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2013 1
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Page 1
Role of vitamin D and calcium signaling in epidermal wound healing.
Bikle DD. Bikle DD. J Endocrinol Invest. 2023 Feb;46(2):205-212. doi: 10.1007/s40618-022-01893-5. Epub 2022 Aug 13. J Endocrinol Invest. 2023. PMID: 35963983 Free PMC article. Review.
PURPOSE: This review will discuss the role of vitamin D and calcium signaling in the epidermal wound response with particular focus on the stem cells of the epidermis and hair follicle that contribute to the wounding response. ...RESULTS: Following wounding the stem …
PURPOSE: This review will discuss the role of vitamin D and calcium signaling in the epidermal wound response with particular focus o …
CDH1 is Identified as A Therapeutic Target for Skin Regeneration after Mechanical Loading.
Huang X, Liang X, Zhou Y, Li H, Du H, Suo Y, Liu W, Jin R, Chai B, Duan R, Li H, Li Q. Huang X, et al. Int J Biol Sci. 2021 Jan 1;17(1):353-367. doi: 10.7150/ijbs.51309. eCollection 2021. Int J Biol Sci. 2021. PMID: 33390855 Free PMC article.
Furthermore, the forkhead box O1/Kruppel-like factor 4 (FOXO1/KLF4) pathway was activated and contributed to the keratinocyte dedifferentiation. In vivo studies showed that mechanical loading and Cdh1 interference facilitated epidermal dedifferentiation and p …
Furthermore, the forkhead box O1/Kruppel-like factor 4 (FOXO1/KLF4) pathway was activated and contributed to the keratinocyte dediffe …
p120-catenin down-regulation and epidermal growth factor receptor overexpression results in a transformed epithelium that mimics esophageal squamous cell carcinoma.
Lehman HL, Yang X, Welsh PA, Stairs DB. Lehman HL, et al. Am J Pathol. 2015 Jan;185(1):240-51. doi: 10.1016/j.ajpath.2014.09.008. Am J Pathol. 2015. PMID: 25529795 Free PMC article.
The molecular pathogenesis underlying the invasive mechanism of ESCC is not well known because of the lack of existing models to study this disease. p120-Catenin (p120ctn) and the epidermal growth factor receptor (EGFR) have each been implicated in several cancers, includi …
The molecular pathogenesis underlying the invasive mechanism of ESCC is not well known because of the lack of existing models to study this …
IRF6 Regulates the Delivery of E-Cadherin to the Plasma Membrane.
Antiguas A, DeMali KA, Dunnwald M. Antiguas A, et al. J Invest Dermatol. 2022 Feb;142(2):314-322. doi: 10.1016/j.jid.2021.06.031. Epub 2021 Jul 24. J Invest Dermatol. 2022. PMID: 34310950 Free PMC article.
IRF6 is a transcription factor that is required for craniofacial development and epidermal morphogenesis. Specifically, Irf6-deficient mice lack the terminally differentiated epidermal layers, leading to an absence of barrier function. ...However, the mechanisms by …
IRF6 is a transcription factor that is required for craniofacial development and epidermal morphogenesis. Specifically, Irf6-deficien …
ZNF185 is a p63 target gene critical for epidermal differentiation and squamous cell carcinoma development.
Smirnov A, Lena AM, Cappello A, Panatta E, Anemona L, Bischetti S, Annicchiarico-Petruzzelli M, Mauriello A, Melino G, Candi E. Smirnov A, et al. Oncogene. 2019 Mar;38(10):1625-1638. doi: 10.1038/s41388-018-0509-4. Epub 2018 Oct 18. Oncogene. 2019. PMID: 30337687 Free PMC article.
Similarly, limited data are available to understand if deregulation of the cell-cell adhesion programme is important in tumour formation. Here, using the epidermis as an experimental model with the RNA sequencing approach, we identify a novel p63-regulated gene induced dur …
Similarly, limited data are available to understand if deregulation of the cell-cell adhesion programme is important in tumour formation. He …
Obligatory roles of filamin A in E-cadherin-mediated cell-cell adhesion in epidermal keratinocytes.
