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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1969 1
1975 1
1976 1
1978 1
1979 1
1983 1
1984 2
1985 1
1987 3
1988 1
1989 5
1990 2
1991 5
1992 7
1993 10
1994 13
1995 15
1996 14
1997 16
1998 18
1999 27
2000 30
2001 31
2002 47
2003 77
2004 123
2005 182
2006 244
2007 347
2008 450
2009 649
2010 942
2011 1392
2012 1779
2013 2223
2014 2656
2015 3078
2016 3468
2017 4075
2018 4406
2019 5123
2020 5414
2021 5381
2022 4765
2023 4260
2024 4659
2025 5295
2026 407

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54,980 results

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Page 1
Epithelial-Mesenchymal Plasticity in Cancer Progression and Metastasis.
Lu W, Kang Y. Lu W, et al. Dev Cell. 2019 May 6;49(3):361-374. doi: 10.1016/j.devcel.2019.04.010. Dev Cell. 2019. PMID: 31063755 Free PMC article. Review.
Epithelial-to-mesenchymal transition (EMT) and its reversed process, mesenchymal-to-epithelial transition (MET), are fundamental processes in embryonic development and tissue repair but confer malignant properties to carcinoma
Epithelial-to-mesenchymal transition (EMT) and its reversed process, mesenchymal-to-epithelial
EMT, CSCs, and drug resistance: the mechanistic link and clinical implications.
Shibue T, Weinberg RA. Shibue T, et al. Nat Rev Clin Oncol. 2017 Oct;14(10):611-629. doi: 10.1038/nrclinonc.2017.44. Epub 2017 Apr 11. Nat Rev Clin Oncol. 2017. PMID: 28397828 Free PMC article. Review.
This concept, taken together with the identification of the epithelial-to-mesenchymal transition (EMT) programme as a critical regulator of the CSC phenotype, offers an opportunity to investigate the nature of intratumoural heterogeneity and a possible …
This concept, taken together with the identification of the epithelial-to-mesenchymal transition (EMT) programme …
Aquaporin-3 in Cancer.
Marlar S, Jensen HH, Login FH, Nejsum LN. Marlar S, et al. Int J Mol Sci. 2017 Oct 7;18(10):2106. doi: 10.3390/ijms18102106. Int J Mol Sci. 2017. PMID: 28991174 Free PMC article. Review.
AQP3 knockout mice were resistant to skin tumor formation and overexpression correlated with metastasis and poor prognosis in patients with breast or gastric cancer. In cultured cancer cells, increased AQP3 expression stimulated several intracellular signaling pathw …
AQP3 knockout mice were resistant to skin tumor formation and overexpression correlated with metastasis and poor prognosis in patient …
GTSE1-driven ZEB1 stabilization promotes pulmonary fibrosis through the epithelial-to-mesenchymal transition.
Jin H, Park SY, Lee JE, Park H, Jeong M, Lee H, Cho J, Lee YS. Jin H, et al. Mol Ther. 2024 Nov 6;32(11):4138-4157. doi: 10.1016/j.ymthe.2024.09.029. Epub 2024 Sep 27. Mol Ther. 2024. PMID: 39342428 Free PMC article.
The comprehensive bioinformatics analyses suggested that the increase of GTSE1 in IPF is linked to the enhanced gene signature for the epithelial-to-mesenchymal transition (EMT), leading us to investigate the potential interaction between GTSE1 and EMT …
The comprehensive bioinformatics analyses suggested that the increase of GTSE1 in IPF is linked to the enhanced gene signature for the ep
Cell-Type-Specific Chromatin States Differentially Prime Squamous Cell Carcinoma Tumor-Initiating Cells for Epithelial to Mesenchymal Transition.
Latil M, Nassar D, Beck B, Boumahdi S, Wang L, Brisebarre A, Dubois C, Nkusi E, Lenglez S, Checinska A, Vercauteren Drubbel A, Devos M, Declercq W, Yi R, Blanpain C. Latil M, et al. Cell Stem Cell. 2017 Feb 2;20(2):191-204.e5. doi: 10.1016/j.stem.2016.10.018. Epub 2016 Nov 23. Cell Stem Cell. 2017. PMID: 27889319 Free PMC article.
Epithelial to mesenchymal transition (EMT) in cancer cells has been associated with metastasis, stemness, and resistance to therapy. Some tumors undergo EMT while others do not, which may reflect intrinsic properties of their cell of orig
Epithelial to mesenchymal transition (EMT) in cancer cells has been associated with metastasis, stemness,
EMT: An Update.
Thiery JP. Thiery JP. Methods Mol Biol. 2021;2179:35-39. doi: 10.1007/978-1-0716-0779-4_6. Methods Mol Biol. 2021. PMID: 32939712
Epithelial Mesenchymal Transition (EMT) initially discovered as a key developmental mechanism is now shown to be indirectly involved in fibrosis and is contributing to the progression of carcinomas. ...The debate as to whether EMT is essential for the dissemi
Epithelial Mesenchymal Transition (EMT) initially discovered as a key developmental mechanism is now shown to be indire
Deciphering epithelial-to-mesenchymal transition in pancreatic cancer.
Friend C, Parajuli P, Razzaque MS, Atfi A. Friend C, et al. Adv Cancer Res. 2023;159:37-73. doi: 10.1016/bs.acr.2023.02.008. Epub 2023 Mar 23. Adv Cancer Res. 2023. PMID: 37268401 Review.
Epithelial to mesenchymal transition (EMT) is a complex cellular program that alters epithelial cells and induces their transformation into mesenchymal cells. ...Under homeostatic conditions, initiation of EMT is mediated by
Epithelial to mesenchymal transition (EMT) is a complex cellular program that alters epithelial cells
Epithelial-to-mesenchymal transition and cancer stem(-like) cells in head and neck squamous cell carcinoma.
Chen C, Zimmermann M, Tinhofer I, Kaufmann AM, Albers AE. Chen C, et al. Cancer Lett. 2013 Sep 10;338(1):47-56. doi: 10.1016/j.canlet.2012.06.013. Epub 2012 Jul 4. Cancer Lett. 2013. PMID: 22771535 Review.
Head and neck squamous cell carcinoma (HNSCC) is the 6th commonest cancer worldwide. Relapse, thought to involve cancer stem(-like) cells (CSCs), and the development of metastases are common and survival rates remain low. Epithelial-to
Head and neck squamous cell carcinoma (HNSCC) is the 6th commonest cancer worldwide. Relapse, thought to involve cancer …
Growth Hormone and the Epithelial-to-Mesenchymal Transition.
Brittain AL, Basu R, Qian Y, Kopchick JJ. Brittain AL, et al. J Clin Endocrinol Metab. 2017 Oct 1;102(10):3662-3673. doi: 10.1210/jc.2017-01000. J Clin Endocrinol Metab. 2017. PMID: 28938477 Review.
A mechanism by which GH may play this role in cancer is through the induction of the epithelial-to-mesenchymal transition (EMT). During the EMT process, epithelial cells lose their defining phenotypes, causing loss of cellular adhesion an …
A mechanism by which GH may play this role in cancer is through the induction of the epithelial-to-mesenchymal trans
54,980 results
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