypertrophy.

Howes AL, Miyamoto S, Adams JW, Woodcock EA, Brown JH. Howes AL, et al. Among authors: brown jh. J Mol Cell Cardiol. 2006 May;40(5):597-604. doi: 10.1016/j.yjmcc.2005.12.003. Epub 2006 Feb 8. J Mol Cell Cardiol. 2006. PMID: 16466740

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