Tu CL, You M. Tu CL, et al. J Dermatol Sci. 2014 Feb;73(2):142-51. doi: 10.1016/j.jdermsci.2013.09.007. Epub 2013 Sep 26. J Dermatol Sci. 2014. PMID: 24120284 Free PMC article.
BACKGROUND: Extracellular Ca(2+) (Cao(2+))-induced E-cadherin-mediated cell-cell adhesion plays a critical role in promoting differentiation in epidermal keratinocytes. Our previous studies show that the calcium-sensing receptor (CaR) regulates keratinocyte c …
BACKGROUND: Extracellular Ca(2+) (Cao(2+))-induced E-cadherin-mediated cell-cell adhesion plays a critical role in promoting differentiation …
Decoding a gene expression program that accompanies the phenotype of sporadic and basal cell nevus syndrome-associated odontogenic keratocyst.
Kalogirou EM, Foutadakis S, Koutsi MA, Vatsellas G, Vlachodimitropoulos D, Petsinis V, Sklavounou A, Agelopoulos M, Tosios KI. Kalogirou EM, et al. J Oral Pathol Med. 2022 Aug;51(7):649-658. doi: 10.1111/jop.13325. Epub 2022 Jul 1. J Oral Pathol Med. 2022. PMID: 35665542
RESULTS: 2654 and 2427 differentially expressed genes were captured to characterize the transcriptome of sporadic and basal cell nevus syndrome-associated odontogenic keratocysts, respectively. Gene ontologies related to "epidermis/skin development" and "keratinocyte …
RESULTS: 2654 and 2427 differentially expressed genes were captured to characterize the transcriptome of sporadic and basal cell nevus syndr …
E-cadherin and aquaporin-3 are downregulated in wound edges of human chronic wounds.
Ernstsen CV, Riishede A, Iversen AKS, Bay L, Bjarnsholt T, Nejsum LN. Ernstsen CV, et al. APMIS. 2023 Aug;131(8):403-409. doi: 10.1111/apm.13332. Epub 2023 Jun 2. APMIS. 2023. PMID: 37267058
Chronic wounds are defined as wounds that fail to proceed through the normal phases of wound healing; a complex process involving different dynamic events including migration of keratinocytes in the epidermis. Chronic wounds are estimated to affect 1-2% of the human …
Chronic wounds are defined as wounds that fail to proceed through the normal phases of wound healing; a complex process involving different …
TRPV4 regulates matrix stiffness and TGFβ1-induced epithelial-mesenchymal transition.
Sharma S, Goswami R, Zhang DX, Rahaman SO. Sharma S, et al. J Cell Mol Med. 2019 Feb;23(2):761-774. doi: 10.1111/jcmm.13972. Epub 2018 Nov 18. J Cell Mol Med. 2019. PMID: 30450767 Free PMC article.
Specifically, we found that: (a) genetic ablation or pharmacological inhibition of TRPV4 blocked matrix stiffness and TGFbeta1-induced EMT in normal mouse primary epidermal keratinocytes (NMEKs) as determined by changes in morphology, adhesion, migration and alterat …
Specifically, we found that: (a) genetic ablation or pharmacological inhibition of TRPV4 blocked matrix stiffness and TGFbeta1-induced EMT i …
[Expression of β-catenin in Skin Lesions of Patients with Scleroderma and Its Effect on Epithelial-Mesenchymal Transition of Human Epidermal Keratinocytes].
Liu JJ, Yang HF, Li YJ, Chen YM. Liu JJ, et al. Sichuan Da Xue Xue Bao Yi Xue Ban. 2019 Sep;50(5):654-659. Sichuan Da Xue Xue Bao Yi Xue Ban. 2019. PMID: 31762233 Chinese.
OBJECTIVE: To investigate the expression of beta-catenin in the skin lesions of patients with systemic scleroderma (SSc) and its effect on epithelial-mesenchymal transition (EMT) of human epidermal keratinocytes. METHODS: The expression of beta-catenin, Snail1 and E …
OBJECTIVE: To investigate the expression of beta-catenin in the skin lesions of patients with systemic scleroderma (SSc) and its effect on e …
22 results