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Year | Number of Results |
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O-GlcNAc modification is an endogenous inhibitor of the proteasome.
Cell. 2003 Dec 12;115(6):715-25. doi: 10.1016/s0092-8674(03)00974-7.
Cell. 2003.
PMID: 14675536
Free article.
Muscle-specific overexpression of NCOATGK, splice variant of O-GlcNAcase, induces skeletal muscle atrophy.
Huang P, Ho SR, Wang K, Roessler BC, Zhang F, Hu Y, Bowe DB, Kudlow JE, Paterson AJ.
Huang P, et al.
Am J Physiol Cell Physiol. 2011 Mar;300(3):C456-65. doi: 10.1152/ajpcell.00124.2010. Epub 2010 Dec 22.
Am J Physiol Cell Physiol. 2011.
PMID: 21178104
Free PMC article.
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Increased O-GlcNAc causes disrupted lens fiber cell differentiation and cataracts.
Wang K, Ho SR, Mao W, Huang P, Zhang F, Schwiebert EM, Kudlow JE, Paterson AJ.
Wang K, et al.
Biochem Biophys Res Commun. 2009 Sep 11;387(1):70-6. doi: 10.1016/j.bbrc.2009.06.132. Epub 2009 Jul 3.
Biochem Biophys Res Commun. 2009.
PMID: 19577582
Free PMC article.
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Preferential reduction of beta cells derived from Pax6-MafB pathway in MafB deficient mice.
Nishimura W, Rowan S, Salameh T, Maas RL, Bonner-Weir S, Sell SM, Sharma A.
Nishimura W, et al.
Dev Biol. 2008 Feb 15;314(2):443-56. doi: 10.1016/j.ydbio.2007.12.009. Epub 2007 Dec 23.
Dev Biol. 2008.
PMID: 18199433
Free PMC article.
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