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Transactivation by NF-IL6/LAP is enhanced by phosphorylation of its activation domain.
Trautwein C, Caelles C, van der Geer P, Hunter T, Karin M, Chojkier M. Trautwein C, et al. Among authors: Caelles C. Nature. 1993 Aug 5;364(6437):544-7. doi: 10.1038/364544a0. Nature. 1993. PMID: 8336793
One of the members of the bZIP family of transcriptional activators is NF-IL6/LAP (IL-6 DBP, C/EBP beta, CRP2). NF-IL6/LAP protein is highly expressed in liver nuclei, where it has been implicated as a master regulator of the acute-phase response, induced by interleukin-6 (IL-6) and other inflammatory mediators. ...We now show that stimulation of the protein kinase C pathway increases the phosphorylation of Ser 105 within the activation domain of NF-IL6/LAP, and enhances its transcriptional efficacy....
One of the members of the bZIP family of transcriptional activators is NF-IL6/LAP (IL-6 DBP, C/EBP beta, CRP2). NF-IL6/LAP protein is …
Glucocorticoid-induced apoptosis of human leukemic cells is caused by the repressive function of the glucocorticoid receptor.
Helmberg A, Auphan N, Caelles C, Karin M. Helmberg A, et al. Among authors: Caelles C. EMBO J. 1995 Feb 1;14(3):452-60. EMBO J. 1995. PMID: 7859735 Free PMC article.
In this study, we show that an activation-deficient glucocorticoid receptor mutant is as effective as the wild-type receptor in repression of AP-1 activity, inhibition of interleukin-2 production, inhibition of c-myc expression and induction of apoptosis. ...
In this study, we show that an activation-deficient glucocorticoid receptor mutant is as effective as the wild-type receptor in repression o …
Glucocorticoids antagonize AP-1 by inhibiting the Activation/phosphorylation of JNK without affecting its subcellular distribution.
González MV, Jiménez B, Berciano MT, González-Sancho JM, Caelles C, Lafarga M, Muñoz A. González MV, et al. Among authors: Caelles C. J Cell Biol. 2000 Sep 4;150(5):1199-208. doi: 10.1083/jcb.150.5.1199. J Cell Biol. 2000. PMID: 10974006 Free PMC article.
Inhibition of the c-Jun NH(2)-terminal kinase (JNK) signaling pathway, the main mediator of AP-1 activation, has been described in extracts of hormone-treated cells. ...As a result, c-Jun NH(2)-terminal domain phosphorylation and induction were impaired. Dexamethasone did not block the TNF-alpha-induced JNK nuclear translocation, but rather induced, per se, nuclear accumulation of the enzyme. ...
Inhibition of the c-Jun NH(2)-terminal kinase (JNK) signaling pathway, the main mediator of AP-1 activation, has been described in ex …
UV irradiation induces the murine urokinase-type plasminogen activator gene via the c-Jun N-terminal kinase signaling pathway: requirement of an AP1 enhancer element.
Miralles F, Parra M, Caelles C, Nagamine Y, Félez J, Muñoz-Cánoves P. Miralles F, et al. Among authors: Caelles C. Mol Cell Biol. 1998 Aug;18(8):4537-47. doi: 10.1128/mcb.18.8.4537. Mol Cell Biol. 1998. PMID: 9671463 Free PMC article.
Moreover, our results indicated that wild-type N-terminal c-Jun, but not mutated c-Jun (Ala-63/73), was able to mediate UV-induced uPA transcriptional activity. ...
Moreover, our results indicated that wild-type N-terminal c-Jun, but not mutated c-Jun (Ala-63/73), was able to mediate UV-ind …
Upon Wnt stimulation, Rac1 activation requires Rac1 and Vav2 binding to p120-catenin.
Valls G, Codina M, Miller RK, Del Valle-Pérez B, Vinyoles M, Caelles C, McCrea PD, García de Herreros A, Duñach M. Valls G, et al. Among authors: Caelles C. J Cell Sci. 2012 Nov 15;125(Pt 22):5288-301. doi: 10.1242/jcs.101030. Epub 2012 Sep 3. J Cell Sci. 2012. PMID: 22946057 Free PMC article.
The gammaTuRC revisited: a comparative analysis of interphase and mitotic human gammaTuRC redefines the set of core components and identifies the novel subunit GCP8.
Teixidó-Travesa N, Villén J, Lacasa C, Bertran MT, Archinti M, Gygi SP, Caelles C, Roig J, Lüders J. Teixidó-Travesa N, et al. Among authors: Caelles C. Mol Biol Cell. 2010 Nov 15;21(22):3963-72. doi: 10.1091/mbc.E10-05-0408. Epub 2010 Sep 22. Mol Biol Cell. 2010. PMID: 20861304 Free PMC article.
